text:
}},
Note = {{5th Congrees of the Immunology-of-Diabetes-Society, MADRAS, INDIA, FEB
   14-16, 2001}},
Organization = {{Novo Nordisk Pharma India Ltd; Natl Inst Hlth; Peptor Ltd; Bayer;
   Juvenile Diabet Fdn Int; Novo Nordisk A/S}},
Abstract = {{Blood cells from subjects with hypertension and/or diabetes mellitus
   have been successfully studied in the past to gain insight into
   pathological alterations of several signal transduction pathways.
   Diabetes mellitus is also considered to be a disease of abnormal
   cellular Ca2+ metabolism, as metabolic derangements of Ca2+, transport
   have been noticed both in the prediabetic state and as a consequence of
   hyperglycemia and oxidative stress. In this report, we used peripheral
   blood lymphocytes from type 2 diabetes patients and control subjects to
   study and delineate different mechanisms of Ca2+ turnover that determine
   the level of cytosolic calcium (Ca-i). While demonstrating the specific
   Ca2+ turnover alterations, we suggest that insights into the
   pathophysiology of diabetic complications originating from signal
   transduction defects could be conveniently studied using blood cell
   types such as lymphocytes and that such studies could lead to the
   identification of new molecular drug targets.}},
ISSN = {{0077-8923}},
ISBN = {{1-57331-362-9}},
Unique-ID = {{ISI:000176952100073}},
}

@inproceedings{ ISI:000176952100079,
Author = {Rajan, SK and Gokhale, SM},
Editor = {{Sanjeevi, CB}},
Title = {{Cardiovascular function in patients with insulin-dependent diabetes
   mellitus - A study using noninvasive methods}},
Booktitle = {{IMMUNOLOGY OF DIABETES: AUTOIMMUNE MECHANISMS AND THE PREVENTION AND
   CURE OF TYPE 1 DIABETES}},
Series = {{ANNALS OF THE NEW YORK ACADEMY OF SCIENCES}},
Year = {{2002}},
Volume = {{958}},
Pages = {{425-430}},
Note = {{5th Congrees of the Immunology-of-Diabetes-Society, MADRAS, INDIA, FEB
   14-16, 2001}},
Organization = {{Novo Nordisk Pharma India Ltd; Natl Inst Hlth; Peptor Ltd; Bayer;
   Juvenile Diabet Fdn Int; Novo Nordisk A/S}},
Abstract = {{This study used noninvasive methods to assess disturbance in
   cardiovascular function in insulin-dependent diabetes mellitus (IDDM)
   patients. Cardiovascular function and the autonomic nervous system of 38
   IDDM patients were assessed, and the presence or absence of left
   ventricular dysfunction was determined. Fifty-six percent of the
   patients were found to have autonomic neuropathy. Twelve percent had
   left ventricular diastolic dysfunction; none had left ventricular
   systolic dysfunction. Heart disease in IDDM patients was found to
   constitute a separate entity, termed diabetic cardiomyopathy. All IDDM
   patients with left ventricular diastolic dysfunction had evidence of
   autonomic neuropathy. However, there was no correlation with left
   ventricular systolic dysfunction. Also, there was no correlation between
   left ventricular dysfunction and microvascular complications of diabetes
   mellitus.}},
ISSN = {{0077-8923}},
ISBN = {{1-57331-362-9}},
Unique-ID = {{ISI:000176952100079}},
}

@inproceedings{ ISI:000177601200126,
Author = {Kim, YS and Sunwoo, S and Sung, EJ and Kim, SW},
Book-Group-Author = {{KORUS
   KORUS}},
Title = {{A retrospective cohort study on the risk factors for
   noninsulin-dependent diabetes mellitus (NIDDM) in Korea}},
Booktitle = {{KORUS-2002: 6TH RUSSIAN-KOREAN INTERNATIONAL SYMPOSIUM ON SCIENCE AND
   TECHNOLOGY, PROCEEDINGS}},
Year = {{2002}},
Pages = {{502-507}},
Note = {{6th Russian-Korean International Symposium on Science and Technology
   (KORUS-2002), NOVOSIBIRSK STATE TECH UNIV, NOVOSIBIRSK, RUSSIA, JUN
   24, 2001-JUN 30, 2002}},
Organization = {{Univ Ulsan; Tomsk Polytech Univ; IEEE Electron Devices Soc}},
ISBN = {{0-7803-7427-4}},
Unique-ID = {{ISI:000177601200126}},
}

@inproceedings{ ISI:000177158000010,
Author = {Kozak, LP and Rossmeisl, M},
Editor = {{Klimes, I and Sebokova, E and Howard, BV and Ravussin, E}},
Title = {{Adiposity and the development of diabetes in mouse genetic models}},
Booktitle = {{LIPIDS AND INSULIN RESISTANCE: THE ROLE OF FATTY ACID METABOLISM AND
   FUEL PARTITIONING}},
Series = {{ANNALS OF THE NEW YORK ACADEMY OF SCIENCES}},
Year = {{2002}},
Volume = {{967}},
Pages = {{80-87}},
Note = {{4th International Meeting on Lipids and Insulin Resistance, SMOLENICE
   CASTLE, SLOVAKIA, AUG 29-SEP 02, 2001}},
Organization = {{Astra Zeneca; European Commiss; Berlin Chemie; Coca Cola Beverages; Eli
   Lilly; Fournier Labs; GlaxoSmithKline; Lipha Merck; NY Acad Sci; Novo
   Nordisk; Pfizer; Roche Diagnost Syst; Servier; Slovak Diabetes Soc}},
Abstract = {{While it is certain from the analysis of transgenic models of
   lipodystrophy that a critical mass of adipose tissue is necessary to
   prevent the development of diabetes, the reasons why diabetes develops
   in one obese individual and not in another need to be further
   investigated. This is also one of the major questions associated with
   the current obesity epidemic and the development of the metabolic
   syndrome. The severe lipodystrophic models do not really address this
   big problem. In this presentation, we point out that models exist among
   inbred strains of mice that can contribute towards finding answers to
   the diabetes found in the metabolic syndrome. The differences
   responsible for the phenotypic variations are undoubtedly multifactorial
   and involve many genes, but there are powerful genetic resources to
   investigate these problems.}},
ISSN = {{0077-8923}},
ISBN = {{1-57331-368-8}},
ResearcherID-Numbers = {{Rossmeisl, Martin/C-1892-2012}},
Unique-ID = {{ISI:000177158000010}},
}

@inproceedings{ ISI:000177158000040,
Author = {Collier, G and Walder, K and De Silva, A and Tenne-Brown, J and
   Sanigorski, A and Segal, D and Kantham, L and Augert, G},
Editor = {{Klimes, I and Sebokova, E and Howard, BV and Ravussin, E}},
Title = {{New approaches to gene discovery with animal models of obesity and
   diabetes}},
Booktitle = {{LIPIDS AND INSULIN RESISTANCE: THE ROLE OF FATTY ACID METABOLISM AND
   FUEL PARTITIONING}},
Series = {{ANNALS OF THE NEW YORK ACADEMY OF SCIENCES}},
Year = {{2002}},
Volume = {{967}},
Pages = {{403-413}},
Note = {{4th International Meeting on Lipids and Insulin Resistance, SMOLENICE
   CASTLE, SLOVAKIA, AUG 29-SEP 02, 2001}},
Organization = {{Astra Zeneca; European Commiss; Berlin Chemie; Coca Cola Beverages; Eli
   Lilly; Fournier Labs; GlaxoSmithKline; Lipha Merck; NY Acad Sci; Novo
   Nordisk; Pfizer; Roche Diagnost Syst; Servier; Slovak Diabetes Soc}},
Abstract = {{DNA-based approaches to the discovery of genes contributing to the
   development of type 2 diabetes have not been very successful despite
   substantial investments of time and money. The multiple gene-gene and
   gene-environment interactions that influence the development of type 2
   diabetes mean that DNA approaches are not the ideal tool for defining
   the etiology of this complex disease. Gene expression-based technologies
   may prove to be a more rewarding strategy to identify diabetes candidate
   genes. There are a number of RNA-based technologies available to
   identify genes that are differentially expressed in various tissues in
   type 2 diabetes. These include differential display polymerase chain
   reaction (ddPCR), suppression subtractive hybridization (SSH), and cDNA
   microarrays. The power of new technologies to detect differential gene
   expression is ideally suited to studies utilizing appropriate animal
   models of human disease. We have shown that the gene expression
   approach, in combination with an excellent animal model such as the
   Israeli sand rat (Psammomys obesus), can provide novel genes and
   pathways that may be important in the disease process and provide novel
   therapeutic approaches. This paper will describe a new gene discovery,
   beacon, a novel gene linked with energy intake. As the functional
   characterization of novel genes discovered in our laboratory using this
   approach continues, it is anticipated that we will soon be able to
   compile a definitive list of genes that are important in the development
   of obesity and type 2 diabetes.}},
ISSN = {{0077-8923}},
ISBN = {{1-57331-368-8}},
Unique-ID = {{ISI:000177158000040}},
}

@inproceedings{ ISI:000180194800104,
Author = {Schlotthauer, G and Nicolini, GA and Gamero, LG and Torres, ME},
Book-Group-Author = {{IEEE
   IEEE
   IEEE}},
Title = {{Type I diabetes: Modeling, identification and non-linear model
   predictive control}},
Booktitle = {{SECOND JOINT EMBS-BMES CONFERENCE 2002, VOLS 1-3, CONFERENCE
   PROCEEDINGS: BIOENGINEERING - INTEGRATIVE METHODOLOGIES, NEW
   TECHNOLOGIES}},
Series = {{PROCEEDINGS OF ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE ENGINEERING
   IN MEDICINE AND BIOLOGY SOCIETY}},
Year = {{2002}},
Pages = {{226-227}},
Note = {{24th Annual International Conference of the
   Engineering-in-Medicine-and-Biology-Society/Annual Fall Meeting of the
   Biomedical-Engineering-Society (EMBS 2002 BMES), HOUSTON, TX, OCT 23-26,
   2002}},
Organization = {{Engn Med \& Biol Soc; Biomed Engn Soc; Natl Sci Fdn; Natl Inst Hlth;
   Natl Inst Biomed Imaging \& Bioengn; Whitaker Fdn}},
Abstract = {{Patients with Diabetic Type I nearly always need a therapy with insulin.
   The most desirable treatment would be to mimic the operation of a normal
   pancreas. In this work we model a patient affected with this pathology,
   identify it with a neural network, and evaluate a control strategy known
   as Nonlinear Model Predictive Control as an approach to command an
   insulin pump using the subcutaneous route.}},
ISSN = {{1094-687X}},
ISBN = {{0-7803-7612-9}},
ResearcherID-Numbers = {{Torres, Maria/A-5982-2010}},
ORCID-Numbers = {{Torres, Maria/0000-0002-1512-9507}},
Unique-ID = {{ISI:000180194800104}},
}

@inproceedings{ ISI:000180194800105,
Author = {Wilinska, ME and Schaller, H and Schaupp, L and Pieber, TR and Chassin,
   LJ and Hovorka, R},
Book-Group-Author = {{IEEE
   IEEE
   IEEE}},
Title = {{Modeling interstitial glucose kinetics in subjects with type 1 diabetes
   during physiological conditions}},
Booktitle = {{SECOND JOINT EMBS-BMES CONFERENCE 2002, VOLS 1-3, CONFERENCE
   PROCEEDINGS: BIOENGINEERING - INTEGRATIVE METHODOLOGIES, NEW
   TECHNOLOGIES}},
Series = {{PROCEEDINGS OF ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE ENGINEERING
   IN MEDICINE AND BIOLOGY SOCIETY}},
Year = {{2002}},
Pages = {{228-229}},
Note = {{24th Annual International Conference of the
   Engineering-in-Medicine-and-Biology-Society/Annual Fall Meeting of the
   Biomedical-Engineering-Society (EMBS 2002 BMES), HOUSTON, TX, OCT 23-26,
   2002}},
Organization = {{Engn Med \& Biol Soc; Biomed Engn Soc; Natl Sci Fdn; Natl Inst Hlth;
   Natl Inst Biomed Imaging \& Bioengn; Whitaker Fdn}},
Abstract = {{The relationship between interstitial glucose (IG) and plasma glucose
   (PG) is not fully understood. We investigated this relationship during
   physiological conditions in subjects with type I diabetes. Nine subjects
   were studied over 12h. IG was measured every 30 and PG every 15min. Nine
   alternative compartment models were formulated assessing one or more
   effects including insulin and saturability effects. Model parameters
   were estimated from IG using PG as forcing function. Acceptable
   precision (CV<100\%) was achieved for most parameter estimates. Model
   selection process was based on Akaike criterion. The model which best
   explained the experimental data included both the effect of insulin on
   glucose transfer from plasma to interstitial fluid (IF) and the presence
   of zero-order removal of glucose from IF.}},
ISSN = {{1094-687X}},
ISBN = {{0-7803-7612-9}},
ResearcherID-Numbers = {{Hovorka, Roman/J-4123-2016}},
ORCID-Numbers = {{Hovorka, Roman/0000-0003-2901-461X}},
Unique-ID = {{ISI:000180194800105}},
}

@article{ ISI:000172890600019,
Author = {Plougmann, S and Hejlesen, OK and Cavan, DA},
Title = {{DiasNet - a diabetes advisory system for communication and education via
   the internet}},
Journal = {{INTERNATIONAL JOURNAL OF MEDICAL INFORMATICS}},
Year = {{2001}},
Volume = {{64}},
Number = {{2-3, SI}},
Pages = {{319-330}},
Month = {{DEC}},
Note = {{16th Medical Informatics Europe Congress/45th Annual Congress of the
   German-Association-for-Medical-Informatics-Biometry-and-Epidemiology,
   Hanover, GERMANY, JUN, 2000}},
Organization = {{German Assoc Med Informat Biometry \& Epidemiol}},
Abstract = {{Intensive diabetes treatment can lead to a substantial reduction of the
   rate of the complications associated with diabetes. However, a number of
   patients may have poor control despite specialist care, and this along
   with devolution of care to non-specialists suggests that alternative
   interventions should be developed. The present paper describes an
   Internet based system where more emphasis is put on patient empowerment,
   the keywords being education and communication. The DiasNet system is
   based on a well documented decision support system, Dias, designed for
   use by clinicians. The scope of DiasNet has been widened from being used
   by clinicians to give advice on insulin dose, to also being used by
   patients as a tool for education and communication. Patients can
   experiment with their own data, adjusting insulin doses or meal sizes.
   In this way different therapeutic and dietary alternatives can be tried
   out, allowing the patient to gain experience in achieving glycaemic
   control. DiasNet is implemented in JAVA according to the client/server
   principle, enabling a new way of communication between patient and
   clinician: in case of any problems, the patient simply phones the
   clinician, who immediately, using his or her office PC, can take a look
   at the data the patient has entered. (C) 2001 Published by Elsevier
   Science Ireland Ltd.}},
DOI = {{10.1016/S1386-5056(01)00214-3}},
ISSN = {{1386-5056}},
Unique-ID = {{ISI:000172890600019}},
}

@article{ ISI:000172512900004,
Author = {Park, J and Edington, DW},
Title = {{A sequential neural network model for diabetes prediction}},
Journal = {{ARTIFICIAL INTELLIGENCE IN MEDICINE}},
Year = {{2001}},
Volume = {{23}},
Number = {{3}},
Pages = {{277-293}},
Month = {{NOV}},
Abstract = {{This paper presents a neural network (NN) model to evaluate an existing
   Health Risk Appraisal (HRA)(2) for diabetes prediction over 3 years
   (1996-1998) based on a simulated learning algorithm on individual
   prognostic process, using the repeatedly measured HRAs of 6142
   participants.
   The approach uses a sequential multi-layered perceptron (SMLP) with
   backpropagation learning, and an explicit model of time-varying inputs
   along with the sequentially obtained prediction probability, which was
   obtained by embedding a multivariate logistic function for consecutive
   years.
   The study captures the time-sensitive feature of associating risk
   factors as predictors to the occurrence of diabetes in the corresponding
   period. This approach outperforms the baseline classification and
   regression models in terms of gains (average profit: 0.18) and
   sensitivity (86.04\%) for a test data.
   The result enables a time-sensitive disease prevention and management
   program as a prospective effort. (C) 2001 Elsevier Science B.V. All
   rights reserved.}},
DOI = {{10.1016/S0933-3657(01)00086-0}},
ISSN = {{0933-3657}},
Unique-ID = {{ISI:000172512900004}},
}

@article{ ISI:000172284400006,
Author = {Fabietti, PG and Calabrese, G and Iorio, M and Bistoni, S and Brunetti,
   P and Sarti, E and Benedetti, MM},
Title = {{A mathematical model describing the glycemic response of diabetic
   patients to meal and i.v. infusion of insulin}},
Journal = {{INTERNATIONAL JOURNAL OF ARTIFICIAL ORGANS}},
Year = {{2001}},
Volume = {{24}},
Number = {{10}},
Pages = {{736-742}},
Month = {{OCT}},
Abstract = {{Nine type 1 diabetic patients were studied for 24 hours. During this
   period they were given three calibrated meals. The glycemia was
   feedback-controlled by means of an artificial pancreas. The blood
   concentration of glucose and the infusion speed of the insulin were
   measured every minute. The experimental data referring to each of the
   three meals were used to estimate the parameters of a mathematical model
   suitable for describing the glycemic response of diabetic patients at
   meals and at the i.v. infusion of exogenous insulin. From the estimate a
   marked dispersion of the parameters was found, both interindividual and
   intraindividual. Nevertheless the models thus obtained seem to be usable
   for the synthesis of a feedback controller, especially in view of
   creating a portable artificial pancreas that now seems possible owing to
   the realization (so far experimental) of sufficiently reliable glucose
   concentration sensors.}},
ISSN = {{0391-3988}},
Unique-ID = {{ISI:000172284400006}},
}

@inproceedings{ ISI:000174951000040,
Author = {Montani, S and Bellazzi, R and Portinale, L and Stefanelli, M},
Editor = {{Blanzieri, E and Portinale, L}},
Title = {{Evaluating a multi-modal reasoning system in diabetes care}},
Booktitle = {{ADVANCES IN CASE-BASED REASONING, PROCEEDINGS}},
Series = {{LECTURE NOTES IN ARTIFICIAL INTELLIGENCE}},
Year = {{2001}},
Volume = {{1898}},
Pages = {{467-478}},
Note = {{5th European Workshop on Case-Based Reasoning (EWCBR 2000), TRENT,
   ITALY, SEP 06-09, 2000}},
Abstract = {{In the context of Insulin Dependent Diabetes Mellitus care, we developed
   a decision support system that relies on a tight integration of Case
   Based Reasoning and Rule Based Reasoning methodologies. In this paper,
   we aim at presenting the evaluation strategy we have defined to test the
   system accuracy, safety and reliability, and the first results obtained
   both on simulated and on real patients data. Reliability was positively
   judged by a group of expert diabetologists; an increase in the
   performances of the system is foreseen as new knowledge will be
   acquired, through its usage in clinical practice.}},
ISSN = {{0302-9743}},
ISBN = {{3-540-67933-2}},
Unique-ID = {{ISI:000174951000040}},
}

@inproceedings{ ISI:000172901700151,
Author = {Bissell, R and Fernando, B and Scobie, I and Briscoe, E and Simmonds, D
   and Trigg, P and McDowell, D},
Editor = {{Patel, VL and Rogers, R and Haux, R}},
Title = {{Implementing real-time system interfaces to support the shared care of
   diabetes patients}},
Booktitle = {{MEDINFO 2001: PROCEEDINGS OF THE 10TH WORLD CONGRESS ON MEDICAL
   INFORMATICS, PTS 1 AND 2}},
Series = {{STUDIES IN HEALTH TECHNOLOGY AND INFORMATICS}},
Year = {{2001}},
Volume = {{84}},
Pages = {{599-603}},
Note = {{10th World Congress on Medical Informatics (MEDINFO 2001), LONDON,
   ENGLAND, 2001}},
Organization = {{McGill Univ, Ctr Med Educ; Columbia Univ, Dept Med Informat}},
Abstract = {{This paper describes a collaborative project: the Medway integrated Care
   Support (MEDICS) Project, involving GP and Hospital system suppliers, an
   NHS Trust, General Practitioners and the NHS Information Authority. The
   objective of the project is to assess the Object Management Group's
   (OMG/COPBAMed) Clinical Observations Access Service (COAS) standard, by
   demonstrating its use in implementing a real-time interface between a GP
   system and a hospital clinical system, supporting the shared care of
   diabetes patient.}},
ISSN = {{0926-9630}},
ISBN = {{1-58603-194-5}},
Unique-ID = {{ISI:000172901700151}},
}

@inproceedings{ ISI:000172097600170,
Author = {Svacina, S and Haas, T and Matoulek, M and Hilgertova, J},
Editor = {{Kerckhoffs, EJH and Snorek, M}},
Title = {{Derivative and proportional regulation in physiology of diabetes:
   Modelling of ketone bodies forming as new example}},
Booktitle = {{MODELLING AND SIMULATION 2001}},
Year = {{2001}},
Pages = {{1009-1011}},
Note = {{15th European Simulation Multiconference (ESM 2001), CTU, PRAGUE, CZECH
   REPUBLIC, JUN 06-09, 2001}},
Organization = {{Soc Comp Simulat Int; ASIM; Arbeitsgemeinsch Simulat}},
Abstract = {{There are two regulations in diabetes physiology using perhaps the rapid
   derivative regulator: Early phase of insulin secretion in intravenous
   glucose tolerance test and hormone reaction to hypoglycemia. We have
   found another rapid insulin dependent regulation of ketone
   (betahyroxybutyrate- BOH) forming. The modelling of BOH forming is
   determined by following parameters: nitrogen balance, glucose and
   insulin change during weight reduction. Early ketone bodies forming in
   obesity is an example of dual reaction. The first phase of ketone bodies
   response is a quick reaction to insulin level decrease. The second phase
   forms a plateau with steady state of betahydroxybutyrate forming and
   metabolic extraction. A model with derivative regulator should be used
   for modelling of ketone forming in obesity and diabetes.}},
ISBN = {{1-56555-225-3}},
Unique-ID = {{ISI:000172097600170}},
}

@inproceedings{ ISI:000178871900568,
Author = {Santoso, L and Mareels, IMY},
Book-Group-Author = {{IEEE
   IEEE
   IEEE}},
Title = {{Markov modelling and adaptive control in management of type 1 diabetes
   mellitus}},
Booktitle = {{PROCEEDINGS OF THE 23RD ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE
   ENGINEERING IN MEDICINE AND BIOLOGY SOCIETY, VOLS 1-4: BUILDING NEW
   BRIDGES AT THE FRONTIERS OF ENGINEERING AND MEDICINE}},
Series = {{PROCEEDINGS OF ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE ENGINEERING
   IN MEDICINE AND BIOLOGY SOCIETY}},
Year = {{2001}},
Volume = {{23}},
Number = {{1-4}},
Pages = {{2068-2071}},
Note = {{23rd Annual International Conference of the
   IEEE-Engineering-in-Medicine-and-Biology-Society, ISTANBUL, TURKEY, OCT
   25-28, 2001}},
Organization = {{Natl Sci Fdn; TUBITAK, Sci \& Tech Res Ctr Turkey; ISIK Univ; COMNET;
   EREL Techno Grp; GUZEL SANATLAR Printinghouse; JOHNSON\&JOHNSON Med;
   PFIZER; SIEMENS Med; TURKCELL Iletism Hizmetler A S; ALSTOM Elect Ltd
   Co; GANTEK Technol \& SUN Microsyst; TURCOM Co Grp}},
Abstract = {{We present management of diabetes mellitus as a constrained control
   problem, with very sparse sampling, where no information on dynamics are
   available. This problem has been approached through modelling of the
   system as a Markov decision process, and through direct derivation of
   control rules. Considering the complexity of the system constraints
   imposed by lack of information, we argue that non-model based control is
   the best and simplest method for this problem.}},
ISSN = {{1094-687X}},
ISBN = {{0-7803-7211-5}},
ResearcherID-Numbers = {{Mareels, Iven/H-2745-2012}},
ORCID-Numbers = {{Mareels, Iven/0000-0002-9832-5959}},
Unique-ID = {{ISI:000178871900568}},
}

@article{ ISI:000165429800051,
Author = {Lee, HC and Kim, SJ and Kim, KS and Shin, HC and Yoon, JW},
Title = {{Remission in models of type 1 diabetes by gene therapy using a
   single-chain insulin analogue (Retracted Article. See vol 458, pg 660,
   2009)}},
Journal = {{NATURE}},
Year = {{2000}},
Volume = {{408}},
Number = {{6811}},
Pages = {{483-488}},
Month = {{NOV 23}},
Abstract = {{A cure for diabetes has long been sought using several different
   approaches, including islet transplantation, regeneration of beta cells
   and insulin gene therapy(1). However, permanent remission of type 1
   diabetes has not yet been satisfactorily achieved. The development of
   type 1 diabetes results from the almost total destruction of
   insulin-producing pancreatic beta cells by autoimmune responses specific
   to beta cells(2-6). Standard insulin therapy may not maintain blood
   glucose concentrations within the relatively narrow range that occurs in
   the presence of normal pancreatic beta cells(7). We used a recombinant
   adeno-associated virus (rAAV) that expresses a single-chain insulin
   analogue (SIA), which possesses biologically active insulin activity
   without enzymatic conversion, under the control of hepatocyte-specific
   L-type pyruvate kinase (LPK) promoter, which regulates SIA expression in
   response to blood glucose levels. Here we show that SIA produced from
   the gene construct rAAV-LPK-SIA caused remission of diabetes in
   streptozotocin-induced diabetic rats and autoimmune diabetic mice for a
   prolonged time without any apparent side effects. This new SIA gene
   therapy may have potential therapeutic value for the cure of autoimmune
   diabetes in humans.}},
DOI = {{10.1038/35044106}},
ISSN = {{0028-0836}},
Unique-ID = {{ISI:000165429800051}},
}

@article{ ISI:000087467400008,
Author = {Hernando, ME and Gomez, EJ and Corocy, R and del Pozo, F},
Title = {{Evaluation of DIABNET, a decision support system for therapy planning in
   gestational diabetes}},
Journal = {{COMPUTER METHODS AND PROGRAMS IN BIOMEDICINE}},
Year = {{2000}},
Volume = {{62}},
Number = {{3}},
Pages = {{235-248}},
Month = {{JUL}},
Note = {{34th Meeting of the European-Association-for-the-Study-of-Diabetes,
   BARCELONA, SPAIN, SEP 08-12, 1998}},
Organization = {{European Assoc Study Diabet}},
Abstract = {{DIABNET is a knowledge-based system designed to aid doctors with therapy
   planning in gestational diabetes. The system core is a qualitative
   model, implemented by a Causal Probabilistic Network, that is able to
   detect the insulin effectiveness on a daily basis. DIABNET analyses
   monitoring data and proposes quantitative changes in insulin therapy and
   qualitative diet modifications. This paper proposes an evaluation
   methodology to assess the system performance when working in a real
   scenario. The methodology manages the absence of a gold standard and
   includes. a subjective analysis based on questionnaires and an objective
   analysis based on a quantitative comparison of the system's and experts'
   proposals. The paper also shows the results of two experiments in which
   expert diabetologists evaluated the therapeutical advice provided by
   DIABNET during the follow up of 9 patients with gestational diabetes.
   DIABNET detected the need of a therapy modification in 92\% of the cases
   showing its appropriateness for automatic alarm generation. Around 80\%
   of the proposals were accepted by experts. The evaluation results are
   encouraging and allow characterisation of the system's performance when
   proposing therapy modifications. Evaluation in its turn helps to refine
   the knowledge managed by DIABNET and enables us to look towards the
   further clinical use of DIABNET as a decision tool in gestational
   diabetes integrated in a telemedicine service. (C) 2000 Elsevier Science
   Ireland Ltd. All rights reserved.}},
DOI = {{10.1016/S0169-2607(00)00070-5}},
ISSN = {{0169-2607}},
ResearcherID-Numbers = {{Hernando, M. Elena/A-9575-2008
   DEL POZO GUERRERO, FRANCISCO/H-6668-2015}},
ORCID-Numbers = {{Hernando, M. Elena/0000-0001-6182-313X
   DEL POZO GUERRERO, FRANCISCO/0000-0001-9919-9125}},
Unique-ID = {{ISI:000087467400008}},
}

@article{ ISI:000086633100007,
Author = {Gorman, CA and Zimmerman, BR and Smith, SA and Dinneen, SF and Knudsen,
   JB and Holm, D and Jorgensen, B and Bjornsen, S and Planet, K and
   Hanson, P and Rizza, RA},
Title = {{DEMS - a second generation diabetes electronic management system}},
Journal = {{COMPUTER METHODS AND PROGRAMS IN BIOMEDICINE}},
Year = {{2000}},
Volume = {{62}},
Number = {{2}},
Pages = {{127-140}},
Month = {{JUN}},
Abstract = {{Diabetes electronic management system (DEMS) is a component-based
   client/server application, written in Visual C + + and Visual Basic,
   with the database server running Sybase System 11. DEMS is built
   entirely with a combination of dynamic link libraries (DLLs) and ActiveX
   components - the only exception is the DEMS.exe. DEMS is a chronic
   disease management system for patients with diabetes. It is used at the
   point of care by all members of the diabetes team including physicians,
   nurses, dieticians, clinical assistants and educators. The system is
   designed fur maximum clinical efficiency and facilitates appropriately
   supervised delegation of care. Dispersed clinical sites may be
   supervised from a central location. The system is designed for ease of
   navigation; immediate provision of many types of automatically generated
   reports; quality audits; aids to compliance with good care guidelines;
   and alerts, advisories, prompts, and warnings that guide the care
   provider. The system now contains data on over 34 000 patients and is in
   daily use at multiple sites. (C) 2000 Elsevier Science Ireland Ltd. All
   rights reserved.}},
DOI = {{10.1016/S0169-2607(99)00054-1}},
ISSN = {{0169-2607}},
Unique-ID = {{ISI:000086633100007}},
}

@inproceedings{ ISI:000167100600064,
Author = {Pruna, S and Harris, ND and Dixon, R},
Editor = {{Laxminarayan, S}},
Title = {{Black Sea Tele Diab: Building an information system for management of
   diabetes}},
Booktitle = {{2000 IEEE EMBS INTERNATIONAL CONFERENCE ON INFORMATION TECHNOLOGY
   APPLICATIONS IN BIOMEDICINE, PROCEEDINGS}},
Series = {{IEEE Engineering in Medicine and Biology Society Annual Conference
   (EMBS)}},
Year = {{2000}},
Pages = {{284-289}},
Note = {{Joint Meeting of the 3rd IEEE EMBS International Conference on
   Information Technology Applications in Biomedicine/3rd Workshop of the
   Int-Telemedical-Information-Society, ARLINGTON, VA, NOV 09-10, 2000}},
Organization = {{IEEE, Engn Med \& Biol Soc; Int Telemed Informat Soc}},
Abstract = {{The main objective of the work was to enable quality improvement of
   diabetes services through better monitoring of clinical care in
   countries of the Black Sea area. We have created a diabetes health
   record system based on the Good European Health Record (GEHR). Diabetes
   has served as a prototype for development of a model for chronic illness
   care. This Architecture provides a common data structure for electronic
   health care records, taking into account ethical, legal, security, and
   educational requirements. A wide range of data types (including
   laboratory data, photographs, bio-signals, etc.), created in many
   sources can be recorded The system offers support for the process of
   clinical care and medical education. The BSTD system was developed using
   a modular design and object oriented method approach. The Patient
   Records Function offers options for the management of the EHCRs
   (creation/correction/visualization), such as registration of a new
   patient and recording of his first Basic Information Sheet; the
   recording of a new sheet; the correction/visualization of the sheet; the
   recording of data about the patient's death, etc. The System
   Administration Function allows: the definition of the health care
   facility; the management of persons that use the system or fills in the
   sheets; the management of passwords and access rights for the users; the
   management of units used for measurements; the management of the
   interface of the system. The system is currently undergoing formal
   clinical evaluation in diabetes centers from Romania, Ukraine and
   Moldavia.}},
DOI = {{10.1109/ITAB.2000.892402}},
ISBN = {{0-7803-6449-X}},
Unique-ID = {{ISI:000167100600064}},
}

@inproceedings{ ISI:000088670700017,
Author = {Yamamoto, Y and Yamagishi, SI and Yonekura, H and Doi, T and Tsuji, H
   and Kato, I and Takasawa, S and Okamoto, H and Abedin, J and Tanaka, N
   and Sakurai, S and Migita, H and Unoki, H and Wang, H and Zenda, T and
   Wu, PS and Segawa, Y and Higashide, T and Kawasaki, K and Yamamoto, H},
Editor = {{Numano, F and Gimbrone, MA}},
Title = {{Roles of the AGE-RAGE system in vascular injury in diabetes}},
Booktitle = {{ATHEROSCLEROSIS V: THE FIFTH SARATOGA CONFERENCE}},
Series = {{ANNALS OF THE NEW YORK ACADEMY OF SCIENCES}},
Year = {{2000}},
Volume = {{902}},
Pages = {{163-172}},
Note = {{5th Saratoga International Conference on Atherosclerosis, BARCELONA,
   SPAIN, MAY 18-20, 1999}},
Organization = {{Japan Arteriosclerosis Res Fdn; Japan Atherosclerosis Soc; Japan
   Circulat Soc; Japanese Coll Angiol}},
Abstract = {{This study concerns whether advanced glycation endproducts (AGE) are
   related to microvascular derangement in diabetes, exemplified by
   pericyte loss and angiogenesis in retinopathy and by mesangial expansion
   in nephropathy, AGE caused a decrease in viable pericytes cultivated
   from bovine retina. On the other hand, AGE stimulated the growth and
   tube formation of human microvascular endothelial cells (EC), this being
   mediated by autocrine vascular endothelial growth factor. In AGE-exposed
   rat mesangial cells, type TV collagen synthesis was induced, Those AGE
   actions were dependent on a cell surface receptor for AGE (RAGE),
   because they were abolished by RAGE antisense or ribozyme, The AGE-RAGE
   system may thus participate in the development of diabetic
   microangiopathy. This proposition was supported by experiments with
   animal models; several indices characteristic of retinopathy were
   correlated with circulating AGE levels in OLETF rats. The predisposition
   to nephropathy was augmented in RAGE transgenic mice when they became
   diabetic.}},
ISSN = {{0077-8923}},
ISBN = {{1-57331-246-0}},
Unique-ID = {{ISI:000088670700017}},
}

@inproceedings{ ISI:000166896300311,
Author = {Hejlesen, OK and Jensen, KD and Lervang, HH and Cavan, DA},
Editor = {{Enderle, JD}},
Title = {{A model-based study of the long-term reaction to spontaneous
   hypoglycaemia in type 1 diabetes}},
Booktitle = {{PROCEEDINGS OF THE 22ND ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE
   ENGINEERING IN MEDICINE AND BIOLOGY SOCIETY, VOLS 1-4}},
Series = {{PROCEEDINGS OF ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE ENGINEERING
   IN MEDICINE AND BIOLOGY SOCIETY}},
Year = {{2000}},
Volume = {{22}},
Number = {{1-4}},
Pages = {{1116-1119}},
Note = {{22nd Annual International Conference of the
   IEEE-Engineering-in-Medicine-and-Biology-Society, CHICAGO, IL, JUL
   23-28, 2000}},
Organization = {{IEEE, Engn Med \& Biol Soc; Amer Assoc Physicists Med; Amer Inst Med \&
   Biol Engn; Int Union Phys \& Engn Sci Med; Int Federat Med \& Biol Engn;
   Int Org Med Phys}},
Abstract = {{Diabetes is one of the major chronic diseases and it often leads to a
   substantial loss of quality of life. However, complications can be
   relieved by intensive insulin treatment, which, unfortunately, also
   increases the frequency severe hypoglycaemia (low blood glucose), In
   1959 Somogyi described how insulin overtreatment, leading to
   hypoglycaemia, can cause prolonged hyperglycaemia (elevated blood
   glucose), but numerous studies, especially in the 80ies, have not been
   able to produce significant evidence for his theory, However, in a
   recent model-based study we have introduced a hypothesis suggesting that
   the phenomenon is clinically much more important than previously
   anticipated. The present pilot study assesses the feasibility of
   employing a computer model to data from a 32 hour intensive monitoring
   period from 3 patients to infer the reaction to spontaneous
   hypoglycaemia, thereby contributing to the verification of the
   hypothesis and to an understanding of the phenomenon. Regarded
   separately, the number of patients in the present study is far to small
   to justify any conclusion, but, taken together with our previous
   studies, there seems to be substantial evidence for the existence of the
   hypoglycaemic counter-regulation. Furthermore, the present study seems
   to suggest that cortisol release, triggered by the hypoglycaemic attack,
   may play an important role in provoking the hypoglycaemic
   counter-regulation, and that further studies are clearly required to
   verify this hypothesis.}},
ISSN = {{1094-687X}},
ISBN = {{0-7803-6466-X}},
Unique-ID = {{ISI:000166896300311}},
}

@inproceedings{ ISI:000166896300783,
Author = {Curilem, GMJ and Brasil, LM and Sandoval, RCB and Coral, MHC and de
   Azevedo, FM and Marques, JLB},
Editor = {{Enderle, JD}},
Title = {{Considerations for the design of a tutoring system applied to diabetes}},
Booktitle = {{PROCEEDINGS OF THE 22ND ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE
   ENGINEERING IN MEDICINE AND BIOLOGY SOCIETY, VOLS 1-4}},
Series = {{PROCEEDINGS OF ANNUAL INTERNATIONAL CONFERENCE OF THE IEEE ENGINEERING
   IN MEDICINE AND BIOLOGY SOCIETY}},
Year = {{2000}},
Volume = {{22}},
Number = {{1-4}},
Pages = {{2811-2814}},
Note = {{22nd Annual International Conference of the
   IEEE-Engineering-in-Medicine-and-Biology-Society, CHICAGO, IL, JUL
   23-28, 2000}},
Organization = {{IEEE, Engn Med \& Biol Soc; Amer Assoc Physicists Med; Amer Inst Med \&
   Biol Engn; Int Union Phys \& Engn Sci Med; Int Federat Med \& Biol Engn;
   Int Org Med Phys}},
Abstract = {{Diabetes Mellitus (DM) is a chronic disease affecting a large segment of
   world's population, This article proposes the prototype design of a
   Tutoring System (TS) developed to support the educational needs related
   to DM treatment, The objective Is to guide Type 1 patients and their
   families in the better routines and cautions they should follow, to
   improve their life quality. As the TS design is the first step of the
   project of an Intelligent Tutoring System design, the entire project is
   presented but the first step, the TS design and the pedagogical issues
   are described more in detail in this article.}},
ISSN = {{1094-687X}},
ISBN = {{0-7803-6466-X}},
ResearcherID-Numbers = {{Marques, Jefferson /A-1208-2008
   Azevedo, Fernando/H-6517-2014}},
ORCID-Numbers = {{Marques, Jefferson /0000-0001-8910-4735
   }},
Unique-ID = {{ISI:000166896300783}},
}

license: MIT

license_type: permissive

branch_name: refs/heads/master

extension: bib


text:
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PR = "r1"

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repository: Terentyev/familiar-linux

repo_name: Terentyev/familiar-linux

extension: bb


text:
@misc{
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@misc{
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text:
Automatically generated by Mendeley Desktop 1.19.4
Any changes to this file will be lost if it is regenerated by Mendeley.

BibTeX export options can be customized via Preferences -> BibTeX in Mendeley Desktop

@article{Riley2003,
abstract = {We present an analysis of the first 10 weeks of the severe acute respiratory syndrome (SARS) epidemic in Hong Kong. The epidemic to date has been characterized by two large clusters - initiated by two separate "super-spread" events (SSEs) - and by ongoing community transmission. By fitting a stochastic model to data on 1512 cases, including these clusters, we show that the etiological agent of SARS is moderately transmissible. Excluding SSES, we estimate that 2.7 secondary infections were generated per case on average at the start of the epidemic, with a substantial contribution from hospital transmission. Transmission rates fell during the epidemic, primarily as a result of reductions in population contact rates and improved hospital infection control, but also because of more rapid hospital attendance by symptomatic individuals. As a result, the epidemic is now in decline, although continued vigilance is necessary for this to be maintained. Restrictions on longer range population movement are shown to be a potentially useful additional control measure in some contexts. We estimate that most currently infected persons are now hospitalized, which highlights the importance of control of nosocomial transmission.},
author = {Riley, Steven and Fraser, Christophe and Donnelly, Christl A. and Ghani, Azra C. and Abu-Raddad, Laith J. and Hedley, Anthony J. and Leung, Gabriel M. and Ho, Lai Ming and Lam, Tai Hing and Thach, Thuan Q. and Chau, Patsy and Chan, King Pan and Lo, Su Vui and Leung, Pak Yin and Tsang, Thomas and Ho, William and Lee, Koon Hung and Lau, Edith M.C. and Ferguson, Neil M. and Anderson, Roy M.},
doi = {10.1126/science.1086478},
file = {:Users/sepmein/Qsync/Papers/Riley et al.{\_}2003{\_}Transmission dynamics of the etiological agent of SARS in Hong Kong Impact of public health interventions.pdf:pdf},
issn = {00368075},
journal = {Science},
number = {5627},
pages = {1961--1966},
title = {{Transmission dynamics of the etiological agent of SARS in Hong Kong: Impact of public health interventions}},
volume = {300},
year = {2003}
}

license: Unlicense

repository: sepmein/references

repo_name: sepmein/references


text:
@article{RN428,
 author = {Vincent, B. and LaRue, R. and Sealey, V. L. and Engelke, N.},
 doi = {10.1080/0020739X.2015.1005700},
 journal = {International Journal of Mathematical Education in Science and Technology},
 number = {5},
 pages = {641-657},
 title = {Calculus Students' Early Concept Images of Tangent Lines },
 type = {Journal Article},
 volume = {46},
 year = {2015}
}


text:
DESCRIPTION = "libGLES for ZynqMP with Mali 400"

LICENSE = "Proprietary"
LIC_FILES_CHKSUM = "file://EULA;md5=82e466d0ed92c5a15f568dbe6b31089c"

inherit features_check update-alternatives

ANY_OF_DISTRO_FEATURES = "x11 fbdev wayland"
REQUIRED_MACHINE_FEATURES = "mali400"

PROVIDES += "virtual/libgles1 virtual/libgles2 virtual/egl virtual/libgbm"

FILESEXTRAPATHS:prepend := "${THISDIR}/files:"

REPO ?= "git://github.com/Xilinx/mali-userspace-binaries.git;protocol=https"
BRANCH ?= "xlnx_rel_v2023.1"
SRCREV ?= "b3a772aad859cdadc8513b11c3e995546c20e75e"
BRANCHARG = "${@['nobranch=1', 'branch=${BRANCH}'][d.getVar('BRANCH', True) != '']}"

PV = "r9p0-01rel0"
SRC_URI = " \
    ${REPO};${BRANCHARG} \
    file://egl.pc \
    file://glesv1_cm.pc \
    file://glesv1.pc \
    file://glesv2.pc \
    file://gbm.pc \
    "

PACKAGE_ARCH = "${MACHINE_ARCH}"

S = "${WORKDIR}/git"

# If were switching at runtime, we would need all RDEPENDS needed for all backends available
X11RDEPENDS = "libxdamage libxext libx11 libdrm libxfixes"
X11DEPENDS = "libxdamage libxext virtual/libx11 libdrm libxfixes"

# Don't install runtime dependencies for other backends unless the DISTRO supports it
RDEPENDS:${PN} = " \
    kernel-module-mali \
    ${@bb.utils.contains('DISTRO_FEATURES', 'x11', '${X11RDEPENDS}', '', d)} \
"

# We dont build anything but we want to avoid QA warning build-deps
DEPENDS = "\
    ${@bb.utils.contains('DISTRO_FEATURES', 'x11', '${X11DEPENDS}', '', d)} \
    ${@bb.utils.contains('DISTRO_FEATURES', 'wayland', 'wayland libdrm', '', d)} \
"

# x11 is default, set to "fbdev" , "wayland", or "headless" if required
MALI_BACKEND_DEFAULT ?= "x11"

USE_X11 = "${@bb.utils.contains("DISTRO_FEATURES", "x11", "yes", "no", d)}"
USE_FB = "${@bb.utils.contains("DISTRO_FEATURES", "fbdev", "yes", "no", d)}"
USE_WL = "${@bb.utils.contains("DISTRO_FEATURES", "wayland", "yes", "no", d)}"

MONOLITHIC_LIBMALI = "libMali.so.9.0"

do_install() {
    #Identify the ARCH type
    ${TARGET_PREFIX}gcc --version > ARCH_PLATFORM
    if grep -q aarch64 "ARCH_PLATFORM"; then
	ARCH_PLATFORM_DIR=aarch64-linux-gnu
    else
	ARCH_PLATFORM_DIR=arm-linux-gnueabihf
    fi

    # install headers
    install -d -m 0655 ${D}${includedir}/EGL
    install -m 0644 ${S}/${PV}/glesHeaders/EGL/*.h ${D}${includedir}/EGL/
    install -d -m 0655 ${D}${includedir}/GLES
    install -m 0644 ${S}/${PV}/glesHeaders/GLES/*.h ${D}${includedir}/GLES/
    install -d -m 0655 ${D}${includedir}/GLES2
    install -m 0644 ${S}/${PV}/glesHeaders/GLES2/*.h ${D}${includedir}/GLES2/
    install -d -m 0655 ${D}${includedir}/KHR
    install -m 0644 ${S}/${PV}/glesHeaders/KHR/*.h ${D}${includedir}/KHR/

    install -d ${D}${libdir}/pkgconfig
    install -m 0644 ${WORKDIR}/egl.pc ${D}${libdir}/pkgconfig/egl.pc
    install -m 0644 ${WORKDIR}/glesv2.pc ${D}${libdir}/pkgconfig/glesv2.pc
    install -m 0644 ${WORKDIR}/glesv1.pc ${D}${libdir}/pkgconfig/glesv1.pc
    install -m 0644 ${WORKDIR}/glesv1_cm.pc ${D}${libdir}/pkgconfig/glesv1_cm.pc

    install -d ${D}${libdir}
    install -d ${D}${includedir}

    cp -a --no-preserve=ownership ${S}/${PV}/${ARCH_PLATFORM_DIR}/common/*.so* ${D}${libdir}

    install -Dm 0644 ${S}/${PV}/${ARCH_PLATFORM_DIR}/headless/${MONOLITHIC_LIBMALI} ${D}${libdir}/headless/${MONOLITHIC_LIBMALI}
    ln -snf headless/${MONOLITHIC_LIBMALI} ${D}${libdir}/${MONOLITHIC_LIBMALI}

    if [ "${USE_FB}" = "yes" ]; then
        install -Dm 0644 ${S}/${PV}/${ARCH_PLATFORM_DIR}/fbdev/${MONOLITHIC_LIBMALI} ${D}${libdir}/fbdev/${MONOLITHIC_LIBMALI}
        if [ "${MALI_BACKEND_DEFAULT}" = "fbdev" ]; then
            ln -snf fbdev/${MONOLITHIC_LIBMALI} ${D}${libdir}/${MONOLITHIC_LIBMALI}
        fi
    fi
    if [ "${USE_X11}" = "yes" ]; then
        install -Dm 0644 ${S}/${PV}/${ARCH_PLATFORM_DIR}/x11/${MONOLITHIC_LIBMALI} ${D}${libdir}/x11/${MONOLITHIC_LIBMALI}
        if [ "${MALI_BACKEND_DEFAULT}" = "x11" ]; then
            ln -snf x11/${MONOLITHIC_LIBMALI} ${D}${libdir}/${MONOLITHIC_LIBMALI}
        fi
    else
        # We cant rely on the fact that all apps will use pkgconfig correctly
        sed -i -e 's/^#if defined(MESA_EGL_NO_X11_HEADERS)$/#if (1)/' ${D}${includedir}/EGL/eglplatform.h
    fi
    if [ "${USE_WL}" = "yes" ]; then
        install -m 0644 ${S}/${PV}/glesHeaders/GBM/gbm.h ${D}${includedir}/
        install -m 0644 ${WORKDIR}/gbm.pc ${D}${libdir}/pkgconfig/gbm.pc
        install -Dm 0644 ${S}/${PV}/${ARCH_PLATFORM_DIR}/wayland/${MONOLITHIC_LIBMALI} ${D}${libdir}/wayland/${MONOLITHIC_LIBMALI}
        if [ "${MALI_BACKEND_DEFAULT}" = "wayland" ]; then
            ln -snf wayland/${MONOLITHIC_LIBMALI} ${D}${libdir}/${MONOLITHIC_LIBMALI}
        fi
    fi
}

# We need separate packages to provide multiple alternatives, at this point we install
# everything on the default one but that can be split if necessary
PACKAGES += "${@bb.utils.contains("DISTRO_FEATURES", "x11", "${PN}-x11", "", d)}"
PACKAGES += "${@bb.utils.contains("DISTRO_FEATURES", "fbdev", "${PN}-fbdev", "", d)}"
PACKAGES += "${@bb.utils.contains("DISTRO_FEATURES", "wayland", "${PN}-wayland", "", d)}"
PACKAGES += "${PN}-headless"

# This is default/common for all alternatives
ALTERNATIVE_LINK_NAME[libmali-xlnx] = "${libdir}/${MONOLITHIC_LIBMALI}"

# Declare alternatives and corresponding library location
ALTERNATIVE:${PN}-x11 = "libmali-xlnx"
ALTERNATIVE_TARGET_libmali-xlnx-x11[libmali-xlnx] = "${libdir}/x11/${MONOLITHIC_LIBMALI}"

ALTERNATIVE:${PN}-fbdev = "libmali-xlnx"
ALTERNATIVE_TARGET_libmali-xlnx-fbdev[libmali-xlnx] = "${libdir}/fbdev/${MONOLITHIC_LIBMALI}"

ALTERNATIVE:${PN}-wayland = "libmali-xlnx"
ALTERNATIVE_TARGET_libmali-xlnx-wayland[libmali-xlnx] = "${libdir}/wayland/${MONOLITHIC_LIBMALI}"

ALTERNATIVE:${PN}-headless = "libmali-xlnx"
ALTERNATIVE_TARGET_libmali-xlnx-headless[libmali-xlnx] = "${libdir}/headless/${MONOLITHIC_LIBMALI}"

# Set priorities according to what we prveiously defined
ALTERNATIVE_PRIORITY_libmali-xlnx-x11[libmali-xlnx] = "${@bb.utils.contains("MALI_BACKEND_DEFAULT", "x11", "20", "10", d)}"
ALTERNATIVE_PRIORITY_libmali-xlnx-fbdev[libmali-xlnx] = "${@bb.utils.contains("MALI_BACKEND_DEFAULT", "fbdev", "20", "10", d)}"
ALTERNATIVE_PRIORITY_libmali-xlnx-wayland[libmali-xlnx] = "${@bb.utils.contains("MALI_BACKEND_DEFAULT", "wayland", "20", "10", d)}"

# If misconfigured, fallback to headless
ALTERNATIVE_PRIORITY_libmali-xlnx-headless[libmali-xlnx] = "${@bb.utils.contains("MALI_BACKEND_DEFAULT", "headless", "20", "15", d)}"

# Package gets renamed on the debian class, but we want to keep -xlnx
DEBIAN_NOAUTONAME:libmali-xlnx = "1"

# Update alternatives will actually have separate postinst scripts (one for each package)
# This wont work for us, so we create a common postinst script and we pass that as the general
# libmali-xlnx postinst script, but we defer execution to run on first boot (pkg_postinst_ontarget).
# This will avoid ldconfig removing the symbolic links when creating the root filesystem.
python populate_packages_updatealternatives:append () {
    # We need to remove the 'fake' libmali-xlnx before creating any links
    libdir = d.getVar('libdir')
    common_postinst = "#!/bin/sh\nrm " + libdir + "/${MONOLITHIC_LIBMALI}\n"
    for pkg in (d.getVar('PACKAGES') or "").split():
        # Not all packages provide an alternative (e.g. ${PN}-lic)
        postinst = d.getVar('pkg_postinst:%s' % pkg)
        if postinst:
            old_postinst = postinst
            new_postinst = postinst.replace('#!/bin/sh','')
            common_postinst += new_postinst
    d.setVar('pkg_postinst_ontarget:%s' % 'libmali-xlnx', common_postinst)
}

# Inhibit warnings about files being stripped
INHIBIT_PACKAGE_DEBUG_SPLIT = "1"
INHIBIT_PACKAGE_STRIP = "1"
INHIBIT_SYSROOT_STRIP = "1"

RREPLACES:${PN} = "libegl libgles1 libglesv1-cm1 libgles2 libglesv2-2 libgbm"
RPROVIDES:${PN} = "libegl libgles1 libglesv1-cm1 libgles2 libglesv2-2 libgbm"
RCONFLICTS:${PN} = "libegl libgles1 libglesv1-cm1 libgles2 libglesv2-2 libgbm"

# These libraries shouldn't get installed in world builds unless something
# explicitly depends upon them.
EXCLUDE_FROM_WORLD = "1"
FILES:${PN} += "${libdir}/*"

do_package:append() {

    shlibswork_dir = d.getVar('SHLIBSWORKDIR')
    pkg_filename = d.getVar('PN') + ".list"
    shlibs_file = os.path.join(shlibswork_dir, pkg_filename)
    lines = ""
    with open(shlibs_file, "r") as f:
        lines = f.readlines()
    with open(shlibs_file, "w") as f:
        for line in lines:
            if d.getVar('MALI_BACKEND_DEFAULT') in line.strip("\n"):
                 f.write(line)
}

repository: Xilinx/meta-xilinx

repo_name: Xilinx/meta-xilinx

gha_license_id: MIT

gha_language: C


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  font-family: 'Lato';
  src: url("//assets.teachablecdn.com/fonts/lato/Lato-Black.eot");
  src: url("//assets.teachablecdn.com/fonts/lato/Lato-Black.eot?#iefix") format("embedded-opentype"), url("//assets.teachablecdn.com/fonts/lato/Lato-Black.woff") format("woff"), url("//assets.teachablecdn.com/fonts/lato/Lato-Black.ttf") format("truetype");
  font-weight: 900;
  font-style: normal;
  font-display: swap; }

body{
  background-color: white;
  font-size: 15px;
  color: #596161 !important;
}

*{
  font-family: 'Lato';
}

nav.navbar.navbar-expand-lg.navbar-light.bg-light{
  background-color: white !important;
  padding-left: 10%;
  padding-right: 10%;
}

repository: annisadevin/annisadevin-latihid

repo_name: annisadevin/annisadevin-latihid

gha_license_id: Unlicense

gha_language: HTML

extension: css


text:
ol {
    display: block;
    list-style-type: decimal;
    -webkit-margin-before: 1em;
    -webkit-margin-after: 1em;
    -webkit-margin-start: 0px;
    -webkit-margin-end: 0px;
    -webkit-padding-start: 40px;
}

a, button, h1, h2, h3, h4, h5, h6, input, ol, p, textarea, th, ul {
    margin: 0;
    padding: 0;
    border: 0;
    outline: 0;
    font: inherit;
    vertical-align: baseline;
    background: transparent;
    list-style: none;
    overflow: visible;
}
.widget-floorpicker-slider {
    position: relative;
    transition: all 120ms ease-in-out;
}
.app-vertical-widget-holder {
    position: absolute;
    width: 29px;
    bottom: 33px;
    z-index: 4;
    right: 20px;
    -webkit-transform: translateZ(0);
    transform: translateZ(0);
}
.app-vertical-item {
    margin-top: 3px;
    -webkit-transform: translateZ(0);
    transform: translateZ(0);
}
.widget-floorpicker {
    background: white;
    box-shadow: 0px 1px 4px rgba(0,0,0,0.3);
    text-align: center;
    position: relative;
    width: 29px;
    overflow: hidden;
}
.widget-floorpicker-button {
    z-index: 2;
    color: #666;
    font-size: 14px;
    background-color: rgba(255,255,255,1);
    width: 29px;
    height: 29px;
    cursor: pointer;
    box-sizing: border-box;
    transition: background-color 0.16 ease-out;
}
.widget-floorpicker-button-selected {
    color: red !important;
    background: #f1f1f1;
    font-weight: bold;
}
.widget-floorpicker-button-alternate {
	background-color: #74787B;
	color: white;
}

repository: udayRag/udayIndoor

repo_name: udayRag/udayIndoor


text:
@inproceedings{mitnik_too_2004,
 address = {San Diego},
 author = {Mitnik, Pablo and Zeidenberg, Matthew},
 copyright = {All rights reserved},
 month = {January},
 title = {Too Many Bad Jobs: An Analysis of the Prospects for Career Ladder Initiatives in the Service Economy},
 year = {2004}
}

repository: mzeidenberg/academic-kickstart

repo_name: mzeidenberg/academic-kickstart


text:
DESCRIPTION = "nodeJS Evented I/O for V8 JavaScript"
HOMEPAGE = "http://nodejs.org"
LICENSE = "MIT & BSD-2-Clause & BSD-3-Clause & BSD-4-Clause & ISC & GPLv2 & GPLv3 & AFL-2.0 & GPL-2.0-with-OpenSSL-exception & Zlib"

LIC_FILES_CHKSUM = "file://LICENSE;md5=e804bf1e856481415099460bc54d9316"

DEPENDS = "openssl"
DEPENDS_class-target = "nodejs-native"

SRC_URI = "http://nodejs.org/dist/v${PV}/node-v${PV}.tar.gz"

SRC_URI[md5sum] = "2c00d8cf243753996eecdc4f6e2a2d11"
SRC_URI[sha256sum] = "0043656bb1724cb09dbdc960a2fd6ee37d3badb2f9c75562b2d11235daa40a03"

S = "${WORKDIR}/node-v${PV}"

# v8 errors out if you have set CCACHE
CCACHE = ""

ARCHFLAGS_arm = "${@bb.utils.contains('TUNE_FEATURES', 'callconvention-hard', '--with-arm-float-abi=hard', '--with-arm-float-abi=softfp', d)}"
ARCHFLAGS ?= ""

# Node is way too cool to use proper autotools, so we install two wrappers to forcefully inject proper arch cflags to workaround gypi
do_configure () {
    export LD="${CXX}"

    ./configure --prefix=${prefix} --without-snapshot ${ARCHFLAGS}
}

do_compile () {
    export LD="${CXX}"
    make BUILDTYPE=Release
}

do_install () {
    oe_runmake install DESTDIR=${D}
}

do_install_append_class-target () {
    # install node-gyp node hedaers in /usr/include/node-gyp/
    cd ${D}/${libdir}/node_modules/npm/node_modules/node-gyp/
    export HOME=${D}/usr/include/node-gyp
    sed -i 's/\.node-gyp//' lib/node-gyp.js

    # configure http proxy if neccessary
    if [ -n "${http_proxy}" ]; then
        ${STAGING_BINDIR_NATIVE}/node bin/node-gyp.js --verbose --proxy=${http_proxy} install
    elif [ -n "${HTTP_PROXY}" ]; then
        ${STAGING_BINDIR_NATIVE}/node bin/node-gyp.js --verbose --proxy=${HTTP_PROXY} install
    else
        ${STAGING_BINDIR_NATIVE}/node bin/node-gyp.js --verbose install
    fi
}

RDEPENDS_${PN} = "curl python-shell python-datetime python-subprocess python-crypt python-textutils python-netclient "
RDEPENDS_${PN}_class-native = ""

FILES_${PN} += "${libdir}/node/wafadmin ${libdir}/node_modules ${libdir}/dtrace"
BBCLASSEXTEND = "native"

repository: MCherifiOSS/meta-intel-iot-middleware

repo_name: MCherifiOSS/meta-intel-iot-middleware


text:
.header{
	color: #666;
	background: #fff;
	font-size: .28rem;
	height: 1rem;
	line-height: 1rem;
	position: fixed;
	top: 0;
	left: 0;
	right: 0;
	z-index: 100;
}
.left{
	font-size: .28rem;
	height: .3rem;
	line-height: .3rem;
	margin: .17rem 0 0 .18rem;
}
.btn{
	float: right;
	height: .24rem;
	line-height: .24rem;
	margin: .28rem .24rem 0 0;
}
.slider-box{
	padding-bottom: 20px;
}
.slider{
	margin-top: 1rem;
}
.slider-img{
	width: 100%;
}
.list-box{
	display:flex;
	flex-direction: row;
	flex-wrap: wrap;
	justify-content: space-around;
}
.list{
	background: #fff;
	padding-top: .4rem;
	padding-bottom: .2rem;
	text-align: center;
}
.list img {
	display: inline-block;
    width: .9rem;
    height: .9rem;
}
.list h4{
	font-size: .26rem;
    margin-top: .12rem;
    color: #666;
}
.novice-title h4{
	font-size: .28rem;
	text-align: center;
}
.content{
	display:flex;
	flex-direction: row;
	flex-wrap: wrap;
}
.item{
	width: 50%;
    box-sizing: border-box;
}
.item-first{
	padding: .32rem .2rem;
	text-align: center;
	border-right: 0.01rem solid #ddd;
}
.item-first img{
	width: 1.32rem;
	height: 1.32rem;
	margin: .2rem auto 0;
}
.item h4{
	font-size: .26rem;
	line-height: .4rem;
}
.item-right{
	width: 100%;
}
.item-right-first{
	border-bottom: 0.01rem solid #ddd;
}
.item-right li{
	height: 1.44rem;
	width: 100%;
	padding: .32rem .28rem;
}
.item-right li img{
	height: .8rem;
	width: .8rem;
	float: right;
}
.red{
	color: #f00;
}
.gray{
	color: #999;
	font-size: .24rem;
	margin-top: .06rem;
}

repository: valarMAO/react-JR

repo_name: valarMAO/react-JR

gha_language: JavaScript