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The cold weather may still be biting and the wind blustery this month, but we do keep getting a few days' of respite when the sun shines and it feels vaguely like spring may be coming.
And it's in these milder moments that we're granted a prime opportunity to get out into the garden and put in the leg work that will see our patch bloom into something beautiful come the warmer months. Looking after your garden at this time will also help protect your plants from the frosts that are still likely to hit.
Geoff Hodge from The Scotts Miracle-Gro Company has come up with a diary of jobs to get cracking with this Feb.
Tackle your beds and borders
- If your snowdrops have been growing in the same soil for several years and haven’t flowered so well this year, then it’s a good idea to do something about it. Carefully lift the bulbs – this can be done when they’re still in leaf, or ‘in the green’ – without damaging the roots too much. Split the clumps into smaller sections and replant them at the same depth in soil that has been improved with lots of organic food, such as home-made compost, well-rotted manure or planting compost. Then water them, adding a liquid feed to the water to help them away.
- Dry snowdrop bulbs planted in autumn often don’t grow well because they've dried out too much. Growing snowdrops from plants and planting when flowering has finished, but they’re still in leaf, is usually a better bet. Or better still, buy potted plants. Plants sold in the green are often freshly dug up, so it's vital to water them in as soon as they are planted.
- Deadhead winter-flowering pansies and other winter and spring flowering bedding plants regularly to keep them flowering well. Give them a liquid feed to help improve their chances.
- Sow sweet pea seeds one by one in sweet pea tubes or Rootrainers (available from most garden centres), which allow their roots plenty of room to grow. Fill them with a good seed sowing compost. Then place them in a cold frame, cold greenhouse or conservatory to germinate.
- After a focal point for your garden? Put up an arbour, arch or trellis covered with colourful climbers, such as clematis, wisteria, ivy, honeysuckle, Virginia creeper or climbing roses.
- The young growth of hostas, delphiniums and other perennial plants, can be susceptible to a nasty slug attack at this time of year, so protect vulnerable plants with slug pellets.
- Tidy up herbaceous perennials, removing any dead or damaged leaves plus any dead flower stems that were left on over winter.
Perk up your patio
- Trees, shrubs, fruit and other long-term plants growing in containers add a great touch of colour to any patio. But they will always benefit from topdressing with fresh compost. Scrape away the top 2.5-4cm (1-11/2in) of old compost and replace it with fresh. Mix in some controlled-release plant food into the fresh compost, which will feed the plants throughout spring and summer from just one application.
- Plants that have been in their containers for several years may have become potbound, which may mean they're not thriving as well as they could. These plants need very careful, regular watering throughout spring and summer. If they are already in large pots and you can’t move them into even larger ones, it pays to at least freshen them up by working over the outside of the rootball with a hand fork to remove old and dead roots and old, stale compost.
- Plants may start growing during warmer spells, or even continue growing after the mild winter, so check the compost to make sure it remains moist and doesn’t dry out.
- For a great summer display of colour and scent, plant lily bulbs in pots for a fantastic patio feature.
Love your lawn
This is a great time to create a new lawn, or repair a damaged one, by laying turf, providing the soil isn’t frozen or waterlogged. To ensure you achieve a luscious green lawn to be proud of, it pays to do some soil preparation work first.
- Start by digging over the soil to a depth of 15-23cm (6-9in) and improve it with compost, lawn soil, soil conditioner or similar material and, at the same time, remove large stones, the roots of perennial weeds and other debris. If your soil is very heavy clay, it may even pay dividends to add some sharp sand to the soil too.
- Rake the area flat and level, and then lightly consolidate the soil by walking over the area with your weight on your heels and rake flat and level again. You’re now ready to start laying the turf.
- Lay your first row of turfs along a straight edge. Butt each one closely to the last and ensure good contact with the soil by tamping down firmly with the back of a rake. Lay the next row, again ensuring each turf is pushed right up to its neighbour. Stagger each row in a brickwork pattern. Finally, water the turf well or, better still, water and feed with a liquid plant food to aid rapid establishment.
You can also do this soil preparation if you plan to sow grass seed, but wait until next month to actually do the sowing. This early work is a good idea, as it gives time for weed seeds in the soil to germinate and you can then hoe them off or spray with a weedkiller.
Top tip: In mild weather, at temperatures of 5C (41F) or above, the grass will grow and need mowing. But avoid walking on the lawn if the grass is frozen or wet.
Get going with growing your own
To make sure you grow all the veg you need this year, draw up your cropping plans and order or buy your seeds. When you get your veg seeds, make a month-by-month seed sowing organiser in a cardboard box or similar. This way you won’t forget to sow everything at the right time. Store the seeds in cool, dry conditions until you’re ready to sow.
- At this time of year there is an almost irresistible urge to start sowing seeds outside. But, unless your soil is very well drained and you live in a sheltered mild district, it is far better to wait until spring. Most vegetable seeds that are sown outdoors won’t germinate until the soil temperature reaches 7C (45F) and if sown in cold soil will rot.
- But you can get ready for an early start by covering beds with cloches or sheets of clear polythene. This warms the soil, keeps clay soils from getting too soggy during wet weather and encourages weeds to germinate. These weeds can then be dealt with before sowing to produce a ‘weed-free’ veg bed.
- Make sure you buy your seed potatoes as soon as possible, as the most popular varieties soon sell out. You can then start chitting early seed potatoes, which generally produces a bigger, better crop as a result. Chitting means getting them into growth to produce strong young shoots before planting out. Stand the seed potatoes upright in trays or egg boxes with the ‘eye’ end (the end with the most eyes or buds) uppermost. Keep them somewhere cool but frost free, and in good light.
And when it comes to fruit, this is a great time to plant fruit trees and bushes, providing the soil isn’t waterlogged or frozen solid. Planting bare-rooted fruit needs to be completed by the end of the month.
- When planting, dig over the planting area and make sure the soil is well prepared with plenty of added organic matter – such as well-rotted manure, garden compost or a tree and shrub planting compost.
- Water the roots thoroughly before planting. Plant at the same depth that the plant was originally growing and carefully firm the soil around the roots.
- Trees will need to be staked with a good tree stake and secured with two tree ties.
- Towards the middle or end of the month, give all fruit a good start to the year by applying a granular balanced plant food. This should be applied over the main rooting area – roughly equivalent to the spread of the branches.
- Prune autumn-fruiting raspberries by cutting down all the canes to the ground. Summer raspberries can be cut back to one or two buds above the top of their supporting wires if they’ve grown too tall.
Top tip: Protect the blossom of early-flowering apricots, peaches and nectarines from frost damage by covering plants with fleece. But remove it on warm days, especially when pollinating insects are about.
General gardening jobs
- Keep your garden clear of diseases by raking up and destroying any badly-infected leaves (just burn or bin them).
- Garden birds will still rely on us to put out food for them and they are particularly in need during cold and freezing weather. So top up bird feeders as necessary. And don’t forget to ensure there is a supply of unfrozen, clean water. Bird tables should only be stocked with enough food that the birds can eat without leaving excess from day to day.
- When gardening on wet clay soils, work from a plank of wood, board or similar, rather than treading directly on the soil. This will help spread your weight and help avoid compacting the soil.
There's more gardening jobs in every issue of Yours

domain: agronomy


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Number of Pages: 221
Publication Date: 2003
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Culture Shift: Transforming Your Church from the Inside OutRobert Lewis, Wayne Cordeiro, Warren BirdJossey-Bass / 2005 / Hardcover$22.99 Retail:
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Winning on Purpose: How to Organize Congregations to Succeed in Their MissionJohn KaiserAbingdon Press / 2006 / Trade Paperback$13.49 Retail:3.5 Stars Out Of 5 3 Reviews
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Raising a Modern Day Knight: A Father's Role in Guiding His Son to Authentic Manhood - revised editionRobert LewisTyndale House / 2007 / Trade Paperback$9.79 Retail:5 Stars Out Of 5 26 Reviews
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A city set on a hill, inescapably visible. A lit lamp brilliantly illuminating the darkest room. With compelling metaphors, Jesus described the church and its impact on the world. And indeed, filled with the Holy Spirit, the early church demonstrated a spiritual energy and depth that transformed the surrounding culture. Dont let your church settle for less! Using bridges as a metaphor for "irresistible influence"--or i2, as he calls it--Robert Lewis shows how your church can become a strong, well-traveled link between heaven and earth in your community. In this engaging and uplifting book, Lewis tells the stories and shares the experiences and lessons of Fellowship Bible Church to show * what it will take to reconnect your church with your community * the how-tos of "incarnational bridge building" * true stories of i2 in action * how to expand the i2 effort through new partnerships and adventures * requirements of the church in the 21st century Discover how the power of incarnational bridge-building can impact your church and your community at the annual Church of Irresistible Influence conference. For information, contact Fellowship Associates: www.fellowshipassociates.com; phone (501) 975-5050.
Rob Wilkins is a freelance writer who wrote "Taking the Child's Way Home" and, with various authors, seven other books. He received the Robert F. Kennedy Journalism Award. He lives in Etowah, NC.

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Providing social services for children with disabilities and other children suffering exclusion
Our model is centred on the child, their families and the community, and provides holistic care to children at day care centres.
Many of the 20,000 children with disabilities in Kosovo experience exclusion, stigma and isolation in their everyday lives.
Despite developments in the provision of social and family services since the end of the war, these are still woefully inadequate. Our day care centres fill urgent gaps in social services and have improved development opportunities and life skills for children with special needs. Psychosocial, education and recreational activities have enabled children to develop their skills, while gaining confidence and self-esteem.
Meet Bujar, who attends a daycare centre in Kosovo.
- Provide high quality support for children with disabilities, children facing social exclusions and children affected by domestic violence
- Equip vulnerable children with the skills they need to navigate and integrate into mainstream society
- Support parents’ to learn more about disabilities and empower them to fight for their children’s rights
- Improve Kosovo’s acceptance of and services for children with disabilities
- PEMA staff organize individual and group work to help the children maximise their potential
- Parent-support groups have been set up
- National activities such as open days, special events and national roundtables, increase awareness among the wider community on the needs of vulnerable children and helps to reduce stigma around disabilities
- Expanded the number of day care centres from two to four
- 320 children attend day care centres each month
- Over 650 children and adolescents received care in four day care centres over 2 ½ years
- 350 parents benefited from support groups
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In sub-Saharan Africa 70% of 15-19 year olds don't have comprehensive HIV knowledge. £35 pays for a young person to learn how to protect themselves and master other vital skills. Visit our website to donate now! onetoonechildrensfund.org/wha… #hivawareness #sustainabledevelopment pic.twitter.com/R9ihPNGdQD
In October 2018, it was reported that 12,445 community members were benefiting from our enable services. Help us continue to grow our reach! #socialimpact #hivawareness #mentormothers #healthcare pic.twitter.com/z0exzGmtRl
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domain: sociology


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Cut the carbs (and gluten!) by ditching the noodles and using zucchini and yellow squash instead.
Grilled Vegetable Lasagna1-2 small zucchini
1-2 small yellow squash
1 jar or 4 cups marinara sauce
1 pound ground beef (optional)
6 ounces ricotta cheese
2 cups shredded mozzarella
Slice zucchini and squash about 1/4" thick. Salt and set on paper towels. This will help remove some of the moisture so your lasagna isn't soggy. Meanwhile if you are using ground beef, cook it and add marinara sauce to it.
Grill zucchini and squash over medium heat until soft. If you don't want to cook them completely you will need to cook them longer in the lasagna. Be careful not to cook so long they burn. I did not add oil to them but you could brush them with olive oil if you like before cooking.
Layer in 8x11 or 9x13 pan so that bottom is covered.
Layer with meaty marinara sauce.
Next add 1/2 of the ricotta cheese.
Sprinkle on 1/2 cup mozzarella cheese.
Add rest of slices. I try to go the opposite direction of the other slices and fill in where needed. If you have too many use them for another meal.
Sprinkle with 1/2 cup mozzarella and add the rest of the ricotta (not shown in picture.)
Add rest of marinara sauce.
Sprinkled rest of cheese on top. Bake covered in 350 degree oven for 30 minutes.
Remove foil and cook for another 10-15 or until cheese is brown. Remove from oven and let set for 10 minutes before cutting.
Serve sprinkled with Parmesan Cheese.

domain: food


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The Contessa Corner
A forum for Contessa owners, sailors and dreamers.
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Posts found: 12
Zodiac 8' dinghy (2003)with towing harness, "Cadet FR240". Used less than 10 hrs, mint condition. Cost in 2003 $2398. selling for $1900.00.
Yamaha 2.5HP 4-stroke outboard, model F2.5. Used less than 10 hours, in mint condition. Cost in 2003 $978. selling for $750.
Willing to sell both for $2500.00
"Latvis" sail #339. Excellent condition with sparkling brightwork. Burgundy hull with matching dodger (2005), cockpit awning(2005), cockpit and interior cushions. 8HP Bukh diesel. Harken furling with 4 headsails. Examples of upgrades and additions include Cape Horn self steering system(2005), autopilot (2004), traveller(2004), AGM batteries and Xantrex smart charger(2004), 3-bladed prop, and much more. Currently in Tobermory Ontario. A greatly admired boat and a rare find at C$35, 000. For a more detailed description email Andris at: firstname.lastname@example.org
What size of photo/files do you prefer to have us submit to the photo gallery ? Is there an ideal or preferred size in terms of pixels (pixels x pixels) or some other constraints?
Check out the "Sanipottie" models by Sealand. I installed one of their smaller models (#964?) and its working out well. Freed up a lot of space compared to a plumbed-in system with holding tank.
Last year I replaced the whole works (drive shaft, cutlass bearing and the brass sleeve that houses the bearing) I did this because I suspected that the excessive engine vibration that i was experiencing was due to a slightly bent shaft or some other problem with shaft, bearing, sleeve or all of the above. There is no easy way. I had to remove the whole works, brass sleeve an all. It had to be done by brute force. With the prop shaft out (of course!), I took the biggest pipe wrench I could find, clamped it on to the brass sleeve and removed and discarded the whole works. Replaced it with a new sleeve and bearing and epoxied the sleeve into place. The epoxy work was done after prop shaft, bearing and sleeve were all carefully aligned of course. It will be an even bigger and tougher job if I ever have to remove that sleeve again. And yes, it solved my vibration problems. The old Buhk now runs as "smoothly" as any happy one-lunger can, which is to say that it only appears as if it is trying to throw itself overboard.
I am replacing my old VHF with a new unit. The plan is to use the old unit as a station in my place in Tobermory which has an old style TV antenna atop a 30+ foot tower. Question: Can the old TV antenna (VHF?) be used to receive marine VHF signals in the frequency range that we use?
I am also on the lookout for a dinghy that will fit (while fully inflated and ready to go) on the foredeck of a CO26. I see that the smallest Bombard (their AX1) measures 6' - 7" long by 45" wide and weighs in at 33lbs but I do not know if it will fit on the foredeck when fully inflated. They go for about $1000 new. I am unable to check the fit because right now my Contessa is in Tobermory ON and i live in Toronto. Anyone with experience with the Bombard AX1 or other make that size?
I would be more inclined to anchor the preventer to a block and loop slider mounted on the track rather than to a stanchion but given that I have no experience with ANY sort of preventer rigg, I am open to any and all suggestions!
I would like to hear from anyone with experience of installing a preventer: hardware, sheets, attachment points etc. Any other relavant comments would be much appreciated.
I would be interested to hear about the distances that a Contessa 26 might be expected to cover in a day of sailing in "fair" winds. I am aware of the many variables involved, but I would still like to hear about your experiences with distances covered.
Looks like my question regarding the "Voyager" windvane was too specific. I would be most interested to hear about ANY windvane in use on a Contessa 26. I am planning to equip my Contessa with a windvane but I have not been able to find any evaluations of a vane in use on a Contessa, aside from the Monitor used by Tania A., and testimonials provided by vendors.
I would be most interested to hear from Contessa 26 owners who have any experience with or knowledge of "Voyager" windvanes.
Posts found: 12


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What does this mean and what’s the longshore investing angle?
- We can finally settle the “debate” about industry employers being fat-cats flush with cash. This is not a healthy industry. Healthy industries are rising tides which, pardon the pun, don’t let ships sink. Despite the constant negative newsflow about the finances of shipping companies worldwide, this the-employers-are-so-rich claim has been a constant refrain. Let’s put that to rest.
- Consolidation will continue. All signs point to Hyundai picking at the healthy Hanjin pieces while other weak hands are likely to give way to takeover offers, allowing the few strong operators (Maersk, MSC and CMA-CGM) to be left with more power as an effective oligopoly. It will be much easier for a small, concentrated committee of employers to remain coordinated in the fight against labor
- While it’s not easy to see right now, a Hanjin bankruptcy is a reminder to each remaining surviving shipping line and terminal operator that liabilities are very real, and they carry consequence. What else is a liability to these employers? A union pension. It’s even more important now for holders of those pension benefits to further ratchet expectations lower as it would be unwise to believe one industry can forever survive what is the slow eradication of pension promises nationwide.
Summary: Hanjin’s troubles are a reality check for all parties involved. Remaining employers will eventually have stronger bargaining power, with cross hairs set on financial liabilities like pension plans. These events are transpiring amidst a major economic expansion – the BLS reported the 78th straight month of job growth today – but it’s a scary thought to imagine industry health if we were slowing or already in an economic recession.
It’s now up to members to react accordingly, which means making sure their individual financials are in order in case overall industry deterioration does continue. The time to plan for unknowns is when things are going relatively well, not when they’ve already turned bad. (If only there was a resource to learn about how the longshore 401(k) plan is designed to hinder those efforts. Or maybe even a calculator)

domain: finance


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Genital herpes virus is one of the most usual sexually transmitted conditions globally, it impacts about TWENTY PERCENT of the people. It is recommended bear in mind, that there's yet no hsv cure at this time. It is difficult to http://www.ashasexualhealth.org/stdsstis/herpes/signs-symptoms/ find out the actual amount of people who may have herpes virus, although this quantity is elevating significantly each year. The hottest most interesting stories relating to herpes that came out last week.
Physical distress caused by herpes symptoms is not the only one ıssue for individuals that have been having herpes simplex virus. A sense of shame, aggravation and continuous thinking that most of their love life is at a a conclusion are examples of the commonest things that many people, that have recently found out they've herpes, experience. Most people begin to feel like outcasts, and that's a major problem. When it is all said and done, herpes virus is just a skin condition. It is much like eczema or pimples; annoying to manage but completely controllable. I do not think a person with herpes virus needs to experience disgusting or less charming. The fact is, in case you have must cope with herpes simplex virus, you are a bigger, smarter one who knows the internal operation of Cure For Herpes Over The Counter their body and mind. Certainly nothing may be more charming than that.
Because there is no FEDERAL DRUG ADMINISTRATION authorised herpes virus cure, some people are likely to try any sort of method, and occasionally apply un permitted medical remedies that can result in terrible outcomes. It is essential to apply sound judgment and talk about any remedies with your health specialist to ensure that the selected options will not provoke even more problems.
To illustrate, some alternate wellness authors on the Internet have interpreted case study to show that bleach is safe to try for healing herpes virus if the chlorine bleach is diluted, or chlorine bleach is added to bathtub water.
There are many scientific tests that demonstrate that healthy and balanced lifestyle and clean ways of eating will help stop herpes simplex virus breakouts and help to make the way you live normal, so that you will not experience any kind of herpes symptoms and outbreaks and even would't need to take medications. However you have to consider, herpes virus may stay dormant inside the nerve system cells for many years, and come back again as soon as the immune system is weakened.
Consider plenty of alkaline foods. Consuming alkaline foods and nutrients may help to improve the defenses of the human body which has a lower quantity of acidity. This is the hypothesis behind the pH level of our bodies. Organic, uncooked vegetables and fruits, whole grains, legumes and premium health protein rich meats for instance , fish and 100 % natural chicken breast can assist to make an alkaline condition in the your body.
Many analysts believe, that herpes been around in the Greek civilization more than 2200 years ago. Natural systems of medication, like Ayuverda, have already been used to treat herpes virus for years and years.
Ayurveda treatment concentrates on providing vitality and balance to your entire system. Because the herpes simplex virus cannot be erased from your body system soon after it is developed, ayurveda care for herpes virus works to regulate breakouts by supporting agni for a healthy defense mechanisms. Strong agni enables your body to absorb nutrition, assimilate emotional states and get rid of harmful toxins.
Plenty of general practitioners believe, the fact that Ayuverdic way, healthy diet and life style can easily remove herpes simplex virus symptoms and signs (simply by improving defense system and, as a result, suppressing herpes virus), but they also cannot get rid of hsvcompletely from body system. But the truth is, there were a lot of herpes cure analyses that has confirmed that certain changes in way of life can completely eliminate potential herpes simplex virus flare-ups. Even so, it is important to know, that no matter if a man or woman doesn't have any herpes virus signs of illness, she or he can still be infectious to others (via sexual or, in some instances, skin area contact named herpes virus shedding).
herpes virus shedding appears each time an infected woman / man has little spaces in the pores and skin through which the herpes may be pass on. The herpes virus is mostly referred to as a std . Therefore, intimate contact is among the ways the herpes may be pass on. Men or women become infected with the herpes virus once afflicted by the areas of broken skin area exposing the herpes.
As scary as herpes simplex virus might appear at this time, you should keep in mind there are ways to eliminate hsv symptoms and control - or perhaps prevent - breakouts. Starting a good way of life is a great idea, although you should check with your physician before making any extreme shifts with your way of life and trying any alternative herpes virus cure. Bear in mind you aren't all alone and every 6th person around the world is going through the same situation. Stay calm and don't forget to take pleasure from your life!
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domain: medical


text:
Trailer of ‘Force 2’ garners 10 million views
Mumbai, Oct 8 (IANS) John Abraham starrer “Force 2” trailer has already garnered 10 million views on YouTube within nine days of its release.
The three minute, five second long trailer was released on September 29 and has been viewed by 10,306,575 individuals as noted on Saturday.
Produced by Vipul Amrutlal Shah, the film has John and actress Sonakshi Sinha, who are sharing screen space for the first time in the sequel of 2011’s “Force”.
The makers of “Force 2” have dedicated the trailer to the heroes who have sacrificed their lives for the nation. A special slide has been added to the trailer to salute the unrecognised martyrs of our nation.
“As a producer of the film I am elated. One year of hard work, the pain we took, the injuries artists suffered, the stress we took to give the quality is all worth it today. I think people are connecting with the honesty of the entire team,” Shah said in a statement.
Directed by Abhinay Deo, the film has been co-produced by Shah, JA Entertainment Pvt. Ltd. and Viacom 18 Motion Pictures.

domain: news


While studying at Codarts, the prestigious conservatory of Rotterdam, Céline Bishop discovered, randomly, the voice of Luis Rigou, and was astonished by its unique texture. After moving to Paris, she asked him to become her tango voice. Luis Rigou, whose only experience with tango was as the flute soloist of the Cuarteto Cedron, was at first skeptical, but after a few tryouts agreed to accept the challenge.
After having recorded their first album (released in November 2019), the duo appears in concert with a variety of musical combinations.
As a duo, Céline Bishop at the piano and Luis Rigou, singing. For the “historical” tangos, Luis also plays flutes, mainly transverse, but also a historical “Buffet Crampon” from 1862, with ebony rings, representative of the flute played in tangos from the “Guardia Vieja”.
Tango Secret can also be played in a quartet setting for concerts, with a bandoneon and a double bass, or even as a quintet with the addition of a saxophone.
In their new musical show, they are accompanied by musicians and by Los Guardiola, an extraordinary duo of mime and dance, with the stage direction of Coralie Zahonero from the Comédie-Française and Vicente Pradal.
Céline Bishop, who began playing the piano at the age of six and devoted herself to classical music for many years, discovered the world of Argentine Tango in 2009. It’s at «Tango de Soie» (Lyon) that she took her first steps as a dancer and played her first Tango concerts.
In 2012 Céline set out to study for a Bachelor’s degree in Argentine Tango at Codarts Conservatory, in cosmopolitan and exotic Rotterdam, tutored by Gustavo Beytelmann and Wim Warman, performing all over the country with the well-known Gran Orchestra Típica OTRA.
In 2016 Céline settled in Paris and returned to teaching at the Conservatory of music in Puteaux, while continuing her own studies at Gennevilliers with Juan José and Juanjo Mosalini. Driven by the multitude of Parisian musical encounters, she plays in several ensembles such as La Grossa, Orchestra tipica, Calambre (Tango quartet), and Barco Negro (Fado Trio).
Luis Rigou, while studying music at the National Conservatory of Buenos Aires, played with Jaime Torres and also with Anibal Sampayo. Later, he created the ensemble Maiz, with whom he was awarded the Revelation prize at the Cosquin Festival in 1987. He performed throughout all of South America, as well as in 13 European countries, where he settled in 1989. Invited to France to be part of the Cuarteto Cedron as a flute player, he collaborated with many other artists: Luis Naón, Ricardo Moyano, Minino Garay, Gustavo Beytelmann, Antonio Agri, Nilda Fernández, Sergio Ortega, and the Idan Raichel Project.
However, it was under the name of Diego Modena and his album Ocarina, recorded in 1992, that Luis became world famous. Ocarina was No. 1 in the Hit Parade in 14 countries, including France, and in the Top 10 of 44 countries. He was awarded 57 golden, platinum and diamond discs.
He subsequently recorded 18 other albums, taking on the artistic direction of Luis Llach and in 1995, recorded the famous Complainte de Pablo Neruda with French singer Jean Ferrat. In 1996 he began his long collaboration with Vicente Pradal for Cantique Spirituel, Llanto por Ignacio Sánchez Mejías, Peleas y Melisanda, Vendrá de noche and currently Medianoche.
Additionally, he composes and performs film music (institutional, short and long movies) including: Karim et Sala, by I. Ouedraogo, Special Prize at the Cannes Festival 91, Voleur d’enfants of C. de Chalonge with Marcello Mastroiani, and with E. Makaroff and H. Arntzen, he won the Fondation de France prize at the Biarritz Festival for the film music Médecins du Monde in 1997. In 2004, he composed and recorded Cayetano et la Baleine, a CD-book for Gallimard Jeunesse, which was highly successful.
Currently he performs throughout Europe as a soloist with the ensemble La Chimera in Misa de Indios, Misa Criolla, with the Coro Polifónico de Pamplona and Gracias a La Vida.


And another thing. Enough with this bullshit description of Foley's messages as "over-friendly". Ann Coulter, on that other schmuck Bill O'Reilly's show on Tuesday, downplayed the severity of the messages, several times saying that Foley asking a boy what he wanted for his birthday was no big deal. Well, for anyone who hasn't had the opportunity to view the text of some of the more vile exchanges, please go to this link. Masturbation, hand-jobs, lotion, ejaculation...it has it all. And people like Coulter and other Republican assholes really need to shut the fuck up, because this goes way beyond "over-friendly", and for that uber-bitch Coulter to downplay it and decribe it as something innocent should be grounds for enforced muzzling.


Search Results for Tag: ecosystem
Exploring the North – Further North
Arriving in Tromso, Norway, we are now located just at the Arctic’s doorstep. It is a quiet strange feeling, when you suddenly stand at the edge of something, that is normally so incredibly far away. If it’s far away from you as well and you derive from that that you don’t have to care – hang on. You actually should, as the Arctic often is referred to as earth’s air conditioner. And on hot summer days without (working) air conditioning, you feel perfectly well what it is good for.
If you look at such pictures of the Arctic region it does not at all like an air conditioning. I was actually suprised that it can be that green in the Arctic region, as I always associate of snow and ice with Artic. What about you?
But this ice is only there in winter time – that’s when it fulfils it’s cooling job, by reflecting 80 percent of sunlight back to space which then cannot heat up the ocean or earth anymore. But from March on, ice starts melting due to increasing temperatures. The decreasing sea ice cover uncovers the dark oceans that absorbs the sunlight instead of reflecting it – the cooling air conditioning turns into a heating facility.
So far, the Arctic yet hasn’t been completely ice-free during the warm summer period. But you might remember scientists worrying last summerabout the small area that is left ice covered: The larger the ice free part, the more heat is absorbed by the ocean underneath – in turn, even more ice melts and a vicious circle starts.
Not on the edge of the Arctic, but on the edge of Arctic research, you find scientists working on how this influences the Arctic environment. Sometimes this influence can even be positive: Researchers recently found that microbes in the soil can fix more carbon in a warmer climate.
But yet, researchers cannot tell how Arctic ice cover will evolve, but they work for example on better models to predict what is going to happen. That is especially important, as many earth systems are connected: A warming Arctic sea also affects ocean currents for example, that – connected to each other – span over the whole planet. In turn, oceans take up less CO2 for example, and in consequence more of this greenhouse gas in the air, warms up the whole planet.
DateApril 21, 2013
What is Nature Worth?
For the first time, we have a comprehensive idea of nature’s financial worth: the National Ecosystem Assessment
carried out an intensive study of the UK’s natural environment and how it benefits Britain’s economic prosperity. The result: nature is very important to the UK. According to the study, nature provides Britain with tens of billions of pounds in services every year – all for free.
Hundreds of experts in economics, social sciences and ecology worked together to assess different aspects of the UK’s ecosystems across a series of habitats, like woodlands and cities and farms. Those ecosystems provide services like water purification and crop fertilization that are extremely important to an economy, even when we don’t realize it.
The environment ministry says the study will help shape the UK’s environmental policy. But it’s also important to stock of the state of Britain’s natural wealth: 30% of the natural services they studied are suffering because of climate change.
What is nature worth in your country?
DateJune 6, 2011


LAS VEGAS, NV
JANUARY 5-8, 2017
XIAOMI the Chinese smartphone maker will hold a press conference at CES 2017 in January 5th at the Venetian, Venetian Ballroom C from 11:00 to 11:45 hours.
XIAOMI will present a new product at the event.
The Korean site ETNEWS is confirming that Samsung will introduce the Galaxy A8/A8+ 2018 at CES 2018 event in Las Vegas Jan 9-12. T...


Reinterpretation of a French jacquard from the 18 th century. Printed on linen. This loosely interpreted damask has a fluid watercolor effect. The printing technique creates a mottled Fortuny–like spirit. Printed in Italy at the famous Ratti fashion house. This article is available in 4 colourways.
The inset picture show a part of the full width of: 140 cm
The colours shown are not binding.
|Horizontal repeat:||67 cm|
|Vertical repeat:||68 cm|


Eastbay Sleeveless Poly Jersey - Men's Suit up in the Eastbay Sleeveless Poly Baseball Jersey. This 100% polyester knit baseball jersey features moisture management technology which wicks sweat away from your body, leaving you cool and dry during the pressure moments of the game. This jersey is great for customization and gives your team the winning look. Join the team with the Eastbay Sleeveless Poly Baseball Jersey. Imported.


|MAXDS(4)||Device Drivers Manual||MAXDS(4)|
maxds* at iic?
maxdsdriver provides support for the Maxim DS1624/DS1631/DS1721 temperature sensor. The sensor possesses just one accurate temperature sensor; the value is made available through the sysctl(8) interface.
maxdsdriver first appeared in OpenBSD 3.9.
maxdsdriver was written by Theo de Raadt <firstname.lastname@example.org>.
|July 16, 2013||OpenBSD-current|


text:
Genetics, Immunology, and Covid-19: A Conversation with Dr. Robert Green
Steven Cherry Hi, this is Steven Cherry for TTI/Vanguard.
The world has been concentrating on the epidemiology of the novel coronavirus and Covid-19, and with good reason. We need to know how far and how quickly the virus spreads, and which areas might safely come out of hibernation sooner rather than later.
But ultimately many of the most important questions lie at the level of the individual person.
Why does the disease overcome some people while it leaves others without symptoms entirely? What’s the role of co-morbidities such as obesity, diabetes, and heart disease? How much does it have to do with our immune systems—is there a perfect level of response, attacking the virus vigorously, but not too vigorously? Ultimately, are there genetic differences among people, such that we could predict who will likely get the virus, and at what severity, by looking at their DNA?
And finally, testing and immunity—does getting the disease render you safe to others who haven’t had it yet? Does it shield you from getting it again? Forever? Or could it return in a different, maybe worse form, the way chicken pox comes back as shingles? And what role can the study of genetics play in developing tests and a vaccine?
My guest today won’t have all the answers, but he is a great person to ask the questions of.
Dr. Robert C. Green is a Professor of Medicine at Harvard Medical School, a medical geneticist, and a physician–scientist, who directs the Genomes2People Research Program at Brigham and Women’s Hospital, the Broad Institute, and Harvard Medical School. His M.D. is from the University of Virginia, and he has as well a master’s degree in Public Health, specifically Epidemiology, from Emory University.
Robert, welcome to the podcast.
Robert Green Thanks, Steven. It’s great to be here.
Steven Cherry Robert, your area is preventive genomics and you’re a clinical geneticist. Those seem like two different things.
Robert Green Not so different for me, in that—but you’re right, the science of clinical genetics has been concerned with super-rare diseases in the past and mostly with making a diagnosis. So the classic patient is a child who is identified to have something unusual and the geneticist is called in to try to make a diagnosis, maybe help the family avoid having a second child with the same condition. But you know, genomics has exploded in every domain of medical science. And one of the areas is the degree to which genomics is going to allow us to anticipate, predict, and prevent even common diseases, all sorts of cancers, all sorts of cardiovascular disease, dementias like Alzheimer’s disease. And this brave new world is a world where we’ve been calling preventive genomics. And that’s what I’ve been focusing on most recently.
Steven Cherry Very cool. So what do we know about the genomic structure of SARS-CoV-2?
Robert Green Well, keeping in mind that I’m not a virologist or an immunologist, but I have been looking into some of this because so many people are fascinated and asking questions. We know that this is a particular kind of virus. We know that it is the coronavirus family, everybody’s heard. We know it’s different from the influenza virus that we’re familiar with in that it’s more contagious, it has a longer incubation time, and therefore, it’s got a much higher community-attack rate. And we also know that there is tremendous heterogeneity in terms of how people are impacted. You can have people who literally have no symptoms whatsoever with the infection, to people who have multi-organ systemic failure or two people who, of course, are overcome by the disease and don’t make it. So that’s one of the fascinating things about this, is how broad the symptomatology can be.
Steven Cherry Yeah, it, I guess, lodges in the lungs the way a lot of other infectious diseases do, but it seems to be able to ravage the entire body from head to toe.
Robert Green That’s right. It’s got a particular propensity probably through infection to come in through the nasal epithelial cells and the respiratory cells. But then, it’s very clear, as this has developed, that there are people who have different degrees of liver failure, different degrees of kidney failure, that have G.I. problems, that have brain problems—everything from stroke and seizures to just sort of the confusion that you would expect with such a severe illness—has conjunctivitis and inflammation in the eye membranes, people ... Back to the nose, people can lose their sense of smell, and not so long ago, really a surprising recognition that there can be people who have carditis, an inflammation of the heart itself with the virus. So it’s really got a protean set of presentations in addition to the obvious very severe pulmonary symptoms that people are aware of.
Steven Cherry It’s also been surprising people in terms of factors that might contribute to susceptibility. I mean, I was surprised to hear that blood type might contribute. And what else are we learning?
Robert Green Yeah, that’s right. I think, you know, it’s really important to sort of separate underlying medical vulnerability. And just to reiterate those that you mentioned before. Age seems to be the number one issue. So older people are at greater risk almost regardless of underlying disease and vulnerability. But then there’s a clear gradient where people with lung disease, diabetes, hypertension, immune deficiencies of various kinds, are at increased risk for severe symptoms and death. So that’s sort of the diseases that they have. But if you set that aside, you just sort of take a population of ostensibly healthy people, middle aged or even younger. Some of them, as we said, are getting really severe symptoms and some of them are getting very mild symptoms. And that’s really mysterious. Why is it that some of them are getting such severe attacks and others are doing so well?
Now, people been looking, well, I should just step back, Steven, and say the scientific community, like the frontline medical community, has reacted to this whole pandemic with unbelievable intensity, commitment, collaboration, cooperation and speed. I’ve never seen anything like it in the decades of my scientific career as the intensity with which scientists are working together and they’re working with within whatever domain they can. So in my domain of genetics, we’ve been looking at all the different possible reasons why some people could be more vulnerable than others, what you might call host vulnerability. Because we’re the host for the virus. And as you said, there is some early indication that blood type—that was one of the first indications that certain blood types might be more vulnerable. So, for example, studies from China and New York City were suggesting that a blood type—A as in “apple”—might increase susceptibility while O blood type—O as in “olive”—might be protective. And so that was sort of a very crude genetic distinction between different people.
And then there were other clues. So, for example, we’re pretty sure that the little spikes on the virus attach themselves to a specific protein called the ACE2 receptor. And that particular receptor, like all proteins, it has a genetic code. And it turns out that there is at least 17 different variations in humans of that genetic code. So we’ve already got some early evidence that two of those variations are more resistant to the virus hooking on to your cells. Now, in and of itself, that’s not terribly useful information. It’s not like we’re going to go and identify a few people have that variation, say, hey, it’s OK for you to go outside.
But there are parallels to, for example, HIV virus there and clues like this can help accelerate the discovery of treatments and vaccines. So you may remember in the HIV world that the CCR5-delta 32 variation that’s quite rare was also protective in the same way for HIV people. The virus couldn’t really hook on to people’s cells if you had that variation. And that’s really spurred research on HIV vaccines. And although we don’t have any yet, it’s a starting point. And then by the same token, these kinds of genetic variations might be clues on which the whole world could build some rapid progress for vaccines and treatments.
Steven Cherry You mentioned this, the intensity and speed with which scientists are working together on this, and I guess you mean globally. You’ve been involved in something called the Hosts Genetics Initiative.
Robert Green Yes, that’s right. That’s right. This is an initiative that was first organized from groups at the University of Helsinki and here in town at the Broad Institute. And it bears mentioning that, you know, people have generously donated their DNA to DNA biobanks all over the world, including our MassGeneral–Brigham biobank here in Boston, that has already over one hundred and twelve thousand people who had volunteered and who donated DNA and a subset of those we’ve already genotyped and sequenced. And what’s happening is that collections of biospecimens and collections of DNA that exist in multiple locations all over the world can be leveraged to try to find rapid answers around Covid.
The way it works is this in our health care system, in our MassGeneral–Brigham health care system, you know, these hundred thousand people, a certain percentage of them are getting sick with Covid, and that’s being put down in their medical records. And this DNA analysis we’ve done in the past is already linked to their medical records and they’ve already given broad consent for use of this information in scientific research. So we can quickly find the people who had severe symptomatology and, for example, compare them to the people who had mild symptomatology.
Now, even with one hundred thousand people in our Boston Biobank, it’s going to take us a while to get to the numbers we need to look at this with validity. So what do you do? You coordinate our biobank with somebody else’s biobank and somebody else’s biobank. And these colleagues around the world have just sort of self-aggregated into this remarkable consortium that is aggregating over 600 investigators, over 150 different teams, to try to pull these data together quickly. And so, again, I’m not exaggerating. I’ve never seen anything move this fast. I’ve never seen institutional review boards review protocols so fast. I’ve never seen people so unselfishly dedicate their staff. We have staff that we’ve pulled off of other funded projects and have put on these projects, as has everybody else. It’s pretty remarkable.
Steven Cherry So I guess I guess in part the goal is to create a common framework so that data could be more easily shared and compared, and aggregated, and disaggregated. It’s interesting to me that this large international group did its work using things like Zoom and Slack and Google Docs. Have these tools made a big difference?
Robert Green You’ve named them exactly what we’re using. That’s right. We are putting the documents together on Google Docs. We’re talking to each other on Slack and we’re having a weekly call every week on Zoom with several hundred people. So that’s it ... we’ve been using these, of course, in our small labs. We’ve been using them for smaller collaborations. But that’s exactly how it works.
Steven Cherry A key question right now is how best to treat Covid patients. It seems E.R. physicians and nurses have learned a lot just in the field. For example, early on, the ventilator protocols they used were basically the same as for other respiratory illnesses. And now that they know that the best practices are quite different. But scientists like yourself also studying the role of genetics in treatment?
Robert Green Many people are. And, you know, I have to caveat this by saying I am not treating patients. My wife and my daughter are actually in the front lines. So I am hearing a lot about it. And I’ll tell you what I know. But there are peculiarities. For example, it turns out that the kind of lung disease that people get with Covid allows you to get rid of some of the CO2, even as your oxygen is falling. And what that means is the air hunger that people typically get when their oxygen is falling, which is signaled by the CO2 accumulation is not as present. And my colleagues in the emergency room are describing scenarios where they’re watching somebody talk on the telephone to their family—sit there and talk—looking perfectly normal while the oximeter shows their PO2, their oxygen level in their blood, is dropping to 90, to 85, to 80, to below 80, to the point where they suddenly pass out and then they’re in respiratory failure. So this is just one example of the way that E.R. docs and pulmonary specialists, in fact, all the clinicians on the frontlines have learned to anticipate this kind of rapid pulmonary decline.
And so there’s lots of nuances like this. I wouldn’t be in a position to name them all, but there’s lots of nuances like this where the clinicians who are taking care of patients are learning special ways of anticipating problems and special ways of managing problems. And you know, Steven, this is one of the nuances that kind of gets lost when we’re talking about when to go back to work and when to open up society and all of this, because we’re not just sheltering in place and wearing masks in order to push this out for some abstract reason. We’re buying time to figure out how to best manage patients so that when people get sick, they are treated better and better and they survive more. We were literally buying time for people to live longer because the medical system is less overwhelmed and is learning from every single case that comes through.
Steven Cherry Have scientists found any role of genetics when it comes to treatment?
Robert Green We’ve written a paper that we’ve submitted actually that we have proposed a sort of hypothetical set of advantages. If you had a system—which my group has been advocating for years—if you had a system where your DNA and my DNA was already available along with our electronic medical record and it was already integrated, then yes, I believe that there would be some distinct advantages. There would be certain kinds of rare diseases that would make you more susceptible to metabolic crises if you were in the intensive care with Covid. There would be certain kinds of genetic changes that would make you more likely to have a bad effect if you were intubated with a certain anesthesia drug. There would be certain kinds of genetic changes that would put your lungs at more risk for pulmonary failure and therefore you should probably be monitored more closely.
So it’s mostly hypothetical, Steven, because we don’t have that genetic information available in the day-to-day care of patients. But it’s just another example. I don’t want to take anything away from the severity of this pandemic. It’s horrible. It’s a life-changing once-in-a-generation sort of event. But this is a fast-moving pandemic and we are dealing with millions of deaths a day from the slow-moving pandemics of cancer, heart disease, and Alzheimer’s disease. And in all of these pandemics, fast and slow-moving. If we had genetic information integrated into our health care, I think we could do a better job of taking care of people every day.
Steven Cherry Let’s turn to testing. The antigen testing, that is for whether Covid-19 is active—that is, if a person currently has the disease—is very different from antibody or serologic testing—that is whether a person has been exposed to the disease and has developed antibodies for it. Why are they so different and why is the latter testing seemingly so much harder to get right?
Robert Green That’s exactly right. We’re really talking about two very different kinds of tests. The antigen testing or the testing for the virus is looking for the genetic signature of the virus itself. That’s where they’re sticking that swab up your nose or back your throat. They’re scraping it, they’re trying to get some virus on that swab. They then put that into buffers and then quickly reverse-transcribe it and see DNA, amplify it up through quantitative PCR, and—which is just a fancy way of saying they’re going to turn it from RNA into DNA and measure the amount. And if the amount is high enough, you’re positive. And so there that test is actually saying, yep, you’ve got virus in your nose and therefore you’re infected. And if it’s positive, that test is very reliable. I think you can believe it if you are positive for that PCR test. The trouble is, if it’s negative, it’s—and we think there maybe is somewhere between 10 and 30 percent false negatives—so that test needs to be continually improved. There seem to be people who have a lot of the virus residing in their nasopharynx and others that don’t. Maybe it’s in their gut or it’s deeper in their lungs. And so you don’t pick it up when you swab the nasopharynx. So that’s the PCR diagnostic test for viral infection.
As you said, the other kind of testing is what we hope to have soon, which is serological testing or antibody testing. This is a test for specific antibodies that your own body is making in response to the virus. And there are dozens of companies rushing to make these. They there are EUAs—emergency use authorizations—from the FDA to all these companies. So they’re able to produce them with very little validation data. It’s a necessary Wild West, but Steven, it’s a Wild West. We don’t know the accuracy of all these tests by far yet. And there’s pretty good data that’s kind of in the opposite direction here. So if you’re negative on the antibody test, you’re probably still vulnerable if you’re positive. It’s not quite clear yet whether you’re protected or not because you could be—it could be a false positive or you could have antibodies to an earlier virus that wasn’t the Coronavirus that got a so-called cross-reacted with this particular test.
So everybody’s struggling with this. But eventually, eventually, the antibody test is going to give us really good insight into who’s been had it and is presumably at least temporarily immune to re-catching it. Now, all of these statements are said with humility at this point, because we really don’t know how much immunity is conferred. We really don’t know, if it is conferred, how long it lasts. And we really don’t know if you can, after a certain period of time, catch the same virus again or next year catch a slightly mutated version of the same virus, like with influenza.
Steven Cherry What would it take to know the answers to those questions with more than a presumption?
Robert Green It really takes the kind of trials that actually everyone is jumping into action and doing, you know, in my hospital, for example, at Brigham and Women’s. There is a tremendous there’s an actual data mart, they call it. There is an actual committee adjudicating hundreds of research studies to make sure that they don’t unnecessarily overlap and that they’re working collaboratively together. And so blood samples are being taken and analyzed. Symptoms are being recorded, bioinformaticians are scraping data from the emergence from the electronic medical record and from the emergency rooms. Samples are being donated—even in the midst of being super-sick with this illness, people are being asked if they will donate samples and they’re saying, yes.
You know, if ever there was sort of a nobility to participating in research both in the past where we have these DNA biobanks already available and in the present crisis where sick people are thinking about other people than themselves. This is really bringing out the best in our scientists and in our patients. It’s amazing.
Steven Cherry That is great to hear. Finally, what role does genomics play in developing a vaccine?
Robert Green Well, it plays a major role. And again, I won’t present myself as an expert in this arena. But in general terms, the idea is that you create something in your body that actually gets in the way of the virus, either attaching on, or the virus replicating. So either it never gets into you in the first place or once it gets into you, you’ve essentially disabled it.
And one of the most obvious areas is this spike I mentioned in the beginning because we know that that’s the mechanism by which it attaches to the cell. We know it attaches—the spike specifically attaches to this receptor. So, for example, one of many strategies is that you develop a vaccine that looks like the spike. So your body makes an antibody to that spike and that antibodies now circulating around in your body. And when the virus comes along, it says “Aha! Here’s what I was built to respond to!” And it goes after that spike in it, you know, covers it, or it blocks it or it destroys it, or allows your body to destroy it.
But there are I think at last count there were over 90 different vaccine strategies that were being pursued. And some of them are quite sophisticated. Some of them are really, really interesting. This is bringing out creativity at the same time that is bringing out so much cooperation with people.
Steven Cherry Experts in public medicine have been warning that it will take 12, 18, maybe even more months, to develop a vaccine. But that seems based on our experience of diseases of years ago without maybe some of the new tools and technologies that we have and maybe also the greater collaboration we spoke of. Is there any reason to hope that maybe it won’t take that long this time around? And I don’t mean just new and better ways to communicate and share and analyze data. But for example, is there a role for gene editing using CRISPR or something else?
Robert Green Let me answer the first part as best I can. More based on just the news that you and I are both seeing than any special knowledge. But luckily there were a couple of groups that were already so sophisticated in developing special vaccine technologies, particularly for RNA viruses that they had a jump on this. So there is some hope that they will develop potentially effective viruses faster than that typical 18–24 months. So, yes, there’s hope on that.
Now as to the gene editing, I think gene editing has turned out to be this all-purpose tool in the laboratory that helps in many, many ways. So, for example, if you created a particular—if you wanted to create a particular protein that had certain confirmations or that had certain antigenic properties that you would then use in your vaccine, you know, gene editing is a potent tool to help you create that. So absolutely, gene editing is, I think, here to stay. It’s in the workflow of almost every advanced biotechnical group working on things. I don’t think we’re talking about gene editing in the sense of gene-editing human beings to be resistant or gene editing something like that. But we’re definitely talking about gene editing as a workhorse tool in the production of treatments and vaccines.
Steven Cherry It does occur to me that people have for a while now had an idea of getting rid of malaria by introducing mosquitoes that can’t reproduce, creating those mosquitoes with CRISPR. If you could create a version of the virus that does a poor job of replicating in the body—that would be a way to create a kind of immunity or vaccine.
Robert Green Well, that’s right. And you know, there’s precedent for that. Weakened viruses are live—live viruses are used in vaccines all the time. They’re weakened and modified in ways that don’t harm the human being typically, but do create the immunological response to protect you from infection with the bad virus. So that’s absolutely one of the principles. And you can use a lot of different techniques for doing that, including various degrees of modification of a virus that could include gene editing these days for sure.
Steven Cherry So overall, are you optimistic about the next three or six or twelve months or pessimistic?
Robert Green I wish we had a better national response to this. I wish we had ... We’re not arguing on a national level about the value of science. I wish we didn’t have conspiracy theories arising and dividing us. I wish that we were in some manner more aligned around the importance of evidence-based information and science as a value for our society. But I hope ... I’m not ... Overall I’m optimistic because I believe this is a teachable moment for America and the world, that those societies that prepared, those societies that were organized, the societies that had long-term vision, that believe their scientists—have done so much better than we have, and those societies that were not as well prepared.
And I’m extremely optimistic that this once-in-a-generation crisis will give us the flexibility to transform ourselves. I mean, just to pick one example, Steven, how long have we been talking about telemedicine? How long have we been saying, “You know, half of medical visits could be conducted over the telephone or by videoconference?” And yet we had barely moved in this direction at all. And I think that what we’re seeing now every single day is that doctors have, en masse, transformed their practice to telemedicine practices, reimbursers have—insurance companies have had to say, of course, we’re going to reimburse these trends, these telemedicine visits.
So you’ve got this sudden transformative change in the way we practice medicine. And I can’t see us going backward from that. And I think we’re going to see that in education. I think we’re going to see that in the workplace. I think we’re going to see that in politics if we can get past the objections to voting by mail. There is a possibility that this crisis brings positive transformation that we’ve needed for a long time and a lot of different societal areas.
Steven Cherry Not least of those, this will make us better prepared for the next pandemic.
Robert Green Absolutely. Absolutely.
Steven Cherry Well, Robert, I myself, I’m not entirely sure if I’m an optimist or a pessimist, but I’m greatly encouraged by the work being done behind the scenes, largely out of the eyesight of the public and journalists, by doctors and scientists and researchers, especially those special individuals who all three such as yourself. Thank you for your work and for giving us this time today.
Robert Green Thank you so much for having me and good luck to you.
Steven Cherry We’ve been speaking with Dr. Robert Green, a medical geneticist and physician–scientist at the Harvard Medical School, about the roles genetics and genomics play in understanding the Coronavirus and in treating Covid-19. This interview was recorded March 9, 2020. Our audio recording and engineering was done remotely by Gotham Podcast Studio. Our music is by Chad Crouch.
For TTI/Vanguard, I’m Steven Cherry.
This interview was recorded May 9, 2020.
Audio engineering by Gotham Podcast Studio, New York, N.Y.
Music by Chad Crouch
We welcome your comments @ttivanguard and @techwiseconv
Note: Transcripts are created for the convenience of our readers and listeners. The authoritative record of TTI/Vanguard’s audio programming is the audio version.
Keywords: genetics, genomics, Coronavirus, Covid-19, CRISPR, DNA, medical records, data, Robert Green, Steven Cherry, TTI/Vanguard, technology


text:
Ithaca Peak Blue Beads Turquoise Magnesite Nuggets 36" Strand
Offered here are genuine Turquoise Magnesite Nuggets with Ithaca Peak Blues. The natural nugget texture and matrix is captivating, no two beads are the same. The rounded tumbled nuggets range in size between 8mm and 12mm. The genuine stone has a good weight and feel. This long 36 inch strand will add lots of natural character to your beaded jewelry, accessories, or arts and crafts of all kinds. We offer a large variety of Turquoise Magnesite beads for whatever your jewelry or craft, be sure to check out the other listings to find the perfect beads for your needs.

domain: design


text:
BENEFITS EXPERT: Benefits support available with a granddaughter moving in
I currently just get my State Pension, along with Housing Benefit and council tax support, I currently do not work but I am considering a part-time job if this will help to support her. There is only myself in the property. I am confused as to how I can claim additional support to cover her living costs. She is 14 and still in full time education. I have been told to claim Tax Credits, other people have stated Pension Credit whilst others have stated Universal Credit. I know I can claim Child Benefit for her.
A. In this situation you are correct that Child Benefit can be claimed, as for the remaining benefits the position is as follows.
Currently Universal Credit cannot be claimed by anyone of State Retirement Age.
You have not confirmed your current State Pension Income but in this situation you should apply for Pension Credit to see if the addition of your granddaughter to the household will entitle you to some Pension Credit.
If you do successfully get at least one pence of Pension Credit (you may get more, this will depend on your State Pension), it will then mean you may also get an increase in your Housing Benefit and 100% of your Council Tax will be paid.
If you do seek any form of employment, whether part-time or full-time, then you cannot currently make a claim for Tax Credits that would include amounts for your granddaughter, unless you currently get Working Tax Credits and could then request a review to include assistance for your granddaughter.
If you claim Pension Credit and then start a job then depending on your salary you may or may not still be entitled to Pension Credit.
I would advise you have a benefit check giving an indication of your likely wage to see if working would end Pension Credit.
In general it is always best to retain entitlement to Pension Credit, especially the Guarantee element as it entitles you to other help for example maximum Housing benefit, Council Tax Support, free dental care, etc.


Canadian government votes to reopen nuclear reactor
Health Imaging News, December 18, 2007
The Canadian House of Commons has passed emergency legislation to reopen the Ontario nuclear reactor that produces the majority of the world's supply of medical isotopes. The site was shut down for safety maintenance, and the bill required all party support to override the advice of the Canadian Nuclear Safety Commission and restart the 50-year-old reactor.
- Sharp HealthCare Leaves Pioneer ACO Program
- Acute Kidney Injury Gets New Focus
- CNO Leads $1M Charge for New Scrubs, Uniforms
- Interventional Radiology No Longer a Sub-Specialty
- NFP Hospitals' Revenue Growth at 'All-Time Low'
- Half of All Primary Care, Internal Medicine Jobs Unfilled in 2013
- MA an Insurance Proving Ground for Providers
- Targeting Self-Insured Populations
- mHealth Tackles Readmissions
- States Without Medicaid Expansion Search for Alternatives


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The University of Findlay volleyball team started off the season with a pair of wins on September 1 at the Hillsdale Tournament. They began the day with a 3-0 (25-16, 25-17, 25-12) win against Saginaw Valley State and also added a 3-0 (25-13, 25-23, 25-14) victory over Glenville State.
Defensively, junior Kara Scherger led the team with 13 digs.
Winner once again led the Oilers in their victory over Glenville State with a double-double. She recorded 12 kills and added 11 digs in the win.
Kin contributed six kills while sophomore Izzy Murphy tallied six kills as well.
Olson recorded 30 assists and Scherger had 11 digs in the win.
The Oilers, who are now 2-0 on the season, will be back in action tomorrow against Michigan Tech and Indianapolis.


MIAMI — A British man, a Florida man and a Florida teen hacked the Twitter accounts of prominent politicians, celebrities and technology moguls to scam people around globe out of more than $100,000 in Bitcoin, authorities said Friday.
Graham Ivan Clark, 17, was arrested Friday in Tampa, where the Hillsborough State Attorney’s Office will prosecute him as adult. He faces 30 felony charges, according to a news release. Meanwhile, Mason Sheppard, 19, of Bognor Regis, U.K., and Nima Fazeli, 22, of Orlando, were charged in California federal court.
In one of the most high-profile security breaches in recent years, hackers sent out bogus tweets on July 15 from the accounts of Barack Obama, Joe Biden, Mike Bloomberg and a number of tech billionaires including Amazon CEO Jeff Bezos, Microsoft co-founder Bill Gates and Tesla CEO Elon Musk. Celebrities Kanye West and his wife, Kim Kardashian West, were also hacked.
The tweets offered to send $2,000 for every $1,000 sent to an anonymous Bitcoin address.
“There is a false belief within the criminal hacker community that attacks like the Twitter hack can be perpetrated anonymously and without consequence,” U.S. Attorney David L. Anderson for the Northern District of California said in a news release. “Today’s charging announcement demonstrates that the elation of nefarious hacking into a secure environment for fun or profit will be short-lived.”
The most prominent member of the trio, according to news reports is the “alleged mastermind” (quoting the Daily Beast) 17-year old Graham Ivan Clark:
A Tampa teenage “mastermind” was one of three people charged on Friday for their alleged roles in a huge Twitter hack that targeted some of the world’s wealthiest and most influential people.
The Hillsborough State Attorney’s Office filed 30 felony charges against Graham Ivan Clark, 17, for “scamming people across America” in the July 15 attack. The scheme “stole the identities of prominent people” and “posted messages in their names directing victims to send bitcoin” to accounts associated with the Tampa teen, prosecutors said.
“He’s a 17-year-old kid who apparently just graduated high school,” State Attorney Andrew Warren said in a Friday press conference. “But make no mistake, this was not an ordinary 17-year-old. This was a highly sophisticated attack on a magnitude not seen before.”
The U.S. Attorney’s Office Northern District of California charged two others for their role in the hack: Mason “Chaewon” Sheppard, 19, from Bognor Regis, in the United Kingdom; and Nima “Rolex” Fazeli, 22, from Orlando, Florida.
The case rose to greater prominence, not due to the amount of money swindled, but due in large part to the media’s obsession with Twitter. To note, it wasn’t just The Beast who covered the arrest. Breitbart, The Daily Caller and Mediaite also covered the arrest. Even TMZ got in on the act and posted their snarky take on the arrest:
Some of the most high-profile celebs, politicians and business leaders can thank a Florida teen for hacking their Twitter accounts … according to cops, who’ve busted the alleged mastermind.
Twitter says the hackers fooled its employees into handing over high-level administrative credentials over the phone in a “spear-phishing attack,” which allowed the hackers to target other Twitter employees with access to account support tools and ultimately tweet from the celeb accounts.
Kids these days.
We’ll continue to track the case as it moves closer to trial.


text:
On January 16, 2007, New Line Home Entertainment will release Texas Chainsaw Massacre: The Beginning (Unrated) on DVD. The movie features the tale of everybody's favorite cannibalistic family, and will feature tons of bonus materials and extra features. It will be available for the MSRP (Manufacturer's Suggested Retail Price) of $28.98.
- Extended and Deleted Scenes
- Audio commentary with director Jonathan Liebesman and producers Brad Fuller and Andrew Form
- Deleted scenes commentary with director Jonathan Liebesman and producers Brad Fuller and Andrew Form
- "Down to the Bone" behind the scenes documentary
Check out the artwork for the Texas Chainsaw Massacre: The Beginning's rated and unrated DVDs below:
Stay tuned to IGN DVD for more information about this and other upcoming releases!

domain: entertainment


Bezel Material: Stainless Steel. A stainless steel bezel is one of the most beautiful and delicate parts in the watch. The stainless steel bezel provides a durable, endurable and expensive look to the watch.
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Engine: Rolex Calibre 2836. Engine 2836 3200-task swiss movement can improve the accuracy of the watch more than ordinary mechanical movement. Tips: The “A Replica” is applied with Rolex 2813.
Function: Date, Hour, Minute, Second. The watch has many functions. The watch has a indicate date to show date, a minute dial is used to estimate the current time, and a second dial is used to estimate the time after the interval.
Tips for the “A Replica” version watch: This edition is very discounted, please understand that the three sub-dials of the “A Replica” Rolex Daytona are just for display only, not functional. Also, due to differences in lighting and angles, please allow for slight differences between the main image and the real thing – especially for “A Replica” version, if you really care about the differences, it’s better to choose our AAA and AAAAA versions. In addition, you are welcome to view our physical pictures, if necessary, please contact our customer service. Thanks.


text:
The Electro Harmonix Freeze has long become a pedalboard favourite of some of the more adventurous and experimental guitarists, due in no small part to its ability to create lingering, mulit layered textural sounds at the touch of a button. The Freeze allows you to sustain both individual notes and chords for an infinite amount of time with a switchable decay time. In latch mode, the Freeze will infinitely sustain a captured sample of your signal until the footswitch is pressed again, allowing for smooth transitions between notes. The Electro Harmonix Freeze is the perfect partner for anyone who loves experimenting with layering different effects.
Founded in 1968, Electro Harmonix are the original pioneers of the guitar stomp box world. The first company to really offer a wide range of affordable guitar pedals, Electro Harmonix is responsible for some of the most recognisable guitar tones in rock, and today they continue to innovate as much as they have over the last 50 years.
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- Comes with 9.6DC 200 power supply
- Handles input gains from pedal boards

domain: hobby


Southeastern shot 66.7 percent from three-point range and put four players in double-figures en route to picking up head coach Darin Grover's 200th career win, dropping Northwestern Oklahoma State 76-64 on Thursday night.
The win lifts the Savage Storm to 10-12 on the season overall and 8-8 in Great American Conference play heading into Saturday's 5:30 p.m. rematch with Southwestern Oklahoma State.
Four SE players reached double-digits led by 15 points from Cheree Kiser who finished a perfect 5-for-5 on the night with all of her shots coming from behind the arc.
She was joined by Emily Williams and Haley Schaefer who posted 12 points each, while Breana Small Peppers came off the bench to chip in 10.
Ronay English nearly joined the group with nine points, all off threes, while Bailey Welch finished with eight.
The Storm finished 12-of-18 form long range, including a 7-of-8 performance in the second half. SE finished the game shooting 47.2 percent from the field.
Welch dished out a game-high seven assists, while Kayle Reaves added four.
Schaefer flirted with a double-double, finish9ign with eight rebounds.
NWOSU opened up an early 4-0 lead, but SE answered to tie the game with a layup from Schaefer and a pair of Williams' free throws.
The teams proceeded to trade two-point leads until later in the first half when SE broke a tie with consecutive threes from Kiser and Williams, followed by a pair of Small-Peppers free throws to mount an eight point lead.
That would expand to 31-20 with 3:49 to play in the first after a Schaefer layup and a Williams free throw.
The Rangers would answer with an 11-0 run to tie the game at 31-31 with 34 seconds to play before hitting a pair of free throws to take a two-point lead.
SE would get the last word, however, as English hit a three with 23 seconds left to set the halftime score to 34-33.
The Storm slowly pushed its lead out to six points early in the second half and the teams would trade buckets until Kiser drained a three with 11:58 to play to push the lead to 52-43.
The lead expanded to 14 points following a Williams free throw with 8:10 to play.
NWOSU would cut it back to nine points with 4:21 to play, but another Kiser three with 2:57 remaining would put the lead in double-digits for good as SE pulled away with the 76-64 win.


On Sunday 28 July 2002 03:00 am, The Cunctator wrote:
> What are the articles this person has been changing?
20:08 Jul 27, 2002 Computer
20:07 Jul 27, 2002 Exploit
20:07 Jul 27, 2002 AOL
20:05 Jul 27, 2002 Hacker
20:05 Jul 27, 2002 Leet
20:03 Jul 27, 2002 Root
20:02 Jul 27, 2002 Hacker
19:59 Jul 27, 2002 Hacker
19:58 Jul 27, 2002 Hacker
19:54 Jul 27, 2002 Principle of least astonishment
19:54 Jul 27, 2002 Hacker
19:52 Jul 27, 2002 Trance music
19:51 Jul 27, 2002 Trance music
20:20 Jul 27, 2002 Hacker
20:19 Jul 27, 2002 Hacker
Most of these were complete replacements with discoherent statements.
Such as "TAP IS THE ABSOLUTE DEFINITION OF THE NOUN HACKER" for Hacker.
For the specifics follow http://www.wikipedia.com/wiki/Special:Ipblocklist
and look at the contribs.
So, it seems (if I interpret Jimbo's mail on wikitech and the discussion
here correctly) that most of us would like *some kind* of category
scheme in wikipedia. I do, too! But, we seem to differ on the details
So far, I saw three concepts:
1. Simple categories like "Person", "Event", etc.; about a dozen total.
2. Categories and subcategories, like
"Science/Biology/Biochemistry/Proteomics", which can be "scaled down" to
#1 as well ("Humankind/Person" or something)
3. Complex object structures with machine-readable meta-knowledge
encoded into the articles, which would allow for quite complex
queries/summaries, like "biologists born after 1860".
1. Easy to edit (the wiki way!)
2. Still easy to edit, but making wikipedia browseable by category,
fine-tune Recent Changes, etc.
3. Strong improvement in search functions, meta-knowledge available for
1. Not much of a help...
2. We'd need to agree on a category scheme, and maintenance might get a
3. Quite complex to edit (e.g., "<category type='person'
occupation='biologist' birth_month='5' birth_day='24' birth_year='1874'
For a wikipedia I'd have to write myself, I'd choose #3, but with
respect to the wiki way, #2 seems more likely to achieve consensus (if
there is such a thing;-)
We are faced with an issue of convenience versus freedom when we talk
about licensing images. Because we are a nonprofit charitable
organization with an educational mission, we can easily get non-free
licenses to use images.
This is easily done, but doesn't achieve the goal of building a free
encyclopedia. There is no help for it; to make a free encyclopedia,
one must stick to materials that are free.
Because we are a nonprofit charitable
organization with an educational mission, we can make heavy use of the
doctrine of "fair use" in the US.
When applicable, this may be a good solution.
Clause 7 of the license permits us to combine
independent works, even proprietary works, and this clearly includes
aggregating images and articles stored on the same server.
This is permitted in the sense that it won't violate the GFDL. But
when the other work is not free, this is not a solution--not if the
problem is how to make a better free encyclopedia. Including non-free
images, whether done legally or not, results in a non-free
If that means less images for now, then it means less images for now.
It also means that we have a very strong incentive to develop free
In many cases, people can creatively find ways to take new photos.
Another idea is to criticize the organization that stands in the way
of using the image desired. For instance,
The Foobar company stands in the way of our including a picture of this.
This text, with no link and no details about the image, would name and
shame the Foobar company, but would not help Foobar sell copies of the
Jeff Meyer wrote
>The plan is to deliver an easily searchable repository (content & searchable
>clearinghouse) of quality map content to cover the entirety of human
>history. Along with it would be a freely distributable, open source map
>player. People could then link their existing maps, add modifications, build
>new map content, etc.
I happen to be finishing up my GIS credentials and geography/cartography degree
right now and was thinking about playing around with the free software GRASS
GIS program this summer. See http://grass.baylor.edu/ and the GRASS Wiki
It would be neat to tie GRASS into MediaWiki for map creation. I'll see if I
can get that to work later this year (at least for creating simple maps).
There is a whole bunch of public domain GIS data out there (mostly in the
U.S....). It would eventually be neat to have a place to do GIS editing on the
Internet to help maintain and extend those data. As it is, the public domain
GIS material created by the U.S. government is made into proprietary products
by GIS firms who clean up, extend, and then sell the data. AFAIK there is no
free content alternative.
WikiGIS perhaps? I better snag that one... Done. WikiGIS.org and WIkiGIS.com
are reserved for the Wikimedia Foundation if and when the foundation wants
Which reminds me, Wikimedia.org is about to expire... I better fix that.
-- Daniel Mayer (aka mav)
Do you Yahoo!?
Get better spam protection with Yahoo! Mail.
> They quote Polish data from 2001. Most other data is 2003 or 2002.
Yes, the Global Reach data are not really homogeneous.
I wrongly assumed all data in the first column were from 2003.
But it does not affect the overall picture that much.
Even when we take the 2004 forecast of 9 million Polish internet users
instead of the 2001 figure of 6.9 milllion,
the pl: ratio is 1 contributor per 29 thousand, which still makes pl: first
on the list.
Again, I'm not sure how reliable the GR figures are.
Surely a forecast is less dependable when based on older figures.
Of course there are a myriad ways to measure succes of each Wikipedia.
For what it's worth I tried to establish the ratio of active contributors
per million internet users that speak that language.
So per language the figure shows that one of x thousand native speakers with
internet access contributed to Wikipedia in that language
For contributors I used wikistats Feb 26 (counts registered users > 10
For internet users per language I used figures from Global Reach (2003)
Of course I don't know how exact these figures are,
but I assume they did some homework.
Note : the GR figures list >native< speakers, so actually languages with
international appeal would show lower participation ratios (higher numbers
in the table) if all web users that might be inclined to use that language
were taken into account.
E.g. GR mentions India specifically as a country where the web language is
English and not Hindi.
Erik Zachte wrote:
> For a quick comparison of layouts of all 53 Main Pages have a look at
>Complete pages are shown, captured at a virtual 1000*3000 screen resolution,
>reduced to 40% size.
>At 40% reduction the text is hardly readable, but it gives a nice comparison
>of overall layout per wikipedia.
It's nice to see that quite a few languages are getting images on their
main pages. I wish I knew enough, or any, Romanian, to understand how
that picture of two frying pans got featured.
ZH appears to have picked up EN's new layout (or did we get it from
them?). I particularly enjoy seeing the right-to-left layouts in Hebrew
I hereby suggest that we start to distribute the press release versions on
Monday (at least for the non-English versions that are ready).
I'm going to create the English Wikipedia version now (based on the Meta
Sorry for the late note - I thought we had another week.
Do you Yahoo!?
Yahoo! Mail SpamGuard - Read only the mail you want.
For a quick comparison of layouts of all 53 Main Pages have a look at
Complete pages are shown, captured at a virtual 1000*3000 screen resolution,
reduced to 40% size.
At 40% reduction the text is hardly readable, but it gives a nice comparison
of overall layout per wikipedia.
This job was easy thanks to url2bmp (Windows freeware)
despite its name it handles other image formats as well,
can be run in batch mode


text:
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text:
Per year, around 800,000 Americans die of heart disease:
Per year, around 100,000 Americans die from adverse response to approved medical care:
Or maybe it’s 250,000:
Per year, around 40,000 Americans die from second-hand smoke:
How do we respond to these highest risks which are rapidly getting worse?
We enable them. We amplify them. We definitely DO NOT combat them.
Severe obesity doubled from 2000 to 2017:
Obesity tripled since 1975:
Oversight on medical science has DECREASED dramatically, owing largely to our blank checks for pharma during the pandemic:
Regarding tobacco use, vaping has increased 1,800% among youth:
Returning to medical oversight for a moment, the irony is that the PREP Act was incredibly controversial when it was introduced by a Republican congressperson, Richard Burr, and signed into law by George W. Bush in 2006. Burr has long had unsavory ties to pharmaceutical and medical interests: https://www.statnews.com/2016/10/25/richard-burr-pharma-biotech-campaign/ . And he was implicated in insider trading at the outset of the pandemic: https://www.propublica.org/article/senator-dumped-up-to-1-7-million-of-stock-after-reassuring-public-about-coronavirus-preparedness.
All of a sudden, nobody cared about this when the pandemic struck.
But our willful naivety and complacency regarding medicine didn’t start in 2020. Nobody cared when the American Heart Association was caught red-handed being bought by pharmaceutical interests: https://www.huffpost.com/entry/health-news_b_4398304.
In fact, for the 30 years prior to the trending hashtag #trustthescience, we haven’t been able to trust the science. In modern contemporary science today, MOST published scientific research CANNOT be corroborated: https://www.nature.com/articles/533452a. Not some. MOST. Not a lot. MOST. That’s right: 70% of scientists cannot replicate claims of their peers. SEVENTY PERCENT.
When less than a third of science is able to be corroborated, how did anyone think #trustthescience made sense? Trust evidence. Distrust people. Trust solid arguments. Distrust consensus and popularity.
Scientific and medical institutions did not ascend to Godlike unquestionability in the 20th century, and they don’t mystically remain infallible for all of time. HUGE science scandals hit the news cycle for one or two days, like 16 years of flawed Alzheimer’s research (https://www.medicalnewstoday.com/articles/alzheimers-study-controversy-what-does-it-mean-for-future-research#What-might-this-mean-for-dementia-research) or outright physics fraud (https://www.nature.com/articles/news020923-9); but we just go back to our previously-scheduled programming.
Sadly, expert scientific consensus tells us very little about evidence or truth. Look no further than Barry Marshall’s 2005 Nobel Prize, and you’ll find how consensus REJECTED evidence for two decades. The scientific consensus DENIED the truth. In fact, it’s hard to believe we would even know Marshall’s story and the bacterial cause of ulcers if his conclusions hadn’t landed us on antibiotic treatment pay dirt. If wealthy/influential people hadn’t had something to gain, could we rightly believe the evidence would’ve prevailed?
I don’t think so. I see zero evidence that we care at all about genuine evidence, data, or risks. It’s hard to see truth prevailing based on truth anymore. It seems to be more tied to financial interests or popular public feelings.
I mean, again, observe our top risks. The top risks have no social media profile templates, no hashtags, no memes, no attention. I see no evidence that people follow science or care about risk. They follow popularity and worry about “risks” that are popularly trending in their little tribes.
Following the food pyramid kills about 1 million Americans every year. And, if anything, our public embrace of unhealthy eating has accelerated. Look at the stats. Following scientific consensus and medical advice kills another 100,000 to 250,000 Americans per year. But we are becoming LESS skeptical of experts and entrenched institutional organizations. Second-hand smoke kills 40,000 per year. But smoke shops are popping up everywhere and tobacco use is resurgent.
Every single day, these things are killing 3,000 Americans. EVERY DAY. It’s like a 9/11 terrorist attack PER DAY EVERY DAY. Motor vehicle accidents take another 100 to 200 lives daily. DAILY. But there is zero outrage. No outcry. These ARE our highest risks. No opinion. No ideology. No politics.
As we go further down the list, we find that opioid overdose and suicide are making a run for the top risks. Next up: falls. Seriously. After cancers, respiratory conditions, all preventable risks, opioids, suicides, and motor vehicle deaths, your next most likely way you’ll die is from falling.
Not a joke. This one lies directly inside my profession. Weak people are at higher risk of fall. And falling is in the top 8 risks of death.
From 1966 to 2020 the TOTAL number of deaths in American mass shootings was LESS than food/medicine/smoking will kill in the first 12 hours of TODAY: https://rockinst.org/gun-violence/mass-shooting-factsheet/. Each hour medicine kills more people inside peaceful countries than guns kill in war zones. TODAY more Americans will die from food and medical recommendations than there will be mass shooting victims for THE YEAR. This MONTH vehicles will kill more people than mass shootings will kill this YEAR.
This year motor vehicles will kill more American children than 40 years of mass shootings have killed ALL PEOPLE OF ALL DEMOGRAPHICS.
This year clinics and hospitals in America will kill more children than all victims of all mass shootings in all countries combined for all of time.
And every single day, twice as many people will die from taking a fall than the combined total victims in gang shootings PLUS individual gun violence PLUS mass shootings PLUS accidental firearm discharge.
I agree that we should address our 10th through 1000th risks. That makes sense. What doesn’t make sense is the public completely oblivious and carefree about the top 9. The time, energy, money, and outrage people spend on any one of their tribe’s select 10th through 1,000th risks EXCEEDS their concern over ALL TOP RISKS combined.
Be passionate. That’s fine. Join your tribe in its fear-mongering over non-risks which never even break the top 8. That’s fine too. What isn’t fine is totally ignoring all the real top risks.
But imagine what a beautiful world we could enjoy if people cared at all about the top health risks.


A fresh appeal for witnesses to a fatal car crash near Gaydon where an 80-year-old woman died has been launched by Warwickshire Police.
The crash happened on the B4451 at about 6pm on Wednesday February 15, and involved a grey Ford Fiesta and a white BMW that failed to stop at the scene.
The driver and passenger from the Fiesta, a 56-year-old woman and an 80-year-old woman, were taken to University Hospital Coventry and Warwickshire with serious injuries, where the 80-year-old died the following day.
PC Stephen Barr of Warwickshire Police said: “Since this tragic fatal collision, many lines of enquiry have been explored and witness evidence has been gathered.
“From CCTV footage we have established that the traffic light controlled junction leading to Gaydon village and along the B4451 toward junction 12 of the M40 was heavily congested with traffic, yet unfortunately only a very limited number of witnesses have come forward to the police.
“Did you see a white coloured BMW or a grey coloured Volkswagen Jetta drive in a manner that caught your attention in the moments leading up to the fatal collision with the Ford Fiesta?
“From accounts taken we are aware of a Mazda MX5 that may well have had an uninterrupted view of the events leading up to the collision.
“That Mazda travelled through the traffic light controlled junction towards junction 12 where it was held in queuing traffic and the driver could have important information.”
PC Barr is urging the driver of the Mazda or anyone else with information to contact him on 01926 415000, and ask for PC 718 Stephen Barr on extension 6110, or email email@example.com
Alternatively, contact police on 101 quoting incident number 310 of February 15.