text:
Does ovarian endometrioma affect the number of oocytes retrieved for in vitro fertilization?
Objective: To investigate the effects of ovarian endometrioma on the number of oocytes retrieved for in vitro fertilization (IVF).Background: The presence of endometrioma may be the most important predictor of a poor reproductive outcome. Literature data suggest that ovarian endometriomas might affect the response to ovarian stimulation and oocyte retrieval.Methods: The present retrospective study evaluates 2,023 women who applied to our center with an infertility complaint. Twenty-nine women with endometriomas (group 1) who were treated with IVF were included in the study. They were compared with 51 women with unexplained infertility (group 2) regarding the number of retrieved oocytes after egg retrieval and number of metaphase II oocytes. The diagnosis of endometrioma was made via ultrasound examination with the identification of low-density cystic masses in the ovaries. The patients underwent a controlled ovarian hyperstimulation (COH) with either the long agonist mini-dose protocol or the multi-dose antagonist protocol.Results: The incidence of endometrioma in infertile women was found to be 1.4 %. The women's ages ranged between 24 and 45 years, and the duration of their infertility ranged between 12 and 216 months. The endometrioma was bilateral in 24 % of the cases. The mean endometrioma diameter was 26.2±7.3 mm for the right ovary and 23.2±6.1 mm for the left ovary. The average number of retrieved oocytes after egg retrieval in groups 1 and 2 was 12.4±8.3 and 12.2±8.6, respectively. The average number of metaphase II oocytes in groups 1 and 2 was 8.6±6.1 and 9.4±7.3, respectively. The number of retrieved oocytes after egg retrieval and the number of metaphase II oocytes in both endometrioma group and unexplained infertile group were similar (p >0.05).Conclusion: Endometrioma did not reduce the number of retrieved oocytes in a COH cycle for IVF treatment. However it should be noted that the ovarian response is affected by the size of endometriomas, bilaterality, previous surgeries, recurrence, and the patient's age (Tab. 1, Ref. 31). Keywords: endometrioma, oocyte, infertility.
Available from: Fernanda Bertuccez Cordeiro
- "Color versions of one or more of the figures in the article can be found online. number of oocytes retrieved during in vitro fertilization (IVF) cycles, but characteristics such as the size of endometriomas, bilaterality, and previous excision surgeries, may affect the ovarian response and oocyte quality [Kiran et al. 2012]. "
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ABSTRACT: Endometriosis is a chronic gynecological condition that affects 10-32% of women of reproductive age and may lead to infertility. The study of protein profiles in follicular fluid may assist in elucidating possible biomarkers related to this disease. For this, follicular fluid samples were obtained from women with tubal factor or minimal male factor infertility who had pregnancy outcomes after in vitro fertilization (IVF) treatment (control group, n ¼ 10), women with endometriosis (endometriosis group, n ¼ 10), along with the endometrioma from these same patients were included (endometrioma group, n ¼ 10). For proteomic analysis, samples were pooled according to their respective groups and normalized to protein content. Proteins were analyzed by in tandem mass spectrometry (MS E) Spectra processing and the ProteinLynx Global Server v.2.5. was used for database searching. Data was submitted to the biological network analysis using Cytoscape 2.8.2 with ClueGO plugin. As a result, 535 proteins were identified among all groups. The control group differentially or uniquely expressed 33 (6%) proteins and equal expression of 98 (18%) proteins was observed in the control and endometriosis groups of which 41 (7%) proteins were further identified and/or quantified. Six (1%) proteins were observed in both the endometriosis and endometrioma groups, but 212 (39%) proteins were exclusively identified and/or quantified in the endometrioma group. There were 9 (1%) proteins observed in both the control and endometrioma groups and there were 139 (25%) proteins common among all three groups. Distinct differences among the protein profiles in the follicular fluid of patients included in this study were found, identifying proteins related to the disease progression and IVF success. Thus, some pathways related to endometriosis are associated with the presence of specific proteins, as well as the absence of others. This study provides a first step to the development of more sensitive diagnostic tests and treatment. Abbreviations: bhCG: beta subunit of human chorionic gonadotropin; FSH: follicle stimulating hormone; FF: follicular fluid; GO: Gene Ontology; GnRH: gonadotropin-releasing hormone; hCG: human chorionic gonadotropins; ICSI: intracytoplasmic sperm injection; IVF: in vitro fertilization; KEGG: Kyoto Encyclopedia of Genes and Genomes; LH: luteinizing hormone; MS: mass spectrometry; MS E : in tandem mass spectrometry; nanoESI MSE: nano Electrospray MSE; nanoUPLCtandem-: nano Ultra Performance Liquid Chromatography tamdem; ROS: reactive oxygen species; VDBP: vitamin D-binding protein
Available from: humrep.oxfordjournals.org
- "Studies on the effect of an endometrioma on the quality of oocytes have reported contradictory results. Some authors showed that endometriosis affected total oocyte number (Suzuki et al., 2005), while others have shown that the presence of an endometrioma did not reduce the total number of retrieved oocytes during IVF treatments (Almog et al., 2011; Kiran et al., 2012). These contradictory sets of results might be explained by the fact that the contralateral intact ovary might adequately compensate for the reduced function of the affected ovary. "
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ABSTRACT: Does the iron content of an endometrioma represent a potential source of toxicity for the adjacent follicles?
The presence of an endometrioma increases iron and H/L ferritin levels, and transferrin receptor (TfR1) mRNA in individual follicles proximal to the endometrioma and is accompanied by reduced oocyte retrieval.
Levels of free iron in endometriotic ovarian cysts are much higher than those in normal serum or in non-endometriotic ovarian cysts. The presence of an endometrioma exerts a detrimental effect on the surrounding healthy ovarian tissue as reflected by a reduced number of developing follicles and oocytes retrieved in IVF cycles.
This is a research study with prospective collection and evaluation of individual follicles (follicular fluid and luteinized granulosa cells) from the affected and the healthy ovaries of 13 women with unilateral endometrioma.
Individual follicular samples (145) were obtained from 13 women with endometriosis-related infertility undergoing IVF-ICSI procedures from May 2012 to March 2013. All women had unilateral endometrioma not previously treated with surgery; the contralateral ovary was free of endometriomas and previous surgery. The average ± SEM age was 35.36 ± 2.5 years with anti-Mullerian hormone levels of 2.03 ± 0.55 ng/ml. Follicles were classified as: (i) proximal follicles, in physical contact with the endometrioma; (ii) distal follicles, present in the affected ovary but not in close contact with the endometrioma and (iii) contralateral follicles, in the contralateral healthy ovary. Iron content was measured by the FerroZine method. H/L ferritin subunits were evaluated by specific enzyme-linked immunosorbant assays. Expression of H ferritin and TfR1 was examined by semi-quantitative RT-PCR. Oocyte retrieval rates and Day 3 embryo quality were analyzed.
Total iron levels were higher in endometrioma-proximal follicles compared with endometrioma-distal ones (P = 0.009) and to follicles in the healthy ovary (P = 0.02). L ferritin was higher in proximal versus distal follicles (P = 0.044) or follicles from the healthy ovary (P = 0.027). H ferritin was higher in the proximal and distal follicles compared with follicles in the healthy ovary (P = 0.042 and P = 0.0067, respectively). H ferritin transcript levels in granulosa cells were higher in proximal follicles versus follicles from healthy ovary (P = 0.02). TfR1 transcript levels were higher in proximal versus distal follicles (P = 0.03) and versus follicles from the healthy ovary (P = 0.04). The oocyte retrieval rate was lower in proximal and distal follicles than in follicles from the healthy ovary (P = 0.001 and P = 0.04, respectively).
This is a study on a relatively small sample size and confirmation in a larger group of patients may be required. The method used to purify luteinized granulosa cells offers the best combination of purity, viability and total number of cells recovered. However, a minor contamination by CD45(+) cells (<5%) cannot be excluded.
This study represents a further in-depth analysis of the toxic influence of the endometrioma content on the surrounding follicles. We demonstrate the presence of iron-related compounds that are potentially toxic to developing ovarian follicles adjacent to the endometrioma during IVF. Our findings provide novel information that suggests that when surgical removal of the endometrioma is not the option, follicle aspiration at sites distant from the endometrioma might increase the probability of retrieving oocytes.
This project was supported by Fondazione Giorgio Pardi, Milan, Italy. The authors have no competing financial interests in relation to the content of this research paper. TRIAL REGISTRATION NUMBER: NA.
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ABSTRACT: Endometriosis is a complex disease of young women in reproductive age. It's responsible for dysmenorrhea, dyspareunia, chronic pelvic pain and infertility. Medical and surgical treatments have different aims. Hormonal suppression tends to stop natural evolution of the disease and surgery enables macroscopic excisions of endometriotic implants. Outcomes depend on the stage and the preoperative symptoms. This article summarizes a review of surgical management of endometriosis describing surgical indications, techniques, and outcomes in terms of pain and fertility.
Data provided are for informational purposes only. Although carefully collected, accuracy cannot be guaranteed. The impact factor represents a rough estimation of the journal's impact factor and does not reflect the actual current impact factor. Publisher conditions are provided by RoMEO. Differing provisions from the publisher's actual policy or licence agreement may be applicable.


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Research, led by Lori Hart, Ph.D., research associate and Constantinos Koumenis, Ph.D., associate professor,and research division director in the Department of Radiation Oncology, both from the Perelman School of Medicine, University of Pennsylvania, and Davide Ruggero, Ph.D., associate professor, Department of Urology, University of California, San Francisco, shows for the first time that the UPR is active in patients with human lymphomas and mice genetically bred to develop lymphomas.
Importantly, when the UPR is inactivated, lymphoma cells readily undergo cell death. Their findings appear online in the Journal of Clinical Investigation and will appear in the December 2012 issue.
“The general implications of our work are that components of this pathway may be attractive anti-tumor targets, especially in lymphomas,” says Koumenis. “Indeed, an enzyme called PERK, a kinase that we found to play a central role in UPR, is already being targeted by several groups, in academia and pharmaceutical companies with specific inhibitors.”
The cancer-causing gene c-Myc paradoxically activates both cell proliferation and death. When the cell becomes cancerous, c-Myc–induced death is bypassed, promoting tumor formation. “A critical feature of c-Myc-overexpressing cells is an increased rate of protein synthesis that is essential for Myc’s ability to cause cancer,” says Tom Cunningham, Ph.D., a postdoctoral fellow in the Ruggero lab. “Myc tumor cells use this aberrant production of proteins to block apoptosis and activate the UPR. These cancer cells depend on Myc-induced increases in protein abundance to survive. Therefore, targeting protein synthesis downstream of Myc oncogenic activity may represent a promising new therapeutic window for cancer treatment,” adds Ruggero.
The accumulation of unfolded proteins in the endoplasmic reticulum, an inner cell component where newly made proteins are folded, initiates a stress program, the UPR, to support cell survival. Normally, UPR kicks in when there is an imbalance in the number of proteins that need to be folded and chaperones, specialized proteins that help fold them.
Analysis of mouse and human lymphomas demonstrated significantly higher levels of UPR activation compared with normal tissues. Using multiple genetic models, the two teams, in collaboration with additional labs in the US and Europe, demonstrated that Myc specifically activated one arm of the UPR, leading to increased cell survival by autophagy.
Autophagy is a survival pathway allowing a cell to recycle damaged proteins when it's under stress and reuse the damaged parts to fuel further growth. Cancer cells might be addicted to autophagy, since this innate response may be a critical means by which the cells survive the nutrient limitation and lack of oxygen commonly found within tumors.
Inhibition of one protein, PERK, in the UPR arm studied, significantly reduced Myc-induced autophagy and tumor formation. What’s more, drug- or genetic-mediated inhibition of autophagy increased Myc-dependent cell death.“Our findings establish a role for UPR as an enhancer of c-Myc–induced lymphomas and suggest that inhibiting UPR may be particularly effective against cancers characterized by c-Myc overexpression,” says Koumenis. “In this context the UPR essentially acts as one of the cell’s rheostats to counterbalance Myc’s runaway cell replication nature and its pro-cell-death tendencies.”
This work included several laboratories, including those of Alan Diehl and Serge Fuchs, both from Penn; Andrei Thomas-Tikhonenko from CHOP and Penn; and Ian Mills from the Oslo University Hospital.
Penn Medicine is one of the world's leading academic medical centers, dedicated to the related missions of medical education, biomedical research, and excellence in patient care. Penn Medicine consists of the Raymond and Ruth Perelman School of Medicine at the University of Pennsylvania (founded in 1765 as the nation's first medical school) and the University of Pennsylvania Health System, which together form a $4.3 billion enterprise.
The Perelman School of Medicine is currently ranked #2 in U.S. News & World Report's survey of research-oriented medical schools. The School is consistently among the nation's top recipients of funding from the National Institutes of Health, with $479.3 million awarded in the 2011 fiscal year.
The University of Pennsylvania Health System's patient care facilities include: The Hospital of the University of Pennsylvania -- recognized as one of the nation's top "Honor Roll" hospitals by U.S. News & World Report; Penn Presbyterian Medical Center; and Pennsylvania Hospital — the nation's first hospital, founded in 1751. Penn Medicine also includes additional patient care facilities and services throughout the Philadelphia region.
Penn Medicine is committed to improving lives and health through a variety of community-based programs and activities. In fiscal year 2011, Penn Medicine provided $854 million to benefit our community.
Karen Kreeger | EurekAlert!
Nanoparticles as a Solution against Antibiotic Resistance?
15.12.2017 | Friedrich-Schiller-Universität Jena
Plasmonic biosensors enable development of new easy-to-use health tests
14.12.2017 | Aalto University
DNA molecules that follow specific instructions could offer more precise molecular control of synthetic chemical systems, a discovery that opens the door for engineers to create molecular machines with new and complex behaviors.
Researchers have created chemical amplifiers and a chemical oscillator using a systematic method that has the potential to embed sophisticated circuit...
MPQ scientists achieve long storage times for photonic quantum bits which break the lower bound for direct teleportation in a global quantum network.
Concerning the development of quantum memories for the realization of global quantum networks, scientists of the Quantum Dynamics Division led by Professor...
Researchers have developed a water cloaking concept based on electromagnetic forces that could eliminate an object's wake, greatly reducing its drag while...
Tiny pores at a cell's entryway act as miniature bouncers, letting in some electrically charged atoms--ions--but blocking others. Operating as exquisitely sensitive filters, these "ion channels" play a critical role in biological functions such as muscle contraction and the firing of brain cells.
To rapidly transport the right ions through the cell membrane, the tiny channels rely on a complex interplay between the ions and surrounding molecules,...
The miniaturization of the current technology of storage media is hindered by fundamental limits of quantum mechanics. A new approach consists in using so-called spin-crossover molecules as the smallest possible storage unit. Similar to normal hard drives, these special molecules can save information via their magnetic state. A research team from Kiel University has now managed to successfully place a new class of spin-crossover molecules onto a surface and to improve the molecule’s storage capacity. The storage density of conventional hard drives could therefore theoretically be increased by more than one hundred fold. The study has been published in the scientific journal Nano Letters.
Over the past few years, the building blocks of storage media have gotten ever smaller. But further miniaturization of the current technology is hindered by...
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domain: news


Kemp Recital Hall
January 22, 2018
Gentry, Sarah Violin, "An Evening of Baroque Music: Sarah Gentry, Violin; January 22, 2018" (2018). School of Music Programs. 3618.
This is the eighty-fourth program of the 2017-2018 season.


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domain: celebrity


The X7 moves from Europe to America for more testing in its natural home.
In September, BMW revealed the X7 iPerformance concept at the 2017 Frankfurt motor show, which previewed a future production flagship SUV with the same name. The study gave us a basic idea of how the first-ever X7 could look like and now, more than two months later, the Bavarian company is continuing work on the vehicle with a spot of testing.
Motor1.com reader Kris Hamburger sent us this fresh batch of spy photos taken in California, which show an X7 test prototype. Naturally, it’s still hiding under heavy camouflage and, interestingly, it’s not wearing production headlights, which we saw on a previous prototype caught testing in Europe.
The all-new X7 will be based on the CLAR platform, which already underpins the 7 Series and 5 Series, as well as the new Rolls-Royce Phantom and the upcoming Cullinan. The powertrain options will be shared with the 7 Series, which means there will be an entry-level six-cylinder engine, turbocharged four-cylinder plug-in hybrid, mid-range V8 and a mighty V12 at the top.
It’s not a big surprise that BMW is testing the X7 in the United States, as the German manufacturer will actually build the final production version of the car at its plant in Spartanburg, South Carolina. Assembly is expected to begin at some point in 2018 and we probably won’t see the model on sale before the second half of the year.
The first to arrive will be the standard seven-seat variant, which will be followed by a four-seater luxury model with a premium price attached. According to a recent report, BMW is considering an even more luxurious vehicle with a coupe-like shape, probably getting the X8 moniker. If approved for production, it will basically be what the X6 is to the X5 – a more stylish version, using the same technologies and wearing an even heftier price tag.


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Creative writing exercises for groups
Exercises for creative writing groups
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Creative writing introduction exercises
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Creative writing exercises for poetry
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Creative writing character development exercises
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Show not tell creative writing exercises
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domain: literature


Sign up for Eagles Extra: Get exclusive news, behind-the-scenes observations and the ability to text directly with reporters
Head coach Nick Sirianni has plenty of question marks on his depth chart this summer, and players on the roster bubble will be under a microscope for the next month. Some of those players won’t even get that long to prove themselves, as the Eagles will need to cut their roster from 90 to 85 players on Aug. 17 and 85 to 80 players on Aug. 24.
Following the preseason, the Eagles will need to cut their roster to 53 players on Aug. 31 before the Week 1 matchup in Atlanta against the Falcons.
Here are the 40 Eagles players who find themselves fighting for jobs on the roster bubble this summer:
QB Nick Mullens: The Eagles dropped undrafted rookie QB Jamie Newman in favor of Mullens, an experienced NFL arm with upside. Mullens is coming off a serious elbow injury, but he is expected to participate in training camp. Mullens, like Joe Flacco, has started more NFL games than Jalen Hurts. He will be competing with himself this summer.
RB Boston Scott: After spending last season as the Eagles’ de facto No. 2 running back, Scott will see heavy competition this offseason. While he might fall down the depth chart, the Eagles are likely to favor his experience over upside in a bottom-of-the-roster battle.
RB Kerryon Johnson: Johnson is coming off an underwhelming three-year stint with the Detroit Lions. While he’s among the best blocking backs in the league, Johnson needs to show versatility this summer to stick around in a major role. His chief competition is Scott.
RB Jordan Howard: Johnson’s arrival likely took some wind out of Howard’s sails. Howard told local media that he didn’t see much interest as a free agent before re-signing with the Eagles this offseason. This summer could be his last stand.
RB Jason Huntley: The Eagles claimed Huntley off waivers following last year’s cutdown deadline. The former staff then chose not to use him. Huntley will need to stick out on special teams, most notably as a returner, to stick around.
RB Adrian Killins: The second-year gadget back worked with the wide receivers throughout the offseason program. Given his versatility and rare speed, Killins is a legitimate dark horse to make the roster.
WR Greg Ward: Last year’s starting slot receiver is the most experienced player at his position, and that’s not really saying much. Ward has been used almost exclusively in the slot over the past two years, but he will need to show enhanced versatility to stick around with his new staff.
WR Quez Watkins: Much like QB Jalen Hurts, Watkins wasn’t able to show off his skills until the home stretch of his rookie season last year. Watkins has special speed and gadget ability, so he has the type of upside worth keeping around. Perhaps he could bring some special teams value this year as well.
WR John Hightower: Unlike Watkins, Hightower was thrust into action early on as a rookie. While he showed the ability to beat defenders on “go” routes as a rookie, he struggled with drops, mental errors and never contributed on special teams. He was benched for the majority of the home stretch. He has a lot to prove to the new staff.
WR J.J. Arcega-Whiteside: The former second-round pick is no longer on scholarship. A rare body type on the Eagles’ roster, Arcega-Whiteside needs to show he can play up to his size, especially in the red zone. He should have a leg up on Ward, Hightower and Watkins if he can provide a presence in scoring position throughout the summer.
TE Caleb Wilson: After spending five games on the active roster last season, Wilson might have some edge in the No. 3 competition if Zach Ertz leaves (trade/release). Wilson needs to stand out on special teams during the preseason.
TE Jack Stoll: The undrafted rookie actually has a shot at a roster spot. With the No. 3 TE battle likely to come down to a pack of afterthoughts and projects, there’s no reason to rule out Stoll.
TE Jason Croom: Croom is the most experienced tight end behind Ertz, Rodgers and Goedert. Perhaps, that added experience will help Croom make the roster.
OT Brett Toth: The Army alum was given some late-season playing time last year. He will need to show progress this summer to stick around.
OT Le’Raven Clark: The veteran lineman is likely to start the summer on the Physically Unable to Perform (PUP) list, as he is recovering from an Achilles injury. If he can make it back in time for the preseason, he could have a shot at sticking around as a deep reserve.
G/C Nate Herbig: Year 3 will be huge for Herbig. Once seen as a possible long-term replacement for right guard Brandon Brooks or center Jason Kelce, Herbig needs to step up with the arrival of Landon Dickerson. Herbig is probably in the best spot of the group behind Brooks, Dickerson and Isaac Seumalo. He lost 30 pounds this summer in preparation for a new offense and coaching staff.
G/T Matt Pryor: While his jack-of-all-trades versatility was appealing last summer, Pryor struggled when given long-term looks in the lineup. He needs to have a monster camp to stick around.
G/C Ross Pierschbacher: The Alabama alum joined the Eagles late last season. Given his background with QB Jalen Hurts, perhaps he will be helped by the familiarity. He needs to beat out Herbig and/or Pryor for a job.
G Sua Opeta: A Jeff Stoutland favorite, Opeta is in for an interesting camp battle this offseason. Opeta needs to show that he’s more than just a guard to stick around. A college offensive tackle, Opeta might see looks on the outside of the line if injuries occur during camp.
G/T Kayode Awosika: An undrafted rookie with notable upside, Awosika is a dark horse for a roster spot. Every summer, Stoutland seems to find an undrafted guard to develop. Awosika is the latest in the long line of under-the-radar competitors.
C Luke Juriga: With the arrival of Dickerson, Juriga’s roster stability is lacking. A former practice-squad call-up, Juriga needs to have a strong summer behind Kelce.
DE Joe Ostman: The annual underdog wants to force the Eagles to keep him on the Week 1 roster. Two years removed from ACL surgery, Ostman is in a better position than he was last year.
DE Tarron Jackson: The sixth-round rookie needs to outduel Ostman to make the roster. While he will look to earn time on defense, his primary responsibility as a rookie will be on special teams.
DT Hassan Ridgeway: The veteran lineman has only played in 14 games over the past two years for the Eagles. However, when he’s played, he’s been a strong fourth defensive tackle in the rotation. He needs to stay healthy this season.
DT T.Y. McGill: The well-traveled defensive tackle played above expectations last year as a practice squad fill-in. He will compete with Ridgeway and others for a 53-man roster spot this season.
DT Raequan Williams: A former undrafted free agent, Williams knows he’s going to need to battle to stick around. He needs to take a big jump in development to outlast the competition.
DT Marlon Tuipulotu: The sixth-round pick could be a solid backup nose tackle as a rookie. If the Eagles keep five defensive tackles, Tuipulotu will almost assuredly be on the depth chart.
LB Davion Taylor: Last year’s third-round pick has a lot to prove this season. The new staff, according to multiple sources, is high on Taylor, but he needs to show he’s more than just an athletic gimmick linebacker. As a backup linebacker, he will need to continue to excel on special teams as well.
LB Shaun Bradley: Bradley flashed a bit as a rookie, especially on special teams. The Mount Holly native is in a good spot to earn a backup job.
LB/S JaCoby Stevens: The Eagles plan to convert Stevens, a college safety, into a linebacker. A solid blitzer with good length and athleticism, Stevens could actually unseat Taylor as the long-term project at the position. He’s an intriguing prospect.
DE/LB Genard Avery: The Eagles have used Avery as a rotational pass rusher over the past two years, and the results have been mediocre, at best. Now, they’ll use him in his more natural position and hope he works out.
CB Craig James: The special teams ace had his second season in Philly marred by injuries. Now healthy, James will look to earn his way into playing time on defense.
CB Michael Jacquet: After flashing as an undrafted rookie, Jacquet will look to establish himself as a consistent member of the defense in his second season.
CB Josiah Scott: The Eagles traded a 2023 sixth-round pick (and cornerback Jameson Houston) to acquire Scott from the Jaguars. He’s a nickel-only corner, who could push Maddox for playing time this summer.
CB Kevon Seymour: From selling tires to playing in the Eagles’ secondary, Seymour had a whirlwind pandemic year. Now, he needs to prove himself to a new coaching staff.
S Marcus Epps: A litany of injuries forced Epps into the lineup last year. He was actually able to lead the team with two interceptions on the season. Now, he will compete to stick around in training camp.
S Andrew Adams: The veteran special teams ace will compete with Epps, Grayland Arnold and Elijah Riley for a spot. Adams needs to be more than a special teams player to make the roster.
S Grayland Arnold: After earning some playing time last season, Arnold needs to show he belongs with a new regime. While he was lauded as an undrafted steal last offseason, he needs to prove he actually can get on the field without injuries this summer.
S Elijah Riley: The former undrafted free agent played on special teams late in his rookie season. A versatile defensive back, Riley is a dark horse for the last safety spot.
P Arryn Siposs: The Eagles let Cameron Johnston walk in free agency to make way for the cheaper, inexperienced Siposs. The team was looking to save some cash, but Siposs could prove to be a risky gamble. While he isn’t competing with anyone on the roster, if Siposs struggles, he could be one-upped by the waiver wire.
Conclusion: With 36 projected locks heading into training camp, these 40 players are competing for 17 roster spots. The roster battles at running back, wide receiver, cornerback, linebacker and safety will be crucial to the Eagles’ success this season. The Eagles are likely to cut several talented linemen this summer, and they’ll hope to retain those players on the practice squad.
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Casa Feliz Historic Home Museum
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You may use the following form to text the link of Casa Feliz Historic Home Museum to a SMS capable phone.
Casa Feliz Historic Home Museum is located at 656 North Park Avenue, Winter Park, FL - 32789. Besides this history museum, there are two more history museums listed in Winter Park. There's also sixteen attractions listed in this city in other categories. You will find the exact location of Casa Feliz Historic Home Museum on the map above. If you need any other information on Casa Feliz Historic Home Museum that is not listed here, please call (407) 628-8200 or visit their website.
None of our visitors shared their reviews on Casa Feliz Historic Home Museum yet. However, we have collected 5 ratings from external sources on this history museum.Write a Review on this History Museum
Following is a list of other museums found within few miles of Casa Feliz Historic Home Museum. You may also view all museums in Winter Park, Florida.
The Charles Hosmer Morse Museum of American Art,
Winter Park Historical Museum,
Albin Polasek Museum & Sculpture Gardens,
Hannibal Square Heritage Center,
Cornell Fine Arts Museum,
Waterhouse Residence And Carpentry Shop Museum,

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Photovoltaic panels come in three types: monocrystalline, polycrystalline, and thin-film. This article will present the main characteristics of polycrystalline photovoltaic panels and thin-film panels.
Polycrystalline Photovoltaic Panels
Polycrystalline photovoltaic panels are made from melted silicon fragments, giving them a mosaic-like appearance. These panels have a square shape and a blue color. Due to being made from multiple silicon crystals, polycrystalline panels do not allow for a significant movement of electrons within each cell.
Characteristics of polycrystalline panels
- In the production of polycrystalline panels, not much raw material is used up. Crystals do not require individual placement and polishing;
- The maximum temperature supported is 85℃, while the minimum temperature is -40℃;
- They have a lower efficiency at high temperatures compared to monocrystalline panels;
- Installation of polycrystalline panels is cheap and simple.
Advantages and disadvantages of polycrystalline photovoltaic panels
- The price of polycrystalline solar panels is lower than monocrystalline ones. They are easier to make, using multiple silicon crystals;
- It does not involve exhaustion of raw material;
- Can be used with batteries;
- The manufacturing process requires very little fossil fuel.
- Lower efficiency compared to monocrystalline panels, resulting from the lack of pure silicon;
- Covers a larger area than monocrystalline panels;
- They may have a shorter lifespan;
- It deteriorates easily at high temperatures.
Thin film photovoltaic panels
Thin film photovoltaic panels have lower efficiency and capacity to produce electricity than monocrystalline and polycrystalline.
Characteristics of thin-layer panels
- The efficiency of converting solar energy into electricity is about 11%;
- Both their price and installation are cheaper than the other two types of panels. Thin layer panels weigh much less and are easier to handle;
- Most thin-film solar panels are often made from cadmium telluride, amorphous silicon, and copper selenide, each of which has a unique manufacturing process.
So now we know what are the main differences between polycrystalline and thin film photovoltaic panels. If you still don’t know which of these panels are more suitable for your home, our experts are at your disposal for any questions you may have.


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Does Alex Rider appear in Russian roulette?
Followed by. What does it take to make a killer? Russian Roulette is the tenth novel in the Alex Rider series, written by Anthony Horowitz. It serves as a prequel or spin-off to the Alex Rider series, but it will mainly focus on the childhood of the assassin that appears in many of the books, Yassen Gregorovich.
How old is Alex Rider in Russian roulette?
An international contract killer has been given his orders. His next target is a fourteen-year-old spy by the name of Alex Rider.
Is Yassen Gregorovich Russian?
Yassen (originally ‘Yasha’ Gregorovich) was born in the village of Estrov, in Russia.
What is Russian roulette?
To gamble foolishly on a risky or potentially ruinous business. The expression refers to a deadly game in which a participant loads a revolver with one bullet, spins the cylinder, and fires at his own head: “If you drink and drive, you’re playing Russian roulette with your life and the lives of others.”
What should I read if I like Alex Rider?
If You Like Alex Rider…
- Traitor Series by Andy McNab.
- Spartan Series by Michael Ford.
- Sure Fire by Jack Higgins.
- Ranger’s Apprentice Series by John Flanagan.
- The Door of No Return by Sarah Mussi.
- Maximum Ride Series by James Patterson.
- Thieves Like Us by Stephen Cole.
- Streams of Babel by Carol Plum-Ucci.
Will there be a 15th Alex Rider book?
Walker Books is marking the 15th anniversary of Anthony Horowitz’s first Alex Rider novel by releasing new editions of the entire series. Walker Books is marking the 15th anniversary of Anthony Horowitz’s first Alex Rider novel by releasing new editions of the entire series.
Where is Estrov?
Estrov was the Russian town where Yassen Gregorovitch was born and grew up in. The town only appeared in spin-off novel Russian Roulette.
Is Russian roulette a prequel?
Russian Roulette/Preceded by
Who is Alex riders dad?
Alex Rider is a title character and the protagonist of the Alex Rider novel series by British author Anthony Horowitz….Alex Rider (character)
|Family||John Rider (father, deceased), Helen Beckett (mother, deceased), Ian Rider (uncle, deceased), Jack Starbright (guardian)|
Is Russian roulette capitalized?
The phrase playing Russian roulette soon came to mean taking any gamble with as much at stake as a bullet in the chamber of a pistol. Related phrases are play Russian roulette, plays Russian roulette, played Russian roulette. Note that Russia is capitalized, because it is a proper noun.
Has anyone died Russian roulette?
On September 10, 1976, Finnish magician Aimo Leikas killed himself in front of a crowd while performing his Russian roulette act in Hartola. He had been performing the act for about a year, selecting six bullets from a box of assorted live and dummy ammunition.