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Modern Slavery and Child Criminal Exploitation
It can take many forms and child criminal exploitation is one of them. Many children, young people and vulnerable adults we work with are forced into committing criminal acts against their will such as drug trafficking and dealing, violent and sexual offences.
Many children and young people do not recognise or admit that they are being exploited in this way. There is a complex victim/perpetrator overlap where the young person is both simultaneously.
How We Help
We work to ensure that the young people are given the right support they need and treated appropriately by the authorities given their vulnerability and the level of coercion they are under. We advocate on their behalf and help them navigate the services and support systems that are set up for victims of modern slavery.
With your support, we can help end this form of modern slavery and ensure that its youngest and most vulnerable victims get the help they need.


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A South Carolina Clomid lawsuit or Serophene lawsuit may be an option for women who took the fertility drug and had a child with birth defects. Clomiphene, a commonly prescribed fertility drug sold under the brand names Clomid and Serophene, is often given to induce ovulation in women that cannot ovulate on their own. However, Clomiphene has been allegedly linked to certain birth defects, particularly those affecting the head, heart, and digestive system. Affected patients and their families may be eligible to file a lawsuit with the help of a South Carolina Clomid lawsuit attorney.
For more information, contact Attorney Group for South Carolina. We offer free, confidential, no obligation consultations. We can help answer your questions, and if you choose to pursue a case we can connect you with an affiliated South Carolina Clomid lawsuit attorney who can assist you throughout the legal process.
What is Clomiphene?
Clomiphene is a prescription medication used to induce ovulation in women who are unable to become pregnant or who have certain medical conditions such as polycystic ovary syndrome. Although the drug has been used as an effective treatment to induce ovulation, Clomiphene poses a number of risks, including enlargement of the ovaries, multiple pregnancies, abdominal pain or discomfort, blurred vision, nausea, and dizziness.
How Does Clomiphene Work?
Clomiphene works by acting on the estrogen receptors at the hypothalamus, an important hormone control center in the brain. The hypothalamus then releases follicle stimulating hormone (FSH) and luteinizing hormone (LH) are to stimulate the production of eggs.
In a similar way, Clomiphene has been prescribed to treat male infertility. While doctors may prescribe Clomiphene to treat such conditions, the drug is not approved by the U.S. Food and Drug Administration (FDA) and is considered “off-label.”
Clomiphene Birth Defect Risks
A study reported on by the Centers for Disease Control and Prevention (CDC), using data from the National Birth Defects Prevention Study (NBDPS), notes a correlation between clomiphene use and serious birth defects, including septal heart defects and muscular ventricular septal defect. Birth defects affecting those children include defects that affect the head, heart, and limbs of the child.
Findings of the study indicate that children of mothers who used Clomiphene experienced the following complications in greater numbers than children of mothers who did not use the drug:
- Unusual brain and skull development known as anencephaly
- Septal heart defects, or abnormal connection between the heart’s lower chambers
- Narrowing of the aorta, a large, oxygen carrying blood vessel
- Esophageal atresia, or underdeveloped esophagus
- Premature closure of the skull known as craniosynostosis
- Omphalocele, a condition in which the intestine or abdominal organs are located outside of the body
- Dandy-Walker malformation, a type of brain malformation
- Muscular ventricular heart defects, or a hole in the wall of the heart’s lower chambers
- An unusual position of the large intestine called cloacal exstrophy
In the case of some defects, such as those affecting the heart, the child may require multiple medical procedures as they age in addition to initial surgical procedures within the first three years of their life.
Fertility Drugs and Autism
Autism Spectrum Disorder (ASD) may include a wide range of conditions, including Asperger’s syndrome; pervasive developmental disorder, not otherwise specified (PDD-NOS); and autistic disorder. Autism Spectrum Disorder is often characterized by persistent deficits in social communication and interaction across a number of different contexts; restricted, repetitive patterns of behavior, interests, or activities; and are usually recognized within the first two years of the baby’s life.
In June 2010, researchers from the Harvard School of Public Health reported to attendees at the International Meeting for Autism Research in Philadelphia that women who used fertility drugs to induce ovulation had nearly twice the risk of having a child with ASD verses women who did not use any fertility treatment.
Other Clomid Side Effects
According to the FDA, the following side effects have also been reportedly associated with ovulation induction therapy with Clomid:
- Cleft palate
- Visual disorders
- Club foot
- Multiple pregnancies
- Down syndrome
- Spina bifida
How a South Carolina Clomid Lawsuit Can Help
Drug makers have a duty to provide safe products. If there are risks of harm associated with their products, they also must provide adequate warnings. If a drug maker fails to fulfill this duty, it could be held liable in lawsuits for injuries that may result.
People injured by bad drugs may be eligible to recover money for:
- Medical Expenses
- Lost Wages
- Pain and Suffering
The families of those who have died may be eligible to recover money for funeral expenses and the pain that comes with losing a loved one.
The Time You Have to Pursue a Claim is Limited. Contact Us Today.
For more information, contact Attorney Group for South Carolina. You can fill out the form on this page or contact us by phone or email.
After you contact us, an attorney will follow up to answer questions that you might have. There is no cost or obligation to speak with us, and any information you provide will be kept confidential.
Please note that the law limits the time you have to pursue a claim or file a lawsuit for an injury. If you think you have a case, you should not delay taking action.

domain: health


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Genesis Today has just released a new vegan superfood called "GenEssentials Greens," a concentrated superfood with organic greens. The new formulation is released after years of behind the scenes work, and now holds the honor of being the strongest green superfood on the market.
GenEssentials is a concentrated vegan superfood supplement with over 75 plant-based ingredients. One serving of GenEssentials delivers the phytonutrients of up to five servings of dark leafy green vegetables and 173 other remarkable nutrients including a raw synergistic blend of herbs, superfruits, enzymes and probiotics that's been doctor-formulated to support digestion, detoxification, healthy energy and so much more."Simply mix the GenEssentials with water or juice in the morning and you're both detoxifying and fortifying every one of the 60 trillion cells that make up your body," said Dr Lindsey Duncan, CEO, Founder and Head Formulator of Genesis Today Inc.
GenEssentials is a pure, healthy product with no chemicals, toxins, colors, preservatives, fillers or pesticides. The product is also completely vegan, gluten-free, GMO-free, dairy-free, sugar-free, yeast-free and soy-free.
Green superfoods are known to improve health, detoxify the body, improve digestion, flush toxins and provide essential vitamins and minerals often missing from our modern-day lives. They have become more popular in recent years, with the resurgence of awareness in healthier lifestyles.
These products are particularly beneficial for people subscribing to vegan and vegetarian lifestyles, as many of the vitamins that are difficult to consume in quantity are available in one serving.
Genesis Today is a leader in the supplement and superfood industry. You can learn more about the GenEssentials line and other leading products here.
Photo credit: Genesis Today

domain: food


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Free Mobile Online Games
Free the Ball is a simple yet highly addictive unblock puzzle game that will keep you hooked to the screen for a long time. In this fantastic brain teaser game your goal is to create a path for the ball to reach the goal by sliding blocks. To keep you entertained for months Free The Ball features not less than three thousand levels. Yes, we are not kidding you - three thousand levels and all of them are completely free without any purchases. Are you ready to take the challenge to solve each and every of these tricky puzzles? Play Free The Ball now and enjoy endless hours of entertainment!
Language: deutsch, english, français, italiano, Nederlands, polski, português, Русский язык, español, Türkçe, Arabic
Rating: 4 out of 5
Epic Chicken Griller
A chicken on one side, dangerous electricity on the other - clearly it is your objective to save the poor little thing from getting shocked, right? You couldn't be more wrong! After all, this fun game is called "Epic Chicken Griller" for a reason! It is all about logic puzzles and of course about properly grilling the chicken. In each level you get some components to route the electricity right to the pullet. While you prepare your serving you have to take care not to electrocute or drop the tomatoes. Who wants a grilled tomato anyway? Play now for free and tease your brain and your tongue!
Office Slacking, the brand new slacking game is here! Anna has a new job working in an office. Of course she doesn’t have any of this “do some actual work” stuff. Instead she texts her friend, makes her nails or just has a bite of chocolate.You have to finish all this mini-tasks before the time is up. When the big red “!” appears on the screen her boss is coming and might catch her slacking off. You lose if you get caught by the boss or if the time is up before finishing all tasks. Play Office Slacking now for free and and enjoy some time off!
Games for Girls, Puzzle Games
Fancy Diver 3
In Fancy Diver 3 it is your task to help the divers to reach the surface. To remove the blocks that keep the divers from reaching the surface, you just have to click or tap on a group of at least three blocks of the same colour. If you manage to remove more blocks with one tap you will be rewarded with mighty boosters like a bomb. The bomb bonus will clear up an whole area and can make all the difference for the locked in divers. It's up to you and your strategic skills, if they can make it to the surface or... well, you will not let this happen, right?! Play Fancy Diver 3 now online and for free and try to save all the divers!
Puzzle Games, Skill, Action Games, Arcade Games

domain: game


Online Insurance Brokers Eye Potential ACA Windfall
Online insurance brokers want consumers to use their websites to purchase health plans under the Affordable Care Act. In March, HHS approved partnerships between states and online insurance brokers but set strict standards on the information that the private websites must display. However, consumer advocates warn that the brokers might undermine the ACA by heavily promoting plans that pay a larger commission.
- "In Obamacare, Online Insurance Brokers See Potential Windfall" (Kliff, "Wonkblog," Washington Post, 4/8).


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Make no mistake about it, A Plague Tale: Innocence is a dark game. And is definitely suited to the Dark Ages setting in which it’s both taking place, and historically based. The developers dedication to historical accuracy is important, and it’s that focus which is the highlight of the second episode of the webseries being produced to chronicle development on the title.
Where the first episode placed the spotlight on the brother and sister dynamic, and the fight for safety and security, which is a crucial element of conflict within A Plague Tale: Innocence, this latest video shines a light on the world itself. Asobo Studio wants to make it very clear to the player the desperate circumstances they’re in.
A Plague Tale: Innocence takes place in 1300s during the infamous Black Plague. This era of history is a bit of mystery to many people. Tons of historical inaccuracies and misconceptions exist about the period, and unsurprisingly, most video games tend to shy away from graphic depictions of the squalor, death and despair the period was rife with. Asobo wants to change that, this is why the authenticity of the narrative is so important. Historical knowledge is important kids, even when it’s gross and uncomfortable.
To help accomplish this realistic feel, the developers are spending a considerable amount of time creating sophisticated models and lighting that make the game world feel real and alive. The art of the games characters and environments will aid in this endeavor by helping to give the player a more accurate depiction of period clothing, housing and daily life. The ultimate goal here is a period piece of an experience that aims to educate the player while telling them an emotional story, and it looks like the plan is proceeding nicely.
Asobo Studio and Focus Home Interactive are looking to create a rich, and at times depressing, narrative, but they can’t do it with historical accuracy alone. There needs to be a real and genuinely immersive story there. And the hope is that the journey of Amicia and Hugo as they flee the plague and other threats will give the game the sense of tension it truly needs to shine.
You can find the newest episode of the developer-produced webseries down below. A Plague: Tale Innocence will finally be releasing for PC, PS4, and Xbox One on May 14.

domain: entertainment


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COVID-19 cases rise for ninth consecutive week, variants continue spreading
Nearly 5.7 million new cases were reported in the last seven-day period, above previous highs, the World Health Organization (WHO) said in its latest coronavirus update published late Tuesday.
The number of deaths from the virus also increased – now for the sixth consecutive week - with more than 87,000 confirmed victims.
Southeast Asia spike
All parts of the world reported falling numbers of infections, apart from Southeast Asia and Western Pacific regions.
And although Southeast Asia reported the highest increases in infections and deaths for the third week in a row, it was India that accounted for the vast majority of cases, with 2.17 million new cases - a 52 per cent increase.
This is the equivalent of nearly four in 10 global cases reported in the past week, followed by the United States (with 406,001 new cases, representing a 15 per cent decrease), Brazil (404,623 new cases, a 12 per cent decrease), Turkey (378,771 cases, a nine per cent decrease) and France (211,674 new cases, a nine per cent decrease).
On the three known coronavirus variants of concern, WHO said that the so-called UK strain has been detected and verified in three more countries since last week, bringing the total to 139; that’s effectively most of the world, except Greenland and several central and southern African nations.
The South African origin variant is in 87 countries and the mutations first found in Brazil and Japan, has been reported in 54.
Monitoring is ongoing into seven other so-called “variants of interest”, the UN health agency said.
Globally, there have been more than 148 million confirmed cases of COVID-19, including 3.1 million deaths, according to WHO.
As of 27 April 2021, a total of 961,231,417 vaccine doses have been administered.

domain: news


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What could be easier than making a stir fry? If you've got random veggies around the house use them to make a delicious Meatless Monday meal. The following stir-fry recipes are a breeze to make and prove that eating vegetarian can still be delicious.
Italian football legends don't come much bigger than Alessandro Del Piero. Fine Dining Lovers spoke to the former Juventus star and World Cup winner about his career, his love of food, and running his N10 restaurant in Los Angeles during the pandemic.
The long-awaited, rescheduled UEFA Euro2020 football championships are upon us, and to whet your appetite, we have selected our starting XI of the best restaurants in the world owned by footballers. See who made our first-team.


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DC Ranch has amenities to fulfill the needs of all residents, many within walking distance. Residents are treated to high-end and convenient access to grocery, dining, retail, business, medical and educational facilities all within the community. In company with the necessities of everyday life, DC Ranch provides incredible recreational and lifestyle venues including neighborhood trails, fitness clubs, spas, tennis, community centers, 47 community parks and 2 private country clubs.
Within each neighborhood there are scenic walking paths and trails that link the 47 community-maintained parks. The paths and trails also lead to the retail complexes within the neighborhood, Copper Ridge School, the community centers, both country clubs, and the hiking trails that are part of the McDowell Sonoran Conservancy and the Gateway Trailhead. There are nearly 33 miles of trails throughout the community for resident use. The paths and walking trails were designed so that no resident would have to cross a street busier than the street in front of their home to get anywhere in the neighborhood.
Every neighborhood has at least one community park and each park has it’s own unique characteristics. All are designed in keeping with the natural landscaping, and shaded sitting areas were designed to take advantage of scenic vistas.
The Desert Camp Community Center, located at the southeast corner of Thompson Peak Parkway and Desert Camp Drive, is an 8,000 square-foot complex sitting on 9 acres. It is a wonderful year-round facility for DC Ranch residents. It is open daily from 6:00 am-10:00 pm during the summer and 7:00 am-9:00 pm during the winter. There are two tennis courts, available by reservation, a heated lap pool, a wading pool for toddlers, a fitness center, locker rooms, playground, sport courts and a full complement of special events to suit the whole family. The facilities can be reserved for private events.
The Homestead Community Center, located on Legacy Boulevard just west of Thompson Peak Parkway serves as host to a variety of community events such as the Farmer’s Market, Dog Days, and Community Meetings. Residents may reserve the center for private events. The Homestead is home to The Playhouse community theatre (launched in 2006), and offers basketball courts and an Adventure Playground.
There are two beautiful private country clubs in DC Ranch – The Country Club at DC Ranch and Silverleaf Club. Membership is through an application and approval process and anyone (not just neighborhood residents) may apply for membership.
Alongside the ease and convenience of these amenities, the security amenities in DC Ranch ensure the peace of mind of its residents. In addition to gated and guard-gated neighborhoods, micro-chipped vehicle tags and roaming patrol, City Of Scottsdale police and fire stations are located within the development.
Every home in DC Ranch is just minutes away from fabulous hiking opportunities and sightseeing. The neighboring Gateway Trailhead, located in the McDowell Sonoran Conservancy, is an outdoor enthusiast’s dream-come-true, containing miles of trails with varying levels of difficulty. It is also suitable for little ones: the Bajada Nature Trail provides games and activities along the hike.
DC Ranch is located 3 miles from a major hospital: Scottsdale Healthcare Thompson Peak Hospital at the corner of Thompson Peak and Scottsdale Rd. Mayo Clinic Hospital in Phoenix is just a 10-minute drive away. The Mayo Clinic, and Scottsdale Healthcare Shea Medical Center are also located in Scottsdale, both within 20 minutes of DC Ranch.
Featured Blog Posts

domain: geography


lighting incorporating LEDs in right colours and a dedicated
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conventional imaging problems such as lighting, noise and other
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Learn to beautify your outdoor spaces with the tips in this article.You can determine what you need, so that you don’t waste time and money on equipment you don’t need, or on seeds that will not work in your environment.
The handles of your gardening tools can double as clever rulers. Just run some measuring tape right on the floor next to the handle. Label the distances onto them using a permanent marker.
Plant some perennials that are slug-proof. Slugs and snails are voracious eaters that can kill a plant in one night. These pests gravitate to young perennials with smooth, herbaceous stems and leaves, thin leaves. Some perennials, particularly perennials that have hairy, tough leaves or a taste that isn’t appetizing. Some varieties of these plants are campanula, campanula, heuchera, and heuchera.
You don’t need a costly chemical treatments for plant mildew.Mix a bit of liquid soap and some baking soda in water. Spray this mix on your plants once a week until the mildew should go away. This mixture will not hurt your plants and the mildew safely.
Pick the proper soil to get the best outcome. You can also create an artificial area with only one type of soil.
Bulbs will give you wonderful flowers that you can enjoy in early spring and summer flowers.Different types of bulbs bloom at different times, so if you choose appropriately, you may have blooms early spring to later summer.
Protect your delicate deciduous shrubs which are tender. Tie together the tops, and loosely cover the wigwam with a blanket or sheet. This method is much better than wrapping the plant in plastic, as it allows air to circulate, which will help to prevent rotting.
If you are going to grow peas, start them indoors before putting them in the ground outside. The seeds will grow better germination rate if planted there first. The seedlings tend to be healthier, which means they can resist pests and diseases better. You could transplant the seedlings outdoors once they are sturdy enough.
Bees will go straight for these plants in the springtime. Spiders, ground beetles, because heather beds are generally undisturbed. Keep this in mind and always wear appropriate gardening gloves.
Using plants that all grow the same length or height result in a flat and uniform looking bed.
Using aspirin and water will help your plants fight diseases. Dissolve aspirin per gallon of water for a plant disease fighting solution.You can easily spray this concoction to help them fight of disease. Try to apply the mixture to the plants at least once in each three weeks.
The ambient temperate of a room with live plants should be kept between 65-75 degrees throughout the daylight hours. The temperature in order to effectively grow.If you wish to keep your house cooler than that, another solution you can utilize is to purchase heat lamps for your organic plants.
A little bit of research will go a long way, especially when you pair it with …

domain: agronomy


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This page was published under a previous government and is available for archival and research purposes.
2011 Regional Award Winner
P R Short and Son - Vineland Station
Ted McMeekin, Minister of Agriculture, Food and Rural Affairs and Philip Short
This innovator built a better basket that keeps fruit fresher longer, with less damage from transport and is more attractive to consumers, who can really see what they are buying. Research for the product included input from retailers who said they needed something they could stack, didn't require a lot of staff handling for display and could extend product shelf life. The recyclable clear plastic container with a lid that everyone sees in the stores now ticked all the boxes - and more. It was brought to market by an ingenious tender fruit grower from Niagara, and continues to enjoy tremendous success. In fact, almost half of the peaches grown in Ontario in 2010 were packed in these containers.


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Interested in Bass Books, CD’s and DVD’s? If so, Join the discussion in the Bass Musician Magazine Community!
One of the fun things about book reviews is that I get to experience lots of interesting and varied material! That said let’s have a look at what we have in our “Book-Bag” for this issue.
“Practice Made Perfect” by Roberto Moretti.
No matter what you do, you have to practice to get good at it or to grow enough to reach your goals. This applies to Bass playing but also carries over to anything in life. Roberto has dissected the whole process and by doing so, he can help us learn to practice more effectively and easier.
We start with an explanation of how learning happens.
Next, we have a “System” that we can apply to our particular skill to enhance our practice efforts.
Lastly, Roberto covers a list of additional things to consider.
This book is an easy read and full of useful tips that I intend to apply to my practice routine as well as to life in general. It might help you too!
“The Les Paul Guitar Book” by Tony Bacon
OK, I know this is not about Basses, but in honor of Les Paul who passed last August 13th, I want to mention this book.
Here is a photo-illustrated book that covers the entire Gibson Les Paul story. Loaded with details and well organized, this tome guides you through the evolution of these guitars.
There is a massive reference listing that covers models from 1952 until 2009.
This book is a must if you are a Les Paul Guitar fan and still a good read even if you aren’t!
“Green Day… 21st Century Breakdown” music book from Alfred publishing
Are you a “Green Day” fan? Did you enjoy this CD? Do you want to play it? You read TAB?
If you answered yes to any of these questions, then this is the book for you. It covers all the music from the May 15, 2009 release quite nicely. There is no CD with this book, but then you probably already have it…Enjoy!
Now lets discuss some books that will help build your musical foundation and take you further along the path of musical growth.
“Theory for the Contemporary Bassist” by Tracy Walton
This book is intended to be ” The ultimate guide for Blues, Rock and Jazz bassists”. I think it does a great job at covering an area often overlooked…. Theory. Written to start at the beginning and using an easy, step by step approach, there is a ton of information here for you to get under your belt!
Topics covered include:
Scales, Keys and Key Signatures
Introducing chords, 7th Chords, Expanding your Chord Vocabulary, Chord Progressions
Pentatonic Scales, Major Scales, Minor Scales
Modes of the Major scale
Passing tones and Approach Notes
The Twelve Bar Blues
Bebop Scales revisiting Slash chords
Lastly I will point out that this book has worksheets for self-study and presents examples in Standard Music Notation as well as TAB.
“Foundation Exercises for Bass” by Chuck Sher
Here is a book that is for every kind and style of bass out there!
I find that this book would be best if used with a live teacher. There is a lot of very concentrated information and it is all Standard Music Notation. Working with your teacher, you can get a lot from this book.
Intermediate level players:
The content here is designed to help you improve if you already have some kind of foundation.
This book is structured so it can be used in lessons and offers the flexibility to be custom tailored to each students needs.
So, all you teachers out there, you might want to check this one out to see if it fits with your teaching approach!
“Intro to Polyrhythms. Contracting and Expanding Time within Form” Vol. 1 by Ari Hoenig & Johannes Weidenmuller
Here we have a very interesting DVD/Book set. Well beyond beginner’s levels (best for intermediate to advanced players) this one works on the more percussive, advanced rhythm element of music, Jazz in particular.
Double bassists and drummers will benefit most from this one but there is material for pianists as well which can carry over to all instrumentalists.
If Jazz trio stuff is your cup of tea, here is a big teapot! Check it out!
Well, that is it for now… Happy reading!
All Books Available at: www.Bassbooks.com and Amazon.com
Interested in Bass Books, CD’s and DVD’s? If so,
Join the discussion in the Bass Musician Magazine Community!

domain: hobby


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Item #: WB-482
Retail Value: 1550.00
Beautifully and sleekly designed, this classic 14k white gold brushed wedding band ring is not only simple but classy.
Ring measures 5mm wide and 2mm thick.
Comfort fit design.
Also available in
18K Gold, and other widths or platinum
||14K White Gold
| Band Width
| Band Depth
We guarantee our 14K White Gold Brushed Wedding Band to be high quality, authentic jewelry, as described above.

domain: design


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This little cocktail concoction is just in time to warm your hearts (and hands!) for Thanksgiving. You know we do a specialty Thanksgiving cocktail every year, like this apple spice libation. Or Lauren Conrad’s kick ass Pilgram Punch I made a couple of years ago. So this year, we’re coming in hot with a crock pot cocktail.
Typically, I’ve been making this drink for Halloween. We’re usually outside waiting for the trick or treaters, and October in the NE means you need more than just a fire to help stay warm. However, the volume this recipe produces is perfect for your large holiday gathering. Bonus, everyone can help themselves! No need to be bartender for the night.
1 jug of apple cider
1/2 bottle Fireball
1 orange, thinly sliced
3-5 whole star anise
1 tbsp whole cloves
Dash of cinnamon
Dash of nutmeg
Pour all of the ingredients into your crock pot and simmer on high for 2 hours. After 2 hours, turn down to low until finished.
For a couple of years now, it’s been a tradition to go to Beardsley Cider Mill and get their hot-out-of-the-oven, mouth-watering, earth-shattering apple cider donuts. But I couldn’t stand the thought of eating them only once a year. I need them in my life like I need another pair of shoes – and I always need another pair of shoes. So I bought a donut pan (shout out to HomeGoods – $9.99!) and tried to recreate them myself. I’m now sharing them with you because trust me, you need them your life too.
And I’m sure Santa wouldn’t mind these with milk instead of cookies this year either.
1 1/2 cups flour
3/4 cup sugar
1/2 cup apple cider
1/4 cup milk
1 tbsp melted butter
2 tsp baking powder
1 tsp salt
1 tsp vanilla extract
1 tsp apple pie spice
1/2 cup brown sugar
1/2 cup sugar
1 tbsp cinnamon
2 tbsp melted butter
Preheat oven to 325 degrees and grease your donut pan. Mix together the flour, sugar, baking powder, apple pie spice, and salt. Once blended mix in the apple cider, eggs, milk, vanilla extract, and butter. Fill each cup 3/4 full and bake them for roughly 15 minutes or until golden brown. Just like a cake or cupcakes, when they feel spongy, they’re done.
While the donuts are baking, mix together the brown sugar, white sugar, and cinnamon for the coating. Once the donuts are done and have had a chance to cool, very lightly brush on the butter and toss the donut in the coating. (Warning: do not use a ton of butter for the coating. It will make the donuts soggy.)
In the spirit of traditions, every Thanksgiving I make a new “signature” cocktail to kick off the meal. I don’t have an ounce of “mixology” in my body, so I take to Pinterest for inspiration. Last year I made Lauren Conrad’s Pilgram Punch (which was dangerously good). This year I made an apple spice recipe from Love and Lemons and it was delicious! I made a few tweaks based on ingredients I already had (waste not, want not) and let me tell you, it went down nice and easy. This is an easy drink to make for all of those upcoming holiday parties.
*Disclaimer – the “base” for this drink needs time to steep and chill, so be sure to make it the night before.
1 red apple, sliced for garnish (they are sooo good to eat after you’ve finished the drink)
In a small saucepan simmer the apple juice with the cinnamon sticks and star anise. When it barely comes to a boil, remove from heat and let steep for 30 minutes, then chill for a couple of hours (or overnight).
When your’e ready to start making cocktails, remove the cinnamon sticks and star anise and set aside for garnish.In a cocktail shaker, shake the spiced apple juice, vodka, lemon, and ice together (work in batches if you need to).
Pour into glasses, add a splash of sparking water, and garnish with apple slices, cinnamon sticks and star anise pods.


UNITED STATES, WASHINGTON (OBSERVATORY) — US President Donald Trump entered into an absentee brawl on Twitter with London Mayor Sadiq Khan, who criticized him for canceling his visit to Poland for events dedicated to the 80th anniversary of the outbreak of World War II.
Earlier, trump canceled a trip to Poland, citing the need to lead measures to eliminate the consequences of Hurricane Dorian. Khan, who participated in events in Poland, said in an interview with Politico that Trump was unable to come because “he was obviously busy – he was fighting a hurricane from a golf course.”
“The incompetent mayor of London, Sadik Khan, was worried that I played a very fast game of golf yesterday,” Trump wrote. At the same time, he wrote both the name and surname of the mayor with errors (Sadiq Khan).
According to Trump, while others can play for hours or weeks to go play golf, he quickly walks through one of his fields. “Very inexpensive – President (Barack) Obama would fly to Hawaii for this,” Trump said.
The current US president has criticized Obama for expensive golf trips in the past. Trump critics claim that after the election, he himself forces taxpayers to spend many millions of dollars on ensuring his safety on vacation and on travel expenses.
Trump also advised Khan to focus on the fight against crime in London, which, according to the American president, “completely out of control.”
“People are afraid to just walk the streets. He is a terrible mayor who should not interfere in our affairs!” – concluded Trump.
Khan and Trump have repeatedly exchanged barbs in absentia. On his first visit to London, Trump called Khan “a complete loser,” and he compared the US president to a tantalizing schoolboy.
This article is written and prepared by our foreign editors writing for OBSERVATORY NEWS from different countries around the world – material edited and published by OBSERVATORY staff in our newsroom.
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text:
I realize that most of you probably don’t like electronic music as much as I do. Oh well. If you are waiting for posts on Brad Paisley you just aren’t going to find them here.
I didn’t know it when I downloaded Metro Area’s pop-happy “Read My Mind” a few years ago that it would lead me to Morgan Geist. Geist is a well-known New York DJ who was one-half of the group Metro Area. He and DJ/producer Darshan Jesrani met in 1996 and started working on music together. From what I can find they started releasing music in 1999.
Geist started releasing under his own name as well as continuing to release stuff for Metro Area. I was drawn to his 2008 album Double Night Time. Geist wrote it during a time he said he felt hopeless because the music industry was dying. He wanted to do something that wasn’t predictable or otherwise “disposable” pop music.
Double Night Time‘s tracks drip with synths and almost numb vocals from Junior Boy’s Jeremy Greenspan. Geist’s sound is like that quick breath you take while looking over your shoulder in a dark alley at night. It’s like knowing you are lost in the wrong neighborhood in Detroit. Or like the soundtrack to a movie about a contract killer who wears tight black leather gloves. It’s cold and dangerous yet stirring at once.
Enjoy some samples from the 2008 album. If you head out to look at the northern lights or a meteor shower this fall, I suggest you take Double Night Time with you.
“City Of Smoke And Flame”
“The Shore” (3:10 mark is awesome)


text:
Estimating indirect mortality impacts of armed conflict in civilian populations: panel regression analyses of 193 countries, 1990–2017
BMC Medicine volume 18, Article number: 266 (2020)
Armed conflict can indirectly affect population health through detrimental impacts on political and social institutions and destruction of infrastructure. This study aimed to quantify indirect mortality impacts of armed conflict in civilian populations globally and explore differential effects by armed conflict characteristics and population groups.
We included 193 countries between 1990 and 2017 and constructed fixed effects panel regression models using data from the Uppsala Conflict Data Program and Global Burden of Disease study. Mortality rates were corrected to exclude battle-related deaths. We assessed separately four different armed conflict variables (capturing binary, continuous, categorical, and quintile exposures) and ran models by cause-specific mortality stratified by age groups and sex. Post-estimation analyses calculated the number of civilian deaths.
We identified 1118 unique armed conflicts. Armed conflict was associated with increases in civilian mortality—driven by conflicts categorised as wars. Wars were associated with an increase in age-standardised all-cause mortality of 81.5 per 100,000 population (β 81.5, 95% CI 14.3–148.8) in adjusted models contributing 29.4 million civilian deaths (95% CI 22.1–36.6) globally over the study period. Mortality rates from communicable, maternal, neonatal, and nutritional diseases (β 51.3, 95% CI 2.6–99.9); non-communicable diseases (β 22.7, 95% CI 0.2–45.2); and injuries (β 7.6, 95% CI 3.4–11.7) associated with war increased, contributing 21.0 million (95% CI 16.3–25.6), 6.0 million (95% CI 4.1–8.0), and 2.4 million deaths (95% CI 1.7–3.1) respectively. War-associated increases in all-cause and cause-specific mortality were found across all age groups and both genders, but children aged 0–5 years had the largest relative increases in mortality.
Armed conflict, particularly war, is associated with a substantial indirect mortality impact among civilians globally with children most severely burdened.
The number of armed conflicts globally is at a record high with 182 wars and minor conflicts recorded in 2017 according to the Uppsala Conflict Data Program (UCDP) . Armed conflict has been highlighted by the United Nations as a major barrier to the implementation and attainment of the Sustainable Development Goals, including improvements in global health . Beyond direct and immediate casualties, armed conflict can produce enduring political instability, destroy welfare systems including health systems, and increase homelessness, unemployment, and poverty which have widespread implications for population health . The destruction of key infrastructure during armed conflict as well as general toxification of the environment can adversely impact clean water and food supplies, further elevating communicable disease risk .
Previous cross-national research on conflict and health has methodological limitations including omitting conflicts that occur between non-state groups [5,6,7,8,9,10,11,12,13,14,15,16], conceptualising the wide spectrum of conflict types and intensities as a simple binary explanatory variable [11,12,13,14,15, 17,18,19,20], and assessing only immediate or short-term impacts [5,6,7,8,9,10,11,12,13,14,15,16, 18,19,20,21,22,23]. Statistical models often lack grounding in a theoretical framework and therefore omit important confounding variables that may explain the association between armed conflict and health [6, 14, 16,17,18, 22]. Models are also prone to a bias in cases where the number of battle-related deaths determines both the armed conflict explanatory variable and health outcome [6,7,8,9,10,11,12,13, 16, 24, 25]. Studies frequently restrict their analyses to an examination of communicable diseases [5, 6, 15, 17, 18, 22] and sub-Saharan Africa [5, 14, 17, 19, 21,22,23,24,25] which limits generalisability to other conditions and settings.
A comprehensive global assessment of armed conflict and health using robust approaches and addressing these limitations is therefore past due. This study aims to assess whether armed conflict of different types and intensities indirectly affects civilian mortality in the short and long term, and to determine inequalities in effect across population sub-groups. We improve on the literature by using a long time period of analysis (1990 to 2017), assessing all conflicts globally irrespective of state involvement, conceptualising conflicts using four different explanatory variables, reconstructing mortality estimates to avoid using battle-related deaths in both the exposure and outcome, and assessing effects by age- and sex-stratified causes of death up to 10 years after conflict.
Design and setting
This study used longitudinal (panel) regression models to assess the impact of armed conflict on mortality rates in 193 countries between 1990 and 2017. Country was the unit of analysis. Longitudinal regression methods are frequently employed in health and economic studies to examine dynamic and transitional associations over time using routine data; examples include the association between democratisation and recession on health [26, 27], and between government health spending on the incidence of disease . Fixed effects specifications were employed, which adjust for time-invariant country-level factors, and can be considered similar to differences-in-differences approaches with multiple time points.
We combined several datasets for our analysis. We obtained armed conflict data from the UCDP Georeferenced Event Dataset (GED) Global v19.1 . The UCDP has collected country-year armed conflict data since 1946, and event-specific data since 1989, which have been used extensively for research purposes [5, 10, 12, 17, 18, 21]. The UCDP GED provides the best estimate of the number of battle-related deaths at the village-day level for all armed conflicts that meet the UCDP definition of having at least 25 battle-related deaths per calendar year . We took data from 1990 to 2017 and converted it into a country-year dataset. The UCDP GED assigns armed conflict events in Palestine to Israel, misclassifying the former as being conflict-free, so both countries were removed from the dataset. Further details on the UCDP methodology are shown in Additional file 1 (Paragraph 1.1).
We obtained country-level mortality data between 1990 and 2017 from the Global Burden of Disease (GBD) study . The GBD study utilises data from various sources to produce internally consistent, statistically modelled annual estimates of mortality by causes of death for each country. Estimates are not restricted to country citizens or nationals, but include migrants and refugees, including those displaced by conflict. The GBD excludes direct battle-related deaths from initial all-cause mortality calculations, reintroducing them in later calculations. Thus, battle-related deaths are listed as a separate cause of death (Category C3.3: terrorism and armed conflict) . The GBD is appropriate for studying excess deaths from armed conflict because no indirect consequences of armed conflict are built into the underlying mortality estimations. Although GBD data are modelled with smoothing functions, investigation of mortality trends for selected countries experiencing conflict shows GBD smoothing functions did not obscure abrupt changes in mortality and are unlikely to introduce bias to this analysis (Additional file 1: Fig. S1).
We developed a conceptual framework (Additional file 2) using existing conflict and health literature which informed the selection of covariates. The framework consisted of seven pathways through which armed conflict may affect civilian mortality, and seven drivers of conflict which were used as potential confounders. To capture changes in country wealth and income, we used the gross domestic product (GDP) per capita in current US dollars, available from the World Bank , and membership of the Organisation for Economic Co-operation and Development (OECD) . For changes in the degree of democratisation, we used the Varieties of Democracy (V-Dem) dataset, specifically the Multiplicative Polyarchy Index (continuous variable, range 0 to 1, created by multiplying five core components of electoral democracy) . We used V-Dem rather than the more commonly used POLITY IV [5, 7, 10, 12, 13] due to better data completeness. For changes in demographic factors, we used data on the proportion of people living with a density > 1000 people/km2, the proportion living in urban areas (both taken from the Institute for Health Metrics and Evaluation (IHME) ), and the age dependency ratio (ratio of under 15s and over 64s to the working population) taken from the World Bank . For changes in the average levels of educational attainment in countries, we used the mean years of educational attainment per capita, separately for males and females, available from the IHME . We captured changes in ethnic group composition using the Historical Index of Ethnic Fractionalisation Dataset, which corresponds to the probability that two randomly drawn individuals within a country are not from the same ethnic group . Finally, to capture changes and shocks in climate-related factors, we took data from the Emergency Disasters Database to control for the presence of droughts and earthquakes , and the IHME to control for the population-weighted mean temperature and the proportion of people living in the 5th quintile of annual rainfall. We were unable to find a suitable dataset for income inequality, although it is plausible that its effects are captured through GDP per capita and educational attainment. Unlike previous research, we did not adjust for health expenditure [5, 7,8,9], the prevalence of HIV/AIDS [11, 13], and refugee movement [5, 15] as these can be considered mediators (i.e. factors on the causal pathway between conflict and health) rather than confounders.
Our main outcome measures were all-cause and cause-specific mortality rates, as reported by the GBD study , but with deaths due to terrorism and armed conflict (Category C3.3) removed. Removing battle-related deaths from all-cause mortality prevents the bias of including battle-related deaths in both the explanatory variable and outcome measure which is commonly found in previous studies [6,7,8,9,10,11,12,13, 16, 24, 25]. The GBD study categorises cause-specific mortality into first-order (communicable, maternal, neonatal, and nutritional diseases; non-communicable diseases [NCDs]; injuries) and second-order causes .
As explanatory variables for armed conflict, we explored four different specifications. Firstly, we used a binary variable indicating the presence of armed conflict per country-year observation (0 = no [< 25 battle-related deaths per country-year], 1 = yes [≥ 25 deaths]) as per the UCDP . This approach is limited as it groups all conflicts together regardless of intensity, so a second specification was the rate of battle-related deaths per 100,000 population as a continuous measure. We used the rate rather than absolute battle-related deaths to avoid bias from emphasising small conflicts in populous countries which are unlikely to exert country-wide effects. Thirdly, as previous research has shown the relationship between battle deaths and civilian mortality to be non-linear , we explored quintiles of the rate of battle-related deaths per 100,000 population. Fourthly, we used intensity cutoffs as per the UCDP  to create a categorical conflict variable (0 = no conflict [< 25 battle-related deaths per country-conflict-year], 1 = minor conflict [25–999 deaths], 2 = war [≥ 1000 deaths]). We used county-conflict-year for this final specification to prevent misclassifying large countries with multiple small conflicts as war-affected (should the total of these conflicts be greater than 1000 battle deaths).
We described our sample using frequencies and means and reported the rates of battle-related deaths and civilian mortality by the different conflict explanatory variables. We presented graphical time trends in the number of conflicts, the number and rate of battle-related deaths, and the mean battle-related deaths per country.
We then used fixed effects linear panel regression methods to assess the relationship between armed conflict and mortality, which was estimated using the following equation:
where i is the country, t is the year, and u is the error term representing unexplained variation. We chose fixed effects over random effects given the likelihood that our error term u was correlated with our covariates (a key assumption that would be violated in random effects) and as indicated by the Hausman test. Fixed effects specifications control for time-invariant observed and unobserved country-level factors (as denoted by i in the equation above) and therefore only assess within-country associations rather than between-country associations. Model fit was assessed using scatter plots and post-regression diagnostics (including the variance inflation factor (VIF), studentised residuals, stem and leaf plots, Cook’s D, and DFITS) to identify collinear variables and outliers. Due to collinearity with female education (VIF > 30) and superior goodness-of-fit, we only included male education. Four data points were considered outliers and dropped (from the model only using a continuous measure for armed conflict) based on having large residuals and high leverage: Rwanda 1994, Bosnia and Herzegovina 1995, Congo 1997, and Eritrea 1999. Four of twelve covariates had missing data: age dependency ratio (4.3%), GDP per capita (5.8%), Multiplicative Polyarchy Index (11.7%), and Ethnic Fractionalisation Index (31.6%); the latter two were omitted from main analyses due to high levels of missing data, but their effects tested in sensitivity analyses (see below).
Our first models separately tested the association between the four explanatory variable specifications for armed conflict and age-standardised all-cause mortality, adjusting for ten covariates (GDP per capita, OECD member, population density, urban living, age dependency ratio, male education, temperature, rainfall, earthquakes, droughts), and year-country fixed effects. Mortality rates were standardised using the GBD world population standard. We included country-clustered Huber-White robust standard errors to account for possible heteroscedasticity and serial correlation. In all models, we lagged the armed conflict variable by 1 year unless otherwise stated to capture indirect deaths in the year after.
Because higher intensity armed conflicts drove the associations, our second models employed the same specification as above but using only the categorical armed conflict exposure variable for war (we present “war” vs. no conflict). These models used age-standardised all-cause and cause-specific mortality rates (first- and second-order causes; the latter helping to explain underlying drivers) and age- and sex-stratified all-cause and cause-specific mortality rates (first-order causes only). We then assessed the lagged effects of war (from 2 to 10 years with a separate model for each year lag) on age-standardised all-cause and cause-specific (first-order causes only) mortality to account for the varying progression of disease pathologies. We used the models’ “war” beta coefficients to calculate the absolute and relative change in civilian mortality and used post-estimation commands to calculate the number of civilian deaths.
In additional analyses, we explored whether armed conflicts that involved particular actors (“armed conflict actor type”) had differential associations with civilian mortality. In accordance with UCDP definitions, we categorised each armed conflict as being state-based (at least one actor is the state of a country), non-state (no actors are the state of a country), or one-sided violence (one actor in armed conflict with civilians). We analysed all four explanatory variable specifications for conflict (i.e. binary, continuous, quintiles, and categorical) by armed conflict actor type, and for countries with only one actor type in each year, we interacted actor type and the rate of battle-related deaths, leaving all specifications identical to prior models.
We undertook multiple sensitivity analyses to check the robustness of findings. First, we tested alternative model specifications with sequential addition of covariates, addition of new covariates that contained high rates of missing data, and random effects. Second, to address the possibility of misclassification bias between deaths due to armed conflict and deaths due to homicide, we repeated our main model removing deaths from interpersonal violence. Third, we used an alternative measure of armed conflict exposure derived from the Major Episodes of Political Violence dataset compiled by the Centre for Systemic Peace . This dataset captures major conflicts globally, and its binary conflict variable (0 = no conflict, 1 = conflict) is 97.6% specific and 97.7% sensitive to the UCDP “war” binary variable (0 = no conflict, 1 = war). More details about how this dataset compares to the UCDP are presented in Additional file 1: Paragraph 1.2. All analyses were conducted in Stata 15.
Between 1990 and 2017, the UCDP recorded 1118 unique armed conflicts involving 102 countries. Conflicts accounted for 1020 of 5404 (18.9%) country-year observations in the dataset, of which 686 (67.3%) were minor conflicts and 334 (32.7%) were wars (Table 1). The highest number of conflicts in any year was 182, recorded in 2017, of which 60.3% were categorised as war (Fig. 1a). Despite an increase in the number (Fig. 1b) and rate (Fig. 1c) of battle-related deaths annually since 2014, these figures were lower than during the decade from 1990 to 2000. Countries in conflict had an average of 19.2 battle-related deaths per 100,000 population, and this was substantially higher for countries in war (54.7) than in minor conflict (2.0) (Table 1). Countries in conflict had an average mortality rate of 1273 per 100,000 population compared with 962 in those without conflict; this was only slightly higher for countries in war (1355) than in minor conflict (1233). By country quintiles of the rate of battle-related deaths, the average mortality rate increased from 998 in the first quintile to 1502 in the fifth (Table 1).
Armed conflict was positively associated with an increase in the age-standardised all-cause mortality rate among all four explanatory variable specifications (Table 2). Each battle-related death per 100,000 population was associated with an increase in civilian mortality by 1.8 per 100,000 population (β 1.8, 95% CI 0.6–3.0). The fifth quintile of armed conflict exposure (i.e. > 5 battle-related deaths per 100,000 population) was associated with an increase in civilian mortality of 99.2 per 100,000 population (β 99.2, 95% CI 28.2–170.3), with other quintiles showing no significant association. Only war was associated with an increase in civilian mortality of 81.5 per 100,000 population (β 81.5, 95% CI 14.3–148.8) (Fig. 2a) with no significant association for minor conflict. The relationship was consistent when armed conflict was expressed as a binary variable (β 26.4, 95% CI − 12.9–65.8) although was non-significant. Post-estimation analysis predicted 29.4 million (95% CI 22.1–36.6) civilian deaths were attributable to all wars over the study period.
As associations identified were driven by the more intense armed conflicts, we used war as our explanatory variable in cause-specific analyses. Wars were associated with an increase in the age-standardised mortality rate from communicable, maternal, neonatal, and nutritional diseases by 51.3 per 100,000 population (β 51.3, 95% CI 2.6–99.9; relative increase 22.6%, 95% CI 1.3–43.8) (Fig. 2b and Additional file 3: Table S3). The association was driven by increased deaths from respiratory infections and tuberculosis, neglected tropical diseases and malaria, enteric infections, and maternal and neonatal disorders (Additional file 4: Fig. S4). Post-estimation analysis estimated 21.0 million (95% CI 16.3–25.6) civilian deaths from these diseases were related to all wars over the study period.
Wars were also associated with an increased civilian mortality from NCDs by 22.7 per 100,000 population (β 22.7, 95% CI 0.2–45.2; relative increase 3.4%, 95% CI 0.1–6.8; number of deaths 6.0 million, 95% CI 4.1–8.0) (Fig. 2c and Additional file 3: Table S3). The association was driven by increased cardiovascular diseases, diabetes and kidney diseases, neoplasms, and digestive diseases (Additional file 4: Fig. S4). Wars were associated with increased civilian mortality from injuries by 7.6 per 100,000 population (β 7.6, 95% CI 3.4–11.7; relative increase 10.9%, 95% CI 5.0–16.9; number of deaths 2.4 million, 95% CI 1.7–3.1) (Fig. 2d and Additional file 3: Table S3). The association was driven by self-harm and interpersonal violence (Additional file 4: Fig. S4).
Differences by sex
Wars were associated with increases in all-cause mortality for males (β 97.0, 95% CI 28.7–165.2; relative increase 8.6%, 95% CI 2.6–14.6), but not females (β 66.6, − 0.6–133.8) (Fig. 2a and Additional file 5: Table S5.1-S5.2). This pattern remained similar for mortality from communicable, maternal, neonatal, and nutritional diseases (Fig. 2b and Additional file 5: Table S5.1-S5.2) and for NCD mortality (Fig. 2c and Additional file 5: Table S5.1-S5.2). Wars were associated with higher increases in mortality from injuries in males (β 12.0, 95% CI 5.9–18.1) than in females (β 3.1, 95% CI 0.4–5.8), but their relative increases were similar (males 11.7%, 95% CI 5.8–17.6; females 8.3%, 95% CI 1.1–15.5) (Fig. 2d and Additional file 5: Table S5.1-S5.2).
Differences by age group
Absolute increases in all-cause mortality associated with war were largest among children under the age of 5 years (β 263.7, 95% CI 49.7–477.7) and adults over the age of 69 years (β 317.3, 95% CI 55.2–579.4) (Fig. 2a and Additional file 6: Table S6.1-S6.5). This pattern was consistent for deaths from communicable, maternal, neonatal, and nutritional diseases (Fig. 2b) and for deaths from injuries (Fig. 2d). However, for NCD mortality, the absolute effects were largest among adults over the age of 69 years only (β 184.1, 95% CI 18.8–349.3) (Fig. 2c and Additional file 6: Table S6.1-S6.5). Relative increases in all-cause and cause-specific mortality associated with war were consistently largest in children aged under 5 years and decreased incrementally as age increased (Fig. 2 and Additional file 6: Table S6.1-S6.5).
Following the onset of war, increases in mortality persisted for 2 years for all-cause mortality and 3 years for mortality from injuries (Additional file 7: Fig. S7). Wide confidence intervals do not preclude longer lagged effects for these outcomes nor for mortality from communicable, maternal, neonatal, and nutritional diseases and mortality from NCDs.
Countries experiencing one-sided violence had a higher average rate of battle-related deaths (21.1) than countries experiencing state-based (8.1) and non-state (2.0) conflicts (Additional file 8: Table S8.1.) This pattern remained similar for armed conflict actor types categorised as war or as quintiles. A visual inspection of scatter plots by armed conflict actor type showed no clear differences in the relationship between the rate of battle-related deaths and civilian mortality (Additional file 8: Fig. S8.1). In a sub-sample of 4133 country-years that only had one armed conflict actor type, no significant association between actor type and the rate of battle-related deaths was found (Additional file 8: Table S8.2) indicating that differential effects in mortality by armed conflict actor type is determined by the intensity of battle rather than the actors involved.
Sensitivity analyses demonstrated the robustness of our findings. Alternative model specifications including random effects (Additional file 9: Table S9.1) did not change our results. The sequential addition of covariates demonstrated model stability (Additional file 9: Table S9.2). The addition of two new covariates with substantial missing data, the Multiplicative Polyarchy Index and the Ethnic Fractionalisation Index, reduced the number of country-year observations from 4754 to 3381 and the number of countries from 183 to 152. However, the model’s output (war coefficient: β 87.7, 95% CI 19.1–156.1) was otherwise similar to our main findings, showing the inclusion of additional potential confounders did not alter our findings. An analysis of age-standardised all-cause mortality omitting deaths from interpersonal violence to assess misclassification bias also showed concordant results (Additional file 9: Table S9.3; war coefficient: β 78.3, 95% CI 11.1–145.5). Replacing the UCDP dataset with the MEPV dataset also showed a positive association between armed conflict and all-cause mortality (β 51.2, 95% CI − 6.8–109.1) although this did not attain statistical significance due to the MEPV dataset including many low-intensity conflicts in its binary conflict variable (mean rate of battle-related deaths compared with the UCDP, 22.7 vs. 53.7; Additional file 1: Table S2.2.).
This study is the first to quantify the indirect impact of armed conflict on mortality in civilian populations globally. We found that armed conflict, irrespective of how it is measured, was positively associated with all-cause mortality. While conflicts can involve a range of different state and non-state actors, we found that it is the intensity of conflict rather than who is involved which determines civilian mortality impact. Wars, the most intense form of armed conflict, were associated with an increase in age-standardised mortality of civilians from all causes by an average of 81.5 per 100,000 population which equated to approximately 29.4 million deaths between 1990 and 2017. Communicable, maternal, neonatal, and nutritional diseases (21.0 million deaths); NCDs (6.0 million deaths); and injuries (2.4 million deaths) all contributed to increased civilian deaths associated with wars. Effect estimates were disproportionately larger for children aged under 5 years, regardless of the cause of death.
Our finding of increased war-associated deaths from respiratory, enteric, and neglected tropical diseases is explained by difficulties in maintaining sanitation, avoiding overcrowded living arrangements, and continuing coverage of immunisations following armed conflict and forced displacement . We also found increased deaths from maternal and neonatal disorders, a finding supported by studies from sub-Saharan Africa [21, 24, 25], which may reflect less access to skilled birth attendants and health centres for delivery. NCDs have been highlighted as a particular health concern in modern-day protracted conflicts , especially in the growing list of conflict-affected countries where the epidemiological transition has occurred and the baseline burden of NCDs is already substantial. Our finding of increased NCD deaths associated with war supports the results of a recent review on the topic which linked both the destruction of health systems and changes to individual behaviours (e.g. increased tobacco and alcohol use) to increased cardiovascular disease risk . Our positive associations between war and injuries, including self-harm and interpersonal violence, also align with recently published literature [40, 41].
This study uses robust statistical methods to comprehensively quantify the impact of armed conflict on excess mortality globally over the last 30 years. We provide a detailed examination of this relationship by armed conflict exposure, across causes of death, and explore differential impacts by population sub-groups. However, there are several limitations. The UCDP relies on journalism and may not include smaller armed conflicts that are not considered newsworthy; however, our findings are driven mainly by the most intense conflicts so it is unlikely that small-scale omissions would affect our results. Other armed conflict datasets such as ACLED are generally incomplete on a global scale and are less suitable for longitudinal cross-national research, e.g. ACLED commenced data collection for Africa in 1997, Asia in 2010, the Middle East in 2016, and Europe in 2018 .
Another limitation is data processing of mortality estimates from the GBD. The modelling methods of the GBD study are complex, and smoothing processes may mask sudden shifts in mortality. While we have shown the presence of civilian mortality spikes in conflict-affected countries that justify our methodological approach (Additional file 1: Fig. S1), we cannot exclude the possibility that data smoothing processes may produce conservative estimates. However, our findings are in line with recent estimates of indirect deaths from several armed conflicts. Applying our model coefficient to the 2003 US-led invasion of Iraq would estimate 20,902 indirect deaths in 2004, which is slightly higher than but similar to Burnham et al.’s average estimate of 16,181 per year . Similarly, our model would estimate 21,603 indirect deaths in Yemen in 2016, whereas a recent UNDP report on the impact of armed conflict estimated an average of 32,750 per year . While other cross-national health data are available, including from the World Health Organization and World Bank, these also have shortcomings in relation to incompleteness and reporting inconsistencies, especially in conflict-affected settings. The GBD is the only source of health data to our knowledge that provides first- and second-order causes of death that can support an examination of the broader mortality patterns observed.
Our analysis is also ecological at the country-level, and our findings may mask focal points of conflict intensity sub-nationally and the likely large inequalities in impact across populations. Fixed effects panel regression produces effect sizes interpreted in relation to the “average country” rather than a global average and is more robust than that typically used by previous cross-national studies in this field. Panel regression models can also be potentially biased from unmeasured confounders which could explain both changes in mortality and conflict status. However, we control for a range of variables that are theoretically justified and serve as key proxies for potential unmeasured confounders. Finally, we were unable to capture the effect for conflict-displaced refugees who died in conflict-free countries, and this necessitates further investigation especially as these populations are likely to be heavily affected by conflict. Limitations relating to possible under-reporting of conflict, the use of country-level data, and data on refugees all suggest our analyses offer conservative estimates of the impact of conflict.
Quantifying the wider health burden of armed conflict is vital to draw the attention of citizens and world leaders to this important global issue. This study provides robust estimates that can help galvanise communities that fight to prevent conflict and to protect civilians. While our study reports average impacts of conflict globally, humanitarian and public health responses must be tailored to the unique context of each armed conflict. Health policy implications of armed conflict typically focus on secondary or tertiary prevention, such as integrating humanitarian aid and peace building initiatives with health system planning and reconstruction , and strengthening accountability for violations of international humanitarian law . While these are important goals, prevention and conflict deterrence is vital. The upstream drivers of armed conflict include socioeconomic inequalities, normalisation of militarism, and small arms and light weapons (SALW) proliferation. Health professionals can play an important role in the prevention of armed conflict, including through documenting its wider physical and mental health burden. Furthermore, health professionals in all countries have the moral responsibility to use their influence and status to protect communities most affected by conflict by advocating for nonviolent and diplomatic political resolution. Public health and medical curricula worldwide should include more on the prevention of armed conflict to promote further awareness, research, and capacity building. Further research should focus on quantifying the impact of armed conflict using a broader range of mortality and morbidity health indicators, examine inequalities in impacts masked by country-level data, and identify effective strategies to preserve health systems and health-promoting institutions to reduce mortality during conflict and post-conflict periods. In particular, linkage studies of civilian mortality and morbidity, including quantitative research and victimisation surveys, in conflicts to specific risk exposures e.g. SALW and explosive remnants of war , can drive powerful evidence-based policy-making through national interventions and multi-lateral agreements.
Armed conflict, especially the most intense types, indirectly impacts civilian mortality. Our estimates suggest that almost 30 million civilian deaths were indirectly attributable to armed conflict globally between 1990 and 2017, two thirds of which were due to communicable, maternal, neonatal, and nutritional diseases. Broader and more robust measures of civilian impacts at subnational and national levels are needed to inform policy and advocacy to prevent war and protect civilians. This could include greater use of linkage studies that incorporate data from routine health and demographic sources, exposure to conflict-specific environmental risks, and quantitative epidemiological methods such as national and subnational victimisation surveys.
Availability of data and materials
All data used in this study are publically available and are cited with URLs.
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Iqbal Z. Health and human security: the public health impact of violent conflict. Int Stud Quart. 2006;50(3):631–49.
Carlton-Ford S. The impact of war, adult HIV/AIDS, and militarization on young children's mortality. Bass, L. (Ed.) Sociological Studies of Children and Youth (Sociological Studies of Children and Youth, Vol. 10). Bingley: Emerald Group Publishing Limited; 2005. p. 231–55.
Li Q, Wen M. The immediate and lingering effects of armed conflict on adult mortality: a time-series cross-national analysis. J Peace Res. 2005;42(4):471–92.
Plümper T, Neumayer E. The unequal burden of war: the effect of armed conflict on the gender gap in life expectancy. Int Organ. 2006;60(3):723–54.
Darrouzet-Nardi AF. Nonviolent civil insecurity is negatively associated with subsequent height-for-age in children aged< 5 y born between 1998 and 2014 in rural areas of Africa. Am J Clin Nutr. 2016;105(2):485–93.
Montalvo JG, Reynal-Querol M. Fighting against malaria: prevent wars while waiting for the “miraculous” vaccine. Rev Econ Stat. 2007;89(1):165–77.
Jenkins D, Marktanner M, Merkel AD, Sedik D. Estimating child mortality attributable to war in Yemen. Int J Dev Issues. 2018;17(3):372–83.
Berrang-Ford L, Lundine J, Breau S. Conflict and human African trypanosomiasis. Soc Sci Med. 2011;72(3):398–407.
Berry I, Berrang-Ford L. Leishmaniasis, conflict, and political terror: a spatio-temporal analysis. Soc Sci Med. 2016;167:140–9.
Kelly JT, Colantuoni E, Robinson C, Decker MR. From the battlefield to the bedroom: a multilevel analysis of the links between political conflict and intimate partner violence in Liberia. BMJ Glob Health. 2018;3(2):e000668.
Kinyoki DK, Moloney GM, Uthman OA, Kandala N-B, Odundo EO, Noor AM, et al. Conflict in Somalia: impact on child undernutrition. BMJ Glob Health. 2017;2(2):e000262.
Østby G, Urdal H, Tollefsen AF, Kotsadam A, Belbo R, Ormhaug C. Organized violence and institutional child delivery: micro-level evidence from sub-Saharan Africa, 1989–2014. Demography. 2018;55(4):1295–316.
Sedda L, Qi Q, Tatem AJ. A geostatistical analysis of the association between armed conflicts and Plasmodium falciparum malaria in Africa, 1997–2010. Malar J. 2015;14(1):500.
Chukwuma A, Ekhator-Mobayode UE. Armed conflict and maternal health care utilization: evidence from the Boko Haram Insurgency in Nigeria. Soc Sci Med. 2019;226:104–12.
Wagner Z, Heft-Neal S, Bhutta ZA, Black RE, Burke M, Bendavid E. Armed conflict and child mortality in Africa: a geospatial analysis. Lancet. 2018;392(10150):857–65.
Wagner Z, Heft-Neal S, Wise PH, Black RE, Burke M, Boerma T, et al. Women and children living in areas of armed conflict in Africa: a geospatial analysis of mortality and orphanhood. Lancet Glob Health. 2019;7(12):e1622–e31.
Bollyky TJ, Templin T, Cohen M, Schoder D, Dieleman JL, Wigley S. The relationships between democratic experience, adult health, and cause-specific mortality in 170 countries between 1980 and 2016: an observational analysis. Lancet. 2019;393(10181):1628–40.
Hone T, Mirelman AJ, Rasella D, Paes-Sousa R, Barreto ML, Rocha R, et al. Effect of economic recession and impact of health and social protection expenditures on adult mortality: a longitudinal analysis of 5565 Brazilian municipalities. Lancet Glob Health. 2019;7(11):e1575–e83.
Lu C, Schneider MT, Gubbins P, Leach-Kemon K, Jamison D, Murray CJ. Public financing of health in developing countries: a cross-national systematic analysis. Lancet. 2010;375(9723):1375–87.
Sundberg R, Melander E. Introducing the UCDP georeferenced event dataset. J Peace Res. 2013;50(4):523–32.
Wang H, Naghavi M, Allen C, Barber RM, Bhutta ZA, Carter A, et al. Global, regional, and national life expectancy, all-cause mortality, and cause-specific mortality for 249 causes of death, 1980–2015: a systematic analysis for the Global Burden of Disease Study 2015. Lancet. 2016;388(10053):1459–544.
World Bank. World Bank Open Data [online]. Available at: https://bit.ly/2yUZmdS [Date of Access 12 Apr 2019].
OECD. Where: Global reach [online]. Available at: https://bit.ly/32nmyj4 [Date of Access 16 Oct 2019].
Coppedge M, Gerring J, Knutsen CH, Lindberg SI, Skaaning S, Teorell J, et al. V-Dem Methodology v8. Varieties of Democracy (V-Dem) Project. 2018. https://www.v-dem.net/media/filer_public/5a/f1/5af198e9-f3e8-4619-b9fd-a8387fdc22a5/v-dem_methodology_v8.pdf.
Global Burden of Disease Collaborative Network. Global burden of disease study 2017 (GBD 2017) covariates 1980–2017. Seattle: Institute for Health Metrics and Evaluation (IHME); 2018.
Drazanova L. Historical Index of Ethnic Fractionalization Dataset (HIEF), https://doi.org/10.7910/DVN/4JQRCL, Harvard Dataverse, V1, UNF:6:z4J/b/PKbUpNdIoeEFPvaw== [fileUNF]. 2019.
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Marshall MG. Major episodes of political violence (MEPV) and conflict regions, 1946-2018 [online]. Available at: https://bit.ly/33iGSWr. Accessed 2 Aug 2020.
Beyrer C, Villar JC, Suwanvanichkij V, Singh S, Baral SD, Mills EJ. Neglected diseases, civil conflicts, and the right to health. Lancet. 2007;370(9587):619–27.
Jawad M, Vamos EP, Najim M, Roberts B, Millett C. Impact of armed conflict on cardiovascular disease risk: a systematic review. Heart (British Cardiac Society). 2019;105:1388–94.
Stamatel JP, Romans SH. The effects of wars on postwar homicide rates: a replication and extension of Archer and Gartner’s classic study. J Contemp Crim Just. 2018;34(3):287–311.
Henderson R, Stark C, Humphry RW, Selvaraj S. Changes in Scottish suicide rates during the Second World War. BMC Public Health. 2006;6(1):167.
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Burnham G, Lafta R, Doocy S, Roberts L. Mortality after the 2003 invasion of Iraq: a cross-sectional cluster sample survey. Lancet. 2006;368(9545):1421–8.
Moyer JD, Bohl D, Hanna T, Mapes BR, Rafa M. Assessing the impact of war on development in Yemen [online]. Available at: https://bit.ly/2XsGPE3 [Date of access 03 June 2020]. 2019.
Spiegel PB. The humanitarian system is not just broke, but broken: recommendations for future humanitarian action. Lancet. 2017;S0140-6736(17):31278–3.
Patel P, Gibson-Fall F, Sullivan R, Irwin R. Documenting attacks on health workers and facilities in armed conflicts. Bull World Health Organ. 2017;95(1):79.
Frost A, Boyle P, Autier P, King C, Zwijnenburg W, Hewitson D, et al. The effect of explosive remnants of war on global public health: a systematic mixed-studies review using narrative synthesis. Lancet Public Health. 2017;2(6):e286–e96.
MJ is supported by the Medical Research Council Doctoral Training Partnership. PHPE is supported by the NIHR School of Public Health Research. The funders had no role in the design, analysis or writing of this manuscript, nor the decision to submit for publication. The corresponding author (MJ) has full access to all the data in the study and had final responsibility for the decision to submit for publication.
Ethics approval and consent to participate
This study used publically available country-level routine data sources and as such was exempt from ethical review.
Consent for publication
The authors declare that they have no competing interests.
Springer Nature remains neutral with regard to jurisdictional claims in published maps and institutional affiliations.
Additional methodological details.
First order causes of death.
Second order causes of death.
Differences by sex.
Differences by age group.
Differences by actor type.
About this article
Cite this article
Jawad, M., Hone, T., Vamos, E.P. et al. Estimating indirect mortality impacts of armed conflict in civilian populations: panel regression analyses of 193 countries, 1990–2017. BMC Med 18, 266 (2020). https://doi.org/10.1186/s12916-020-01708-5

domain: medical


text:
Proton conduction in BaxCe0.8Y0.2O3−α + 0.04ZnO at intermediate temperatures and its application in ammonia synthesis at atmospheric pressure
- 267 Downloads
Dense ceramic samples BaxCe0.8Y0.2O3−α + 0.04ZnO (x = 1, 0.98, 0.96, 0.94) were successfully prepared by a solid-state reaction method, whose sintering temperature was about 573 K lower than a traditional solid state reaction method without sintering aid. XRD patterns indicated that all the samples exhibited a single-phase of orthorhombic perovskite structure. The conduction behavior was investigated by alternating current impedance in wet hydrogen atmosphere at 673–1073 K. It was found that the conductivities were affected by the nonstoichiometric amount of Ba, and increased in the order: σ (x = 0.94) < σ (x = 1) < σ (x = 0.96) < σ (x = 0.98). It was also found that the samples were almost pure ionic conductors and contributed mainly by proton and partially by oxide ion in wet hydrogen atmosphere at 773–1073 K. The ammonia synthesis at atmospheric pressure was successfully conducted using an electrolytic cell based on Ba0.98Ce0.8Y0.2O3−α + 0.04ZnO. The ammonia formation rate reached 2.36 × 10−9 mol s−1 cm−2 under the suitable conditions of 0.8 mA and 773 K.
KeywordsCeramic Sample Electromotive Force Cathode Chamber Ammonia Synthesis High Proton Conductivity
This study was supported by the National Natural Science Foundation of China (no. 20771079).
- 9.Tomita A, Hibino T, Suzuki M, Sano M (2004) J Mater Sci 39:2493. doi: https://doi.org/10.1023/B:JMSC.0000020015.80430.d8 CrossRefGoogle Scholar

domain: literature


Controlling antenna rotators
Controlling antenna rotators
Web Radio Control uses Hamlib
rotctld application to control antenna rotators. Hamlib supports controlling rotator controllers
with serial port, USB or network connection.
The configuration for setting up control for an antenna rotator controller is very similar to the configuration of controlling a radio: each configured rotator needs settings for a unique ID as identifier, the Hamlib rotator controller model number and a reference to the rotator controller serial port or a network address (for a network-based rotator controller).
Add an empty line to the end of the Web Radio Control configuration file, copy the following configuration template to the end of the file and follow the instructions below to fill in the configuration template.
rotatorDevices: - id: "rotator1-id" type: "rotctld" rotctld: localInstance: enabled: true model: ROTATOR_MODEL_NUMBER rotDevice: "ROTATOR_DEVICE" serialSpeed: SERIAL_SPEED
Filling in the configuration example for antenna rotators
Replace the placeholders (e.g.
ROTATOR_MODEL_NUMBER) in the configuration example using the instructions below.
Pay attention to preserve any double quotes and spaces around the placeholders.
The configuration file format is called YAML, which is sensitive to the number of spaces in the beginning of each line. Please make sure to not change the number of spaces!
Some of the following placeholders in the configuration file template depend on the rotator controller models in use,
so you will have to look up them in the
List devices listing of the setup user interface.
The placeholders to be replaced in the configuration file template are:
Rotator model configuration
rotator1-id- An identifier for the antenna rotator to be controlled. The identifier is used internally by Web Radio Control to distinguish between antenna rotators and other devices. You can choose the identifier freely, but it makes sense to use the rotator model as the ID. Note that the ID may only contain characters A-Z, a-z and 0-9. Example:
ROTATOR_MODEL_NUMBER- Rotator model number for controlling the rotator. Please find the model number of your rotator controller model from the list of supported rotator models in the setup user interface (see the screenshot below) or from the documentation on page supported antenna rotators. The manufacturers and models are listed alphabetically. Example:
1701(for Prosistel antenna rotators)
An example screenshot of the list of supported rotator models displayed in the setup user interface:
Rotator control configuration
ROTATOR_DEVICE- Rotator controller serial port or network address in URI reference format documented below.
The setup user interface lists all detected serial ports and adapters under title
Serial ports (for device control), see the screenshot below. Find the serial port that is connected to the rotator from the list of serial ports and copy the text from the
URI referencecolumn here. URI references for serial ports have an
id:prefix and network addresses have prefix
The list of serial ports identifies USB serial port adapters with
tty, usbin the
Subsystem, busfield. Rotator models with a USB connector will usually have the rotator model and some form of serial number in the
id:URI reference, although there is no general rule on what the URI references will look like. With multiple rotators of the same model or when using USB — RS-232 adapters it is usually not possible to know which serial port is connected to which rotator, so you will have to test each URI reference.
For rotator controllers with network connection, use URI references with
net:rotator(domain name) or
SERIAL_SPEED- Rotator serial port communication speed (baud rate). Choose a speed supported by your rotator controller model (for rotator controllers with serial port connection only!). Example:
An example screenshot of the list of serial ports displayed in the setup user interface:
An example of finished rotator configuration
The example below contains configuration for a a Prosistel PST-61D antenna rotator and controller.
This rotator configuration is only an example and cannot be used as it is, because the rotator configuration depends on the rotator equipment you use. This example serves primarily as a reference for checking what a finished configuration for controlling a single rotator looks like.
rotatorDevices: - id: "pst61d" type: "rotctld" rotctld: localInstance: enabled: true model: 1701 serialSpeed: 9600 rotDevice: "id:usb-Prolific_Technology_Inc._USB-Serial_Controller_D-if00-port0"


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Camshaft Position Sensor Cmp
New pcm, crank and CAM sensors start but wont.
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Hangö Gymnasium and Hangö Högstadium
The Lundqvist commercial building, designed by Selim A. Lindqvist and the construction engineer Elia Heikel, is considered Helsinki’s first modern office building, for it contained no apartments at all. Both cast iron and concrete were used in its construction, thus minimising the number of internal walls and achieving flexibility. The building is comparable with the work of the Chicago School in the late 19th century. The original cast-iron pillar structure was completely destroyed in a total refurbishment of the interior carried out in 1981, when a new load-bearing frame was installed. The vertical exterior of the building makes reference to the European art nouveau of the time, and to the historicist styles of the late 19th century, particularly the neo-gothic. Lindqvist’s contacts with central Europe, particularly Germany, were perhaps even more important than his links within Finland. The Lundqvist commercial building is, by Finnish standards, an advanced and well-adapted iron construction whose basic idea could not be more highly developed. It brought Lindqvist and Heikel’s collaboration to a natural conclusion and created the foundation for Lindqvist’s prolific career as an architect for industry and business.
Text: Juhana Lahti / 20th Century Architecture, MFA


text:
Who was eliminated leading into the Dancing with the Stars: Juniors finale? Well, be prepared for a surprise.
As it turns out, the ABC ballroom competition (which is still ongoing even though the flagship show has been over for a while) opted to not send anyone home. With that in mind, it means that Ariana Greenblatt, Sky Brown, Miles Brown, and Mackenzie Ziegler are all still left to compete for the grand prize at the end of all of this.
So of that group, who is the favorite? If you’re going on pure ability, it’s easy to say Ziegler — she scored fantastically tonight and from the onset of this series, it was easy to label her the consensus favorite. Of course, she’s also the most polarizing contender given her past knowledge of dance and performance, and we know that for many purists of the show, they find a lot of that frustrating. (Consider this one of the reasons why Bobby Bones won this past Dancing with the Stars season — he was someone who, despite not having the most skill, connected really well with the public. To be fair, though, the Juniors competition is judged differently and this may not matter anywhere near as much.)
The nice thing about this season is that no matter where you really look in the competition, it’s hard to come up with almost any reason to complain. There are great contenders all across the board with a wide array of talents, and this is a show that is so lighthearted and celebratory in nature that it would be extremely hard to be angry about anyone who wins. This is a series designed to be even lighter and cheerier than the flagship version of the show, which has a fairly lighthearted tone in its own right.
Keeping four contestants around for the finale does feel in some ways fitting for the series’ tone, but beyond just that, we do think that there is a larger advantage that the show has here: Basically, it does give the producers a chance to not have to rely as much on filler or push three contestants to the limit. The competition as a whole is just a whole lot easier to manage when you’ve got a greater percentage of people to check it out.
As for the future beyond this season…
That’s something that we’ll have to wait and see on to a certain extent. Nonetheless, we do remain hopeful that there will be more Dancing with the Stars: Juniors, given that it is fairly low-cost and fun as a whole.
What do you think about Dancing with the Stars: Juniors and the non-elimination decision that we had tonight? Be sure to share now in the comments. (Photo: ABC.)


text:
Fairy Tale Dreams - Poem by Moh Ishu
Fairies, witches, pixies and goblins,
Perhaps the dwarfs and Snow White;
Or candies and cookies in silver tins—
Oh my, what a sight!
Maybe once or twice, the hero dies
And tears roll from your eyes;
But, sad it is for the heroine,
Who all her life will sit and cry!
Sometimes a little wild it gets,
With dragons and unicorns—
It's a lot better than our TV sets,
Because it's you on the chariot, by stallions drawn.
Or you'd be the gallant guy,
The villain you'd punch and kick.
Perhaps you'd make the heroine smile,
Oh! How romantic….
Comments about Fairy Tale Dreams by Moh Ishu
Read this poem in other languages
This poem has not been translated into any other language yet.
Still I Rise
The Road Not Taken
If You Forget Me
Edgar Allan Poe
Stopping By Woods On A Snowy Evening
I Do Not Love You Except Because I Love You

domain: artistic


10KVA CNC Gear Chamfering Machine SIEMENS 802D 2-Axis CNC System: Quick Details: This is a 2-axis CNC machine designed primarily for chamfering the shift gears in the gear boxes used for automobiles. ...
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The Nexa NP-1060 is an 15 inch Point of Sale Terminal with a 64Gb Hard, 4Gb SSD Hard Drive & Windows 10 OS. A range of POS Software can be installed onto this terminal including cloud based instead of using a tablet.
The Nexa NP-1060 15" All-in-One Touch Screen POS Terminal with Windows 10 Operating System. It is Fanless and has an Intel J1900 Quad core processor and 4Gb RAM as standard.
Windows 10 IoT Pre Loaded (64 bit)
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4GB RAM - Upgradeable to 8GB64GB
64Gb Removable SSD (Solid State Drive)
4 x USB ports
3 x RJ45 Serial/RS232 Ports
1 x RJ 12v Cash Drawer Port
1 x VGA port
1 x Gigabit LAN
Power Supply concealed within the stand
2 Year Return to Base Warranty (On-site Optional)
Dimensions: 384mm x 324mm x 194mmWeight: 5.8kg
Operating Temperature: 0 - 40 degrees
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Cash Register Warehouse | POS Systems
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text:
NTAGI approves Covovax vaccine for 12 to 17 age group
New Delhi, Apr 29: The Standing Technical Sub-Committee of the NTAGI has recommended the inclusion of the Serum Institute's Covid jab Covovax in the national anti-coronavirus vaccination programme for children aged 12 to 17 years, sources said on Friday.
The National Technical Advisory Group on Immunisation (NTAGI) panel, at its meeting on Friday, also discussed reducing the gap between the second and precaution doses of vaccine from nine to six months and administering the additional dose early (before nine months) to those travelling abroad for education, employment, sporting events and business meetings.
But it could not reach a decision on the matter related to the precaution dose, sources said. The vaccination of children aged five to 11 years was not on the agenda of the NTAGI Standing Technical Sub-Committee (STSC) meeting, they said.
On the Serum Institute's vaccine Covovax, an official source said, "The COVID-19 working group of the NTAGI had earlier reviewed data related to Covovax and okayed it. The NTAGI's Standing Technical Sub-Committee which met on Friday has recommended that the vaccine can be used in the covid vaccination drive for 12-17 years age group."
India's drug regulator had approved Covovax for restricted use in emergency situations in adults on December 28 last year and in the 12-17 age group, subject to certain conditions, on March 9.
Serum Institute of India (SII) Director for Government and Regulatory Affairs Prakash Kumar Singh had written to the Union Health Ministry recently, requesting for Covovax's inclusion in the immunisation drive for those 12 years and above.
Singh had stated that the Pune-based firm wanted to provide Covovax to private hospitals at Rs 900 per dose plus GST and was waiting for directions to supply it to the Centre. However, the price of the vaccine for the government was not mentioned. The country began inoculating children aged 12-14 from March 16. PTI


text:
PEOPLE who install NGV kits without authorisation from the Road Transport Department are breaking the law.
Currently, 28 installers have a certificate of authorisation from the RTD, said Mohamad Dalip, the RTD officer in charge of NGV conversions.
To obtain proper authorisation from RTD, the installer needs to have credentials from the Department of Skills Development (formerly known as MLVK).
Mohamad said RTD was now receiving applications every day from those wishing to be authorised NGV installers. Previously, there was only one every two weeks.
Motorists can cross-check an installer's credentials with the RTD. A list of authorised NGV installers will soon be listed on the department's website.
The installers also need to issue car owners with conversion certificates which they have to take to Puspakom to get a safety check done.
Mohamed said insurance companies did not cover NGV cars damaged in accidents if they were not properly certified.http://www.nst.com.my/Current_News/NST/ ... index_html


text:
- FOSSIL women watch – Round stainless steel case (diam. 32 mm) in polished and brushed finishing – 5 ATM water pressure resistance construction
- Black genuine leather strap with silver ornaments – Buckle
- Silver dial with silver and luminous indexes and arabic numbers at 6/12 – 3 hand movement – Silver and luminous hands – Mineral glass
- Packed in Fossil collector illustrated tin box (various design)
For the au courant of the timepiece world, this impressive Fossil watch does not disappoint. The true focal point of this watch is the bold silver dial and the silver hands, which rest under a quality mineral crystal. All is secured within a 32mm stainless steel case, and the watch is driven by a precise analog-quartz movement. A 50 meter water resistance is made possible by the considerable structural integrity of this timepiece, ensuring a reliable accessory to last for years to come. Additional features include luminous and measures seconds.