text:
6}

@Article{RSM:Mar2014,
  author =       "N.F. Marrouche and D. Wilber and G. Hindricks and P. Jais
                 and N. Akoum and F. Marchlinski and E. Kholmovski and N.
                 Burgon and N. Hu and L. Mont and T. Deneke and M.
                 Duytschaever and T. Neumann and M. Mansour and C. Mahnkopf
                 and B. Herweg and E. Daoud and E. Wissner and P. Bansmann
                 and J. Brachmann",
  title =        "Association of atrial tissue fibrosis identified by
                 delayed enhancement MRI and atrial fibrillation catheter
                 ablation: the DECAAF study.",
  journal =      j-JAMA,
  year =         "2014",
  month =        "Feb",
  volume =       "311",
  number =       "5",
  pages =        "498--506",
  robnote =      "IMPORTANCE: Left atrial fibrosis is prominent in patients
                 with atrial fibrillation (AF). Extensive atrial tissue
                 fibrosis identified by delayed enhancement magnetic
                 resonance imaging (MRI) has been associated with poor
                 outcomes of AF catheter ablation. OBJECTIVE: To
                 characterize the feasibility of atrial tissue fibrosis
                 estimation by delayed enhancement MRI and its association
                 with subsequent AF ablation outcome. DESIGN, SETTING, AND
                 PARTICIPANTS: Multicenter, prospective, observational
                 cohort study of patients diagnosed with paroxysmal and
                 persistent AF (undergoing their first catheter ablation)
                 conducted between August 2010 and August 2011 at 15
                 centers in the United States, Europe, and Australia.
                 Delayed enhancement MRI images were obtained up to 30 days
                 before ablation. MAIN OUTCOMES AND MEASURES: Fibrosis
                 quantification was performed at a core laboratory blinded
                 to the participating center, ablation approach, and
                 procedure outcome. Fibrosis blinded to the treating
                 physicians was categorized as stage 1 (<10\% of the atrial
                 wall), 2 (>/=10\%-<20\%), 3 (>/=20\%-<30\%), and 4
                 (>/=30\%). Patients were followed up for recurrent
                 arrhythmia per current guidelines using
                 electrocardiography or ambulatory monitor recording and
                 results were analyzed at a core laboratory. Cumulative
                 incidence of recurrence was estimated by stage at days 325
                 and 475 after a 90-day blanking period (standard time
                 allowed for arrhythmias related to ablation-induced
                 inflammation to subside) and the risk of recurrence was
                 estimated (adjusting for 10 demographic and clinical
                 covariates). RESULTS: Atrial tissue fibrosis estimation by
                 delayed enhancement MRI was successfully quantified in 272
                 of 329 enrolled patients (57 patients [17\%] were excluded
                 due to poor MRI quality). There were 260 patients who were
                 followed up after the blanking period (mean [SD] age of
                 59.1 [10.7] years, 31.5\% female, 64.6\% with paroxysmal
                 AF). For recurrent arrhythmia, the unadjusted overall
                 hazard ratio per 1\% increase in left atrial fibrosis was
                 1.06 (95\% CI, 1.03-1.08; P < .001). Estimated unadjusted
                 cumulative incidence of recurrent arrhythmia by day 325
                 for stage 1 fibrosis was 15.3\% (95\% CI, 7.6\%-29.6\%);
                 stage 2, 32.6\% (95\% CI, 24.3\%-42.9\%); stage 3, 45.9\%
                 (95\% CI, 35.5\%-57.5\%); and stage 4, 51.1\% (95\% CI,
                 32.8\%-72.2\%) and by day 475 was 15.3\% (95\% CI,
                 7.6\%-29.6\%), 35.8\% (95\% CI, 26.2\%-47.6\%), 45.9\%
                 (95\% CI, 35.6\%-57.5\%), and 69.4\% (95\% CI,
                 48.6\%-87.7\%), respectively. Similar results were
                 obtained after covariate adjustment. The addition of
                 fibrosis to a recurrence prediction model that includes
                 traditional clinical covariates resulted in an improved
                 predictive accuracy with the C statistic increasing from
                 0.65 to 0.69 (risk difference of 0.05; 95\% CI,
                 0.01-0.09). CONCLUSIONS AND RELEVANCE: Among patients with
                 AF undergoing catheter ablation, atrial tissue fibrosis
                 estimated by delayed enhancement MRI was independently
                 associated with likelihood of recurrent arrhythmia. The
                 clinical implications of this association warrant further
                 investigation.",
  bibdate =      "Sun May 25 06:14:09 2014",
}

@PhdThesis{RSM:Mar70,
  author =       "R.O. Martin",
  title =        "Inverse Electrocardiography: {E}picardial Potentials",
  school =       "Duke University",
  address =      "Durham, NC",
  year =         "1970",
  robnote =      "Martin's thesis on the inverse solution solved not for
                 epicardial potentials but for a sphere placed around
                 the heart., TorsoModel064",
}

@Article{RSM:Mar72,
  author =       "R.O. Martin and T.C. Pilkington",
  title =        "Unconstrained Inverse {ECG}: {E}picardial Potentials",
  journal =      j-BME,
  year =         "1972",
  volume =       "24",
  pages =        "1--11",
  robnote =      "Don't have this but it is the paper wich followed from
                 Martin's thesis on the inverse problem using boundary
                 element method but a statistically determined
                 regularization parameter., TorsoModel065",
}

@Article{RSM:Mar75,
  author =       "M.L. Marcus and R.E. Kerber and J. Ehrhardt and F.M.
                 Abboud",
  title =        "Three Dimensional Geometry of Acutely Ischemic
                 Myocardium",
  journal =      j-C,
  year =         "1975",
  volume =       "5",
  pages =        "254--263",
  robnote =      "read it !, Ischemia019",
}

@InCollection{RSM:Mar80,
  author =       "P.R. Maroko and J.E. Muller and R.A. Kloner and E.
                 Braunwald",
  title =        "The Use of Electrocardiographic Parameters to Assess
                 Alterations in Myocardial Ischemic Damage",
  booktitle =    "Current Controversies in Cardiovascular Disease",
  editor =       "Rapport",
  publisher =    "Saunders",
  address =      "",
  year =         "1980",
  pages =        "257--280",
  robnote =      "An opinion about the value of ST-Segment mapping in
                 evaluating ischemic damage. A nice review with lots of
                 opinion. Looks at mostly dog experiments and the use of
                 ST mapping with several protocols to look at the
                 effects of clinical interventions on the progress and
                 extent of ischemic damage. QRS mapping and patients
                 also looked at. thesis, Ischemia054",
}

@Article{RSM:Mar83,
  author =       "H. Maresch and G. Pfurtscheller and Schuy S.",
  title =        "Brain Function Monitoring: {A} New Method for
                 Simultaneous Recording and Processing of
                 {EEG}-Powerspectra and Brainstem Potentials ({BAEP})",
  journal =      "Biomedizinische Technik",
  year =         "1983",
  volume =       "2",
  pages =        "117--122",
  robnote =      "brain function monitoring, Graz, EEG compressed
                 spectra, brainstem evoked potentials, GenPhysiol009",
}

@Article{RSM:Mar86,
  author =       "M.L. Marcus and D.G. Harrison and C. Wesley and L.F.
                 Hiratzka",
  title =        "Assessing the Physiological Significance of Coronary
                 Obstruction in Man",
  journal =      j-CJC,
  year =         "1986",
  volume =       "Suppl",
  pages =        "195A-199A",
  robnote =      "Very interesting look at the connection between
                 observed stenosis in coronary circulation and the
                 resulting ischemia, or lack of it. This makes a lot of
                 the evaluation schemes look pretty weak. He looks at
                 the coronary dilation reserve as a measure of the
                 capacity of the artery to withstand ischemia and finds
                 a poor corellation with the cases of between 10\% and
                 90\% stenosis. Quantitative coronary angiography,
                 coronary reactive hyperemia. thesis, Ischemia069",
}

@Article{RSM:Mar86b,
  author =       "P. Marzullo and O. Parodi and C. Marcassa and D.
                 Neglia and A. Labbate",
  title =        "When the Electrogram Fails to Detect Site and Extent
                 of Myocardial Ischemia",
  journal =      j-CJC,
  year =         "1986",
  volume =       "Suppl",
  pages =        "85A-90A",
  robnote =      "This paper reviews the limits of the 12-lead ECG in
                 localizing site and extent of myocardial ischemia in
                 man. It talks about radioisotope methods: Thallium 201
                 and ECG-gated microsphere scitography, and how there
                 results indicate coronary perfusion levels. The results
                 didn't lok good for the 12-lead ECG since only with
                 Angina at rest were there good corellations found. Good
                 references and suggestions that surface mapping will
                 provide more and better data., Ischemia066",
}

@Article{RSM:Mar87,
  author =       "H. Marks",
  title =        "Communications Software: Picks and Pans",
  journal =      "PC-Magazine",
  year =         "1987",
  month =        may,
  pages =        "234--235",
  robnote =      "A brief description of communications software for the
                 PC-clones, Crosstalk, Procomm, and others, software
                 review., CompSoft058",
}

@Article{RSM:Mar88,
  author =       "T. Marshall and J. Potter",
  title =        "How the Macintosh {II} NuBus Works",
  journal =      j-BYTE,
  year =         "1988",
  month =        dec,
  pages =        "Mac39--Mac51",
  robnote =      "A brief but fairly technical description of how the
                 NuBus actually runs. A good place to start if you need
                 to know this sort of stuff. Contains some good tips and
                 indicates that the manuals don't tell the whole story.,
                 CompHard134",
}

@Article{RSM:Mar89,
  author =       "T. Marshall and J.M. Tazelaar",
  title =        "Worth the {RISC}",
  journal =      j-BYTE,
  year =         "1989",
  month =        feb,
  pages =        "245--249",
  robnote =      "RISC technology, how it works and where it is being
                 used in today's chips and computers., CompHard180",
}

@InProceedings{RSM:Mar95,
  author =       "S. Martel and S. Lafontaine and D. Bullivant and I.W.
                 Hunter and P.J. Hunter",
  title =        "A Hardware Object-Oriented Cardiac Mapping System",
  pages =        "1647",
  booktitle =    c-EMBS95,
  year =         "1995",
  robnote =      "The Auckland mapping system",
}

@Article{RSM:Mas66,
  author =       "R.E. Mason and I. Likar",
  title =        "A New System of Multiple-lead Exercise
                 Electrocardiography",
  journal =      j-AHJ,
  year =         "1966",
  volume =       "71",
  month =        feb,
  pages =        "196--205",
  robnote =      "A classic reference to a lead system which we are
                 using in the Cath' lab. It allows us to move the leads
                 to the shoulders and hips instead of the limbs and
                 still record essentially the same signals. thesis,
                 ECG071 Peter MacFarlane says one should not use this
                 system as fully equivalent for standard (non exercise)
                 ECG",
}

@Article{RSM:Mas91,
  author =       "S. Masse and E. Sevaptsidis and I.D. Parson and E.
                 Downar",
  title =        "A three-dimensional display for cardiac activation
                 mapping.",
  journal =      j-P,
  year =         "1991",
  volume =       "14",
  number =       "4 Pt 1",
  pages =        "538--45",
  robnote =      "no hard copy, graphics, visualization",
}

@Article{RSM:Mas93,
  author =       "B.M. Massie and D.T. Mangano",
  title =        "Risk stratification for noncardiac surgery: {H}ow (and
                 Why?)",
  journal =      j-C,
  year =         "1993",
  volume =       "87",
  pages =        "1752--1755",
  robnote =      "A Mangano paper that summarizes the situation very
                 well; describes the problem, how to carry out
                 periopertive risk and what can be done once ricks has
                 been determined. anesthesiology, perioperative
                 ischemia",
}

@Article{RSM:Masg2005,
  author =       "L. Massacesi and A. Parigi and A. Barilaro and A. M.
                 Repice and G. Pellicano and A. Konze and G. Siracusa
                 and R. Taiuti and L. Amaducci",
  title =        "Efficacy of azathioprine on multiple sclerosis new
                 brain lesions evaluated using magnetic resonance
                 imaging",
  journal =      "Arch Neurol",
  volume =       "62",
  number =       "12",
  pages =        "1843--7",
  year =         "2005",
}

@InCollection{RSM:Mat67,
  author =       "K. Matsuda and A. Kamiyama and T. Hoshi",
  title =        "Configuration of the Purkinje-ventricular Fiber
                 Junction and its Analysis",
  booktitle =    "Electrophysiology and Ultrastructure of the Heart",
  editor =       "T. Sano and I. Mizuhira and K. Matsuda",
  publisher =    "Grune \& Stratton",
  address =      "New York",
  year =         "1967",
  robnote =      "Ref 22 in Holland and Brooks, PTCA9, the Purkinje
                 fibre insertion sites are more easily knocked out than
                 the Purkinje fibres themselves or the muscle which
                 surrounds them. don't have this one. thesis,
                 Ischemia147",
}

@Article{RSM:Mat84,
  author =       "H. Matsuda and A. Noma",
  title =        "Isolation of Calcium Current and its Sensitivity to
                 Monovalent Cations in Dialysed Ventricular Cells of
                 Guinea-Pig",
  journal =      j-JP,
  year =         "1084",
  volume =       "357",
  pages =        "553--573",
  robnote =      "patch clamp recordings on calcium channels in guinea
                 pig heart cells., CellPhysiol080",
}

@Article{RSM:Mat86,
  author =       "E.K. Matthews",
  title =        "Calcium and Membrane Permeability",
  journal =      "Brit. Med. Bull.",
  year =         "1986",
  volume =       "42",
  pages =        "391--397",
  robnote =      "Read it!, CellPhysiol054",
}

@Article{RSM:Mat87,
  author =       "H. Matsuda and A. Saigusa and H. Irisawa",
  title =        "Ohmic Conductance Through the Inwardly Rectifying {K}
                 Channel and Blocking by Internal $Mg^{2+}$",
  journal =      "Nature",
  year =         "1987",
  volume =       "325",
  month =        jan,
  pages =        "156--159",
  robnote =      "Looking at a K-channel using improved patch clamp
                 technique (single channel and whole cell clamp,
                 techniques for getting rid of Na and Ca currents, as
                 well as separating ionic from displacement currents
                 (low temp) are also described., CellPhysiol078",
}

@Article{RSM:May97,
  author =       "E.M. Maynard and C.T. Nordhausen and R.A. Normann",
  title =        "The Utah intracortical Electrode Array: a recording
                 structure for potential brain-computer interfaces.",
  journal =      "Electroencephalogr Clin Neurophysiol",
  year =         "1997",
  month =        "Mar",
  volume =       "102",
  number =       "3",
  pages =        "228--239",
  robnote =      "We investigated the potential of the Utah Intracortical
                 Electrode Array (UIEA) to provide signals for a
                 brain-computer interface (BCI). The UIEA records from
                 small populations of neurons which have an average
                 signal-to-noise ratio (SNR) of 6:1. We provide specific
                 examples that show the activities of these populations of
                 neurons contain sufficient information to perform control
                 tasks. ",
  bibdate =      "Tue Feb 25 21:02:04 2014",
}

@Article{RSM:McA75,
  author =       "R.E. McAllister and D. Noble and R.W. Tsien",
  title =        "Reconstruction of the Electrical Activity of Cardiac
                 Purkinje Fibres",
  journal =      j-JP,
  year =         "1975",
  volume =       "251",
  pages =        "1--59",
  robnote =      "Another classic on cardiac cell modelling in Purkinje
                 fibres, the McAllister, Noble, Tsien model.,
                 CardCell002",
}

@Book{RSM:McA91,
  author =       "William D. McArdle and F.I. Katch and V.L. Katch",
  title =        "Exercise Physiology: Energy, Nutrition, and Human
                 Performance",
  publisher =    "Lea and Febiger",
  year =         "1991",
  robnote =      "A comprehensive book on exercise physiology. Would
                 make a good text for a course. cycling. Eccles: WE 103
                 M115e 1991.",
}

@Article{RSM:McC81,
  author =       "A.D. McClennan",
  title =        "Theoretical and Practical Considerations concearning
                 an Active Current Source for Intracellular Recording
                 and Stimulation",
  journal =      "Med. \& Biol. Eng. \& Comput.",
  year =         "1981",
  volume =       "1",
  pages =        "659--661",
  robnote =      "microelectrodes, electronics, Case Western University,
                 active current source, electronics, stimulation.,
                 CellPhysiol036",
}

@Article{RSM:McC89,
  author =       "A.D. McCulloch and B. H. Smaill and P. J. Hunter",
  title =        "Regional left ventricular epicardial deformation in
                 the passive dog heart",
  journal =      j-CR,
  year =         "1989",
  volume =       "64",
  pages =        "721--723",
  robnote =      "simulation of mechanical behavior of the heartcc",
  bibdate =      "Tue Dec 05 01:00:19 2000",
}

@Article{RSM:McC91,
  author =       "R.W. McCarley and S.F. Faux and M.E. Shenton and P.G.
                 Nestor and J. Adams",
  title =        "Event-related potentials in schizophrenia: Their
                 biological and clinical correlates and a new model of
                 schizophrenic pathophysiology",
  journal =      "Schizophr. Res.",
  year =         "1991",
  volume =       "4",
  pages =        "209--231",
  robnote =      "",
}

@Article{RSM:McC91b,
  author =       "A.D. McCulloch and J.H. Omens",
  title =        "Non-homogeneous analysis of three-dimensional
                 transmural finite deformation in canine ventricular
                 myocardium",
  journal =      "J. Biomech.",
  year =         "1991",
  volume =       "24",
  pages =        "539--548",
  robnote =      "simulation of mechanical aspects of the heart",
  bibdate =      "Tue Dec 05 01:06:50 2000",
}

@Article{RSM:McC93,
  author =       "R.W. McCarley and M.E. Shenton and B.F. O'Donnell and
                 S.F. Faux and R. Kikinis and P.G. Nestor and F.A.
                 Jolesz",
  title =        "Auditory {P300} abnormalities and left posterior
                 superior temporal gyrus volume reduction in
                 schizophrenia",
  journal =      "Arch. Gen. Psychiatry",
  year =         "1993",
  volume =       "87",
  pages =        "175--184",
  robnote =      "",
}

@Article{RSM:McC98,
  author =       "A.D. McCulloch and D. Sung and J.M. Wilson and R.S.
                 Pavelec and J.H. Omens",
  title =        "Flow-function relations during graded coronary
                 occlusions in the dog: effects of transmural location
                 and segment orientation.",
  journal =      "Cardiovasc Res",
  year =         "1998",
  month =        mar,
  volume =       "37",
  number =       "3",
  pages =        "636--645",
  robnote =      "OBJECTIVE: The aim of this study was to determine the
                 effects of fiber orientation and transmural location on
                 the relationships between regional myocardial flow and
                 three-dimensional systolic wall strain during graded
                 coronary artery occlusions. METHODS: Transmural
                 distributions of three-dimensional strain (by biplane
                 radiography of implanted radiopaque markers) and
                 myocardial blood flows (using fluorescent microspheres)
                 were measured in the ischemic region during graded left
                 anterior descending (LAD) coronary artery occlusions in
                 12 anesthetized dogs. RESULTS: Occlusion of the
                 coronary artery did not significantly alter mean heart
                 rate or end-systolic pressure. As flow decreased during
                 graded occlusions, ischemia significantly changed
                 systolic circumferential, longitudinal, radial, fiber
                 and cross-fiber strains (p < 0.004). There was a
                 significant effect of transmural position on
                 circumferential, cross-fiber and radial strains, but
                 not on fiber or longitudinal strains. Ischemia
                 significantly altered all normal strains:
                 circumferential, longitudinal, fiber, cross-fiber and
                 radial. There was a strong interaction effect between
                 transmural location and blood flow for circumferential,
                 cross-fiber and radial strains, but not fiber or
                 longitudinal strains. CONCLUSION: During non-transmural
                 ischemia, there is evidence of strong transmural
                 tethering in the cross-fiber direction, whereas the
                 fiber-strain flow relation is independent of transmural
                 position. Thus, whether the relationship between local
                 myocardial bloodflow and systolic strain during acute
                 ischemia is dependent on transmural location, depends
                 on segment orientation.",
  bibdate =      "Tue Dec 19 13:33:03 2000",
}

@Article{RSM:McD2003,
  author =       "N. McDannold and M. Moss and R. Killiany and D. L.
                 Rosene and R. L. King and F. A. Jolesz and K. Hynynen",
  title =        "{MRI}-guided focused ultrasound surgery in the brain:
                 tests in a primate model",
  journal =      "Magn Reson Med",
  volume =       "49",
  number =       "6",
  pages =        "1188--1191",
  abstract =     "MRI-guided focused ultrasound was tested in the brains
                 of rhesus monkeys. Locations up to 4.8 cm deep were
                 targeted. Focal heating was observed in all cases with
                 MRI-derived temperature imaging. Subthreshold heating
                 was observed at the focus when the ultrasound beam was
                 targeted with low power sonications, and in the
                 ultrasound beam path during high-power exposures.
                 Lethal temperature values and histologically confirmed
                 tissue damage were confined to the focal zone (e.g.,
                 not in the ultrasound beam path), except when the focus
                 was close to the bone. In that case, damage to the
                 neighboring brain tissue was observed. Focal lesions
                 were observed on histological examination and, in some
                 cases, in MR images acquired immediately after the
                 ultrasound exposures. The capabilities demonstrated in
                 this study will be of benefit for clinical ultrasound
                 therapies in the brain.",
  keywords =     "Animals Brain/pathology/*surgery Macaca mulatta
                 *Magnetic Resonance Imaging Neurosurgical
                 Procedures/methods Temperature *Ultrasonic Therapy",
  year =         "2003",
}

@Article{RSM:McD2011,
  author =       "K.S. McDowell and H.J. Arevalo and M.M. Maleckar and N.A.
                 Trayanova",
  title =        "Susceptibility to arrhythmia in the infarcted heart
                 depends on myofibroblast density.",
  journal =      "Biophys J",
  year =         "2011",
  month =        "Sep",
  volume =       "101",
  number =       "6",
  pages =        "1307--1315",
  robnote =      "Fibroblasts are electrophysiologically quiescent in the
                 healthy heart. Evidence suggests that remodeling following
                 myocardial infarction may include coupling of
                 myofibroblasts (Mfbs) among themselves and with myocytes
                 via gap junctions. We use a magnetic resonance
                 imaging-based, three-dimensional computational model of
                 the chronically infarcted rabbit ventricles to
                 characterize the arrhythmogenic substrate resulting from
                 Mfb infiltration as a function of Mfb density. Mfbs
                 forming gap junctions were incorporated into both infarct
                 regions, the periinfarct zone (PZ) and the scar; six
                 scenarios were modeled: 0\%, 10\%, and 30\% Mfbs in the
                 PZ, with either 80\% or 0\% Mfbs in the scar. Ionic
                 current remodeling in PZ was also included. All
                 preparations exhibited elevated resting membrane potential
                 within and near the PZ and action potential duration
                 shortening throughout the ventricles. The unique
                 combination of PZ ionic current remodeling and different
                 degrees of Mfb infiltration in the infarcted ventricles
                 determines susceptibility to arrhythmia. At low densities,
                 Mfbs do not alter arrhythmia propensity; the latter arises
                 predominantly from ionic current remodeling in PZ. At
                 intermediate densities, Mfbs cause additional action
                 potential shortening and exacerbate arrhythmia propensity.
                 At high densities, Mfbs protect against arrhythmia by
                 causing resting depolarization and blocking propagation,
                 thus overcoming the arrhythmogenic effects of PZ ionic
                 current remodeling.",
  bibdate =      "Tue Mar 3 08:29:44 2015",
  pmcid =        "PMC3177053",
}

@Article{RSM:McD2012,
  author = 	 "K.S. McDowell and F. Vadakkumpadan and R. Blake and
                  J. Blauer, G. Planck and R.S. MacLeod and N.A. Trayanova",
  title = 	 "Methodology for patient-specific modeling of atrial fibrosis
                  as a substrate for atrial fibrillation",
  journal = 	 j-JE,
  year = 	 2012,
  volume = 	 45,
  number = 	 6,
  pages = 	 "640--645",
  Robnote = 	 "First paper with Natalia on AF modeling",
  pmcid =        "PMC3515859",
}

@InProceedings{RSM:McD2012a,
  author = 	 "K.S. McDowell and F. Vadakkumpadan and J. Blauer,
                  G. Planck and R.S. MacLeod and N.A. Trayanova",
  title = 	 "Fibrosis underlies the Breakup of Pulmonary Vein Ectopic
                  Waves in Patient-Specific Atrial Models",
  booktitle = c-HRS,
  year = 	 2012,
  Robnote = 	 "Second presentation of collaboration with NAT on atrial
                  fibrillation"
}

@Article{RSM:McD2013,
  author =       "K.S. McDowell and F. Vadakkumpadan and R. Blake and J.
                 Blauer and G. Plank and R.S. Macleod and N.A. Trayanova",
  title =        "Mechanistic inquiry into the role of tissue remodeling in
                 fibrotic lesions in human atrial fibrillation.",
  journal =      j-BJ,
  year =         "2013",
  month =        "Jun",
  volume =       "104",
  number =       "12",
  pages =        "2764--2773",
  robnote =      "This study used an array of 3D
                 computational models with different representations of
                 fibrosis based on a patient-specific atrial geometry with
                 accurate fibrotic distribution to determine the mechanisms
                 by which fibrosis underlies the degradation of a pulmonary
                 vein ectopic beat into AF. Fibrotic lesions in models were
                 represented with combinations of: gap junction remodeling;
                 collagen deposition; and myofibroblast proliferation with
                 electrotonic or paracrine effects on neighboring myocytes.
                 The study found that the occurrence of gap junction
                 remodeling and the subsequent conduction slowing in the
                 fibrotic lesions was a necessary but not sufficient
                 condition for AF development, whereas myofibroblast
                 proliferation and the subsequent electrophysiological
                 effect on neighboring myocytes within the fibrotic lesions
                 was the sufficient condition necessary for reentry
                 formation. Collagen did not alter the arrhythmogenic
                 outcome resulting from the other fibrosis components.
                 Reentrant circuits formed throughout the noncontiguous
                 fibrotic lesions, without anchoring to a specific fibrotic
                 lesion.",
  bibdate =      "Tue Jul 16 10:25:48 2013",
  pmcid =        "PMC3686346",
}

@Article{RSM:McD2015,
  author =       "K.S. McDowell and S. Zahid and F. Vadakkumpadan and J.
                 Blauer and R.S. MacLeod and N.A. Trayanova",
  title =        "Virtual electrophysiological study of atrial fibrillation
                 in fibrotic remodeling.",
  journal =      "PLoS One",
  year =         "2015",
  volume =       "10",
  number =       "2",
  pages =        "e0117110",
  robnote =      "We aimed to
                 provide a proof-of-concept that patient-specific virtual
                 electrophysiological study that combines i) atrial
                 structure and fibrosis distribution from clinical MRI and
                 ii) modeling of atrial electrophysiology, could be used to
                 predict: (1) how fibrosis distribution determines the
                 locations from which paced beats degrade into AF; (2) the
                 dynamic behavior of persistent AF rotors; and (3) the
                 optimal ablation targets in each patient. Four MRI-based
                 patient-specific models of fibrotic left atria were
                 generated, ranging in fibrosis amount. Virtual
                 electrophysiological studies were performed in these
                 models, and where AF was inducible, the dynamics of AF
                 were used to determine the ablation locations that render
                 AF non-inducible. In 2 of the 4 models patient-specific
                 models AF was induced; in these models the distance
                 between a given pacing location and the closest fibrotic
                 region determined whether AF was inducible from that
                 particular location, with only the mid-range distances
                 resulting in arrhythmia. Phase singularities of persistent
                 rotors were found to move within restricted regions of
                 tissue, which were independent of the pacing location from
                 which AF was induced. Electrophysiological sensitivity
                 analysis demonstrated that these regions changed little
                 with variations in electrophysiological parameters.
                 Patient-specific distribution of fibrosis was thus found
                 to be a critical component of AF initiation and
                 maintenance. When the restricted regions encompassing the
                 meander of the persistent phase singularities were modeled
                 as ablation lesions, AF could no longer be induced. The
                 study demonstrates that a patient-specific modeling
                 approach to identify non-invasively AF ablation targets
                 prior to the clinical procedure is feasible.",
  bibdate =      "Fri Jun 19 06:43:08 2015",
  pmcid =        "PMC4333565",
}

@InCollection{RSM:McD84,
  author =       "T.F. McDonald",
  title =        "Excitation - Contraction Coupling",
  booktitle =    "Physiology and Pathophysiology of the Heart",
  editor =       "N. Speralakis",
  publisher =    "Martinus Nijhoff Pub.",
  address =      "Boston, The Hgue, Dordrecht, Lancaster",
  year =         "1984",
  pages =        "187--1984",
  robnote =      "A good review of E-C coupling, I used it for a
                 seminar, Ca, calcium, voltage clamp, CellPhysiol040",
}

@Article{RSM:McD89,
  author =       "T.F. McDonald and D. Pelzer and W. Trautwein",
  title =        "Dual Action (Stimulation, Inhibition) of {D600} on
                 Contractility and Calcium Channels in Guinea-Pig and
                 Cat Heart Cells",
  journal =      j-JP,
  year =         "1989",
  volume =       "414",
  pages =        "569--586",
  robnote =      "A paper on D-600 and single channel recordings of its
                 action., CardCell043",
}

@Article{RSM:McF53,
  author =       "R. McFee and F.D. Johnston",
  title =        "Electrocardiographic Leads. {I. I}ntroduction.",
  journal =      j-C,
  year =         "1953",
  volume =       "8",
  pages =        "554--68",
  robnote =      "A reference from an article of Milan's on lead theory.
                 Don't have a copy but use it in thesis., ECG094",
}

@Article{RSM:McF54,
  author =       "R. McFee and F.D. Johnston",
  title =        "Electrocardiographic Leads. {II. A}nalysis.",
  journal =      j-C,
  year =         "1954",
  volume =       "9",
  pages =        "255--66",
  robnote =      "A reference from an article of Milan's on lead theory.
                 Don't have a copy but use it in thesis., ECG095",
}

@Article{RSM:McF54b,
  author =       "R. McFee and F.D. Johnston",
  title =        "Electrocardiographic Leads. {III. S}ynthesis.",
  journal =      j-C,
  year =         "1954",
  volume =       "9",
  pages =        "868--80",
  robnote =      "A reference from an article of Milan's on lead theory.
                 Don't have a copy but use it in thesis., ECG096",
}

@Article{RSM:McF68,
  author =       "R. McFee and S. Rush",
  title =        "Qualitative Effects of Thoracic Resistivity Variations
                 on the Interpretation of the Electrocardiograms: the
                 Low Resistance Surface Layer",
  journal =      j-AHJ,
  year =         "1968",
  volume =       "76",
  pages =        "48--61",
  robnote =      "A thesis reference which describes the method used by
                 Gulrajani and Stanley to include the effect of the
                 thoracic muscle layer in the torso inverse
                 calculations., inhomogeneous conductivity",
}

@InProceedings{RSM:McG2007,
  author =       "C. McGann and E. Kholmovski and R.S. Oakes and E.
                 DiBella and R.S. MacLeod and E. Fish and M. Daccarrett
                 and N. Segerson and N.F. Marrouche",
  title =        "Magnetic Resonance Imaging Detects Chronic Left Atrial
                 Wall Injury Post Ablation of Atrial Fibrillation",
  booktitle =    "American Heart Association",
  address =      "Orlando, FL",
  year =         "2007",
  robnotes =     "afib",
}

@Article{RSM:McG2008,
  author =       "C.J. McGann and E.G. Kholmovski and R.S. Oakes and
                 J.E. Blauer and M. Daccarett and N.M Segerson and K.J.
                 Airey and N. Akoum and E.N. Fish and T.J. Badger and
                 E.V.R. DiBella and D. Parker and R.S. MacLeod and N.F.
                 Marrouche",
  title =        "New magnetic resonance imaging based method to define
                 extent of left atrial wall injury after the ablation of
                 atrial fibrillation",
  journal =      j-ACC,
  year =         "2008",
  volume =       "52",
  number =	 "15",
  pages =	 "1263--1271",
  robnote =      "afib Chris paper on post ablation DEMRI",
  bibdate =      "Thu Sep 25 09:05:43 2008",
}

@Article{RSM:McG2011,
  author =       "C. McGann and E. Kholmovski and J. Blauer and S.
                 Vijayakumar and T. Haslam and J. Cates and E. Dibella and
                 N. Burgon and B. Wilson and A. Alexander and M. Prastawa
                 and M. Daccarett and G. Vergara and N. Akoum and D. Parker
                 and R. Macleod and N. Marrouche",
  title =        "Dark regions of no-reflow on late gadolinium enhancement
                 magnetic resonance imaging result in scar formation after
                 atrial fibrillation ablation.",
  journal =      j-ACC,
  year =         "2011",
  month =        "Jul",
  volume =       "58",
  number =       "2",
  pages =        "177--185",
  robnote =      "OBJECTIVES: The aim of this study was to assess acute
                  ablation injuries seen on late gadolinium enhancement (LGE)
                  magnetic resonance imaging (MRI) immediately post-ablation
                  (IPA) and the association with permanent scar 3 months
                  post-ablation (3moPA).  Heterogeneous injuries with HE and NE
                  regions were identified in all patients. Dark NE regions in
                  the left atrial wall on LGE MRI demonstrate findings similar
                  to the no-reflow phenomenon. Although the left atrial wall
                  showed similar amounts of HE, NE, and normal tissue IPA (37.7
                  +/- 13\%, 34.3 +/- 14\%, and 28.0 +/- 11\%, respectively; p =
                  NS), registration of IPA injuries with 3moPA scarring
                  demonstrated that 59.0 +/- 19\% of scar resulted from NE
                  tissue, 30.6 +/- 15\% from HE tissue, and 10.4 +/- 5\% from
                  tissue identified as normal.  Paired t-test comparisons were
                  all statistically significant among NE, HE, and normal tissue
                  types (p < 0.001). Arrhythmia recurrence at 1-year follow-up
                  correlated with the degree of wall enhancement 3moPA (p =
                  0.02). CONCLUSIONS: Radiofrequency ablation results in
                  heterogeneous injury on LGE MRI with both HE and NE wall
                  lesions. The NE lesions demonstrate no-reflow characteristics
                  and reveal a better predictor of final scar at 3 months. Scar
                  correlates with procedure outcomes, further highlighting the
                  importance of early scar prediction.",
  bibdate =      "Sat Jul 2 07:18:49 2011",
  pmcid =        "PMC3746015",
}

@Article{RSM:McG2014,
  author = 	 "C. McGann and N. Akoum and A. Patel and E. Kholmovski and
                  P. Revelo and K. Damal and B. Wilson and J. Cates and
                  A. Harrison and R. Ranjan and N. Burgon and T. Greene and
                  D. Kim and E. DiBella and D. Parker and R.S. MacLeod and
                  N.R. Marrouche",
  title = 	 "Atrial fibrillation ablation outcome is predicted by left
                  atrial remodeling on {MRI}",
  journal = 	 j-CAE,
  year = 	 2014,
  volume = 	 7,
  number = 	 1,
  pages = 	 "23--30",
  Robnote = 	 "We hypothesized that late gadolinium enhancement
                 magnetic resonance imaging (LGE-MRI) can identify left
                 atrial (LA) wall structural remodeling (SRM) and stratify
                 patients who are likely or not to benefit from ablation
                 therapy.
                 Histological validation of SRM was performed comparing
                 LGE-MRI to surgical biopsy. A total of 386 patients (91\%)
                 with adequate LGE-MRI scans were included in the study.
                 Post-ablation, 123 (31.9\%) experienced recurrent atrial
                 arrhythmias over one-year follow-up. Recurrent arrhythmias
                 (failed ablations) occurred at higher SRM stages with
                 28/133 (21.0\%) stage I, 40/140 (29.3\%) stage II, 24/71
                 (33.8\%) stage III, and 30/42 (71.4\%) stage IV. In
                 multi-variate analysis, ablation outcome was best
                 predicted by advanced SRM stage (hazard ratio (HR) 4.89;
                 p<0.0001) and diabetes (HR 1.64; p=0.036) while increased
                 LA volume and persistent AF were not significant
                 predictors. LA wall enhancement was significantly greater
                 in AF patients vs. non-AF controls (16.6+/-11.2\% vs.
                 3.1+/-1.9\%, p<0.0001). Histological evidence of
                 remodeling from surgical biopsy specimens correlated with
                 SRM on LGE-MRI.",
  pmcid =        "PMC4086672",
}

@Article{RSM:McG84,
  author =       "K.C. McGill and L.J. Dorfman",
  title =        "High-Resolution Alignment of Sampled Waveforms",
  journal =      j-BME,
  year =         "1984",
  volume =       "31",
  pages =        "462--468",
  robnote =      "A routine to test Nyquist criteria for sampling of
                 waveforms and how to re-expand tham back to higher
                 resolution for further processing and plotting.,
                 CompSoft008",
}

@Article{RSM:McG87,
  author =       "J.A.S. McGuigan and L.A. Blatter",
  title =        "Sodium/Calcium Exchange in Ventricular Muscle",
  journal =      "Experientia",
  year =         "1987",
  volume =       "43",
  pages =        "1140--1145",
  robnote =      "sodium/calcium exchange, yet again., CardCell011",
}

@Article{RSM:McG90,
  author =       "J. McGuigan and J. Beard",
  title =        "Sound probe peers inside the arteries",
  journal =      "New Scientist",
  year =         "1990",
  month =        jun,
  pages =        "35",
  robnote =      "PTCA, angiography",
}

@Article{RSM:McL74,
  author =       "V.W. McLaughlin and N.C. Flowers and L.G. Horan and
                 H.A.W. Killam",
  title =        "Surface Potential Contributions From Discrete Elements
                 of Ventricular Wall",
  journal =      j-AJC,
  year =         "1974",
  volume =       "34",
  pages =        "302--308",
  robnote =      "A closed chest, postmortem documented prospective
                 study, using difference maps from the body surface of
                 ischemic dogs. The idea was measure BSPM, create an
                 infarct, let the animal recover for a week and then
                 repeat the measurement. The differnce map would then
                 represent those regions of the heart which are
                 inactive. Mention made of the problem that delayed
                 activation can present in interpreting the results,
                 i.e. you cannot tell the difference between ischemic
                 changes and those due to delay, in terms of
                 localization and quantization of the ischemia. thesis,
                 Ischemia118",
}

@Book{RSM:McM77,
  author =       "R.M.H. McMinn and R.T. Hutchings",
  title =        "A Colour Atlas of the Human Anatomy",
  editor =       "",
  publisher =    "Wolfe Medical Publications",
  address =      "London",
  year =         "1977",
  robnote =      "An anatomy atlas I used to produce the coronary
                 circulation on the epicardial displays. thesis,
                 Cardiol018",
}

@Article{RSM:McM79,
  author =       "M.M. McMahon and B.G. Brown and R. Cukinghan and E.L.
                 Rolett and E. Bolson and M. Frimer and H.T. Dodge",
  title =        "Quantitative Coronary Angiography: Measurement of the
                 ``Critical'' Stenosis in Patients with Unstable Angina
                 and Single-Vessel Disease without Collaterals",
  journal =      j-C,
  year =         "1979",
  volume =       "6",
  pages =        "106--113",
  robnote =      "A system of computer-based coronary arteriography,
                 coronary occlusion, stenosis. The system is sort of
                 semi-manual but appears to work., Ischemia008",
}

@Article{RSM:McM94,
  author =       "S.R. McMechan and C.M. Cullen and G. MacKenzie and
                 G.J. Dempsey and G.T. Wright and M. Crawley and J.
                 Anderson and A.A. Adgey",
  title =        "Discriminant function analysis of body surface
                 potential maps in acute myocardial infarction",
  journal =      j-JE,
  year =         "1994",
  volume =       "27",
  number =       "Suppl",
  pages =        "117--120",
  robnote =      "Ulster group using statistical approach to detecting
                 MI in patients from BSMP data",
  bibdate =      "Fri Jun 19 11:37:41 1998",
}

@Article{RSM:McM95,
  author =       "S.R. McMechan and G. MacKenzie and J. Allen and G.T.
                 Wright and G.J. Dempsey and M. Crawleyand J. Anderson
                 and A.A. Adgey",
  title =        "Body surface {ECG} potential maps in acute myocardial
                 infarction",
  journal =      j-JE,
  year =         "1995",
  volume =       "28",
  number =       "Suppl",
  pages =        "184--190",
  robnote =      "The Belfast group and their MI mapping. BSPM",
  bibdate =      "Wed Jun 10 14:14:44 1998",
}

@Article{RSM:McN85,
  author =       "M.T. McNamara and C.B. Higgins and N. Schechtmann and
                 E. Botvinick and M.J. Lipton and K. Chatterjee and E.G.
                 Amparo",
  title =        "Detection and Characteristics of Acute Myocardial
                 infarction in Man with use of Gated Magnetic
                 Resonance",
  journal =      j-C,
  year =         "1985",
  volume =       "4",
  pages =        "717--724",
  robnote =      "A look at ischemia with MRI. Read it!, Ischemia107",
}

@Article{RSM:McP85,
  author =       "D.D. McPherson and B.M. Horacek and D.J.
                 Sutherland and C.S. Armstrong and A. Spencer and T.J.
                 Montague",
  title =        "Exercise Electrocardiographic Mapping in Normal
                 Subjects",
  journal =      j-JE,
  year =         "1985",
  volume =       "18",
  pages =        "351--360",
  robnote =      "Another one of TJ's clinical papers on normal subjects
                 this time, sort of a benchmark study of just what a
                 normal bspm map looks like. thesis, BSPM053",
}

@Article{RSM:McP85b,
  author =       "D.D. McPherson and B.M. Horacek and C.A.
                 Spencer and D.E. Johnstone and L.D. Lalonde and C.L.
                 Cousins and T.J. Montague",
  title =        "Indirect Measurements of Infarct Size, Correlative
                 Variability of Enzyme, Radionuclear, Angiographic and
                 Body Surface Map Variables in 34 Patients during acute
                 Phase of First Myocardial Infarction",
  journal =      "Chest",
  year =         "1985",
  volume =       "8",
  pages =        "841--848",
  robnote =      "thesis, Ischemia003",
}

@Article{RSM:McP86,
  author =       "D.D. McPherson and B.M. Horacek and D.E.
                 Johnstone and L.D. Lalonde and C.A. Spencer and T.J.
                 Montague",
  title =        "{Q}-Wave Infarction: {P}athophysiology of Body Surface
                 Potential map and Ventriculographic Patterns in
                 Anterior and Inferior Groups",
  journal =      j-CJC,
  year =         "1986",
  volume =       "Suppl A",
  pages =        "91A-98A",
  robnote =      "Thesis, BSPM009",
}

@article{RSM:McR82,
  author  = "G.J. McRae and J.W. Tilden and and J.H. Seinfeld",
  title   = "Global sensitivity analysis - a computational implementation
             of the {F}ourier amplitude sensitivity test ({FAST})",
  journal = "Computers and Chemical Engineering",
  volume  = "6",
  issue   = "1",
  pages   = "15--25",
  year    = "1982"
}

@Article{RSM:McV2002,
  author =       "E. McVeigh and O. Faris and D. Ennis and P. Helm and
                 F. Evans",
  title =        "Electromechanical mapping with {MRI} tagging and
                 epicardial sock electrodes.",
  journal =      j-JE,
  year =         "2002",
  volume =       "35 Suppl",
  pages =        "61--64",
  robnote =      "Coupled with the near simultaneous mapping of
                 electrical depolarization with a sock electrode array,
                 we can investigate the relationship between electical
                 activity and mechanical function on a local level in
                 the in vivo heart. Registered fiber angle maps can be
                 also be obtained in the same heart with diffusion
                 magnetic resonance imaging to assist in the
                 construction of the electromechanical model of the
                 whole heart.",
  bibdate =      "Mon Jun 2 08:11:54 2003",
}

@Article{RSM:McV2005,
  author =       "E.R. McVeigh and M.A. Guttman and P. Kellman and A.N.
                 Raval and R.J. Lederman",
  title =        "Real-time, Interactive MRI for cardiovascular
                 interventions.",
  journal =      "Acad Radiol",
  year =         "2005",
  month =        "Sep",
  volume =       "12",
  number =       "9",
  pages =        "1121--1127",
  robnote =       "",
  bibdate =      "Wed Aug 26 13:21:34 2009",
}

@Article{RSM:McV2006,
  author =       "E.R. McVeigh and M.A. Guttman and R.J. Lederman and M. Li
                 and O. Kocaturk and T. Hunt and S. Kozlov and K.A. Horvath",
  title =        "Real-time interactive MRI-guided cardiac surgery: aortic
                 valve replacement using a direct apical approach.",
  journal =      "Magn Reson Med",
  year =         "2006",
  month =        "Nov",
  volume =       "56",
  number =       "5",
  pages =        "958--964",
  robnote =      "Minimally invasive cardiac surgery requires arresting and
                 emptying of the heart, which compromises visualization of
                 the surgical field. In this feasibility study a novel
                 surgical procedure is demonstrated in which real-time MRI
                 is used to guide the placement of a prosthetic aortic
                 valve in the beating heart via direct apical access in
                 eight porcine hearts. A clinical stentless bioprosthetic
                 valve affixed to a platinum stent was compressed onto a
                 balloon-tipped catheter. This was fed through a 15-18-mm
                 delivery port inserted into the left ventricular (LV) apex
                 via a minimally invasive subxyphoid incision. Using
                 interactive real-time MRI, the surgeon implanted the
                 prosthetic valve in the correct location at the aortic
                 annulus within 90 s. In four of the animals immediately
                 after implantation, ventricular function, blood flow
                 through the valve, and myocardial perfusion were evaluated
                 with MRI. MRI-guided beating-heart surgery may provide
                 patients with a less morbid and more durable solution to
                 structural heart disease.",
  bibdate =      "Wed Aug 26 13:21:34 2009",
}

@Article{RSM:McW87,
  author =       "W.A. McWorter and J. M Tazelaar",
  title =        "Creating Fractals",
  journal =      j-BYTE,
  year =         "1987",
  month =        aug,
  pages =        "123--132",
  robnote =      "Seeing a completed drawing of a dragon can't compare
                 to the thrill of watching it being drawn. fractals.,
                 NumMath002",
}

@Article{RSM:Mee89c,
  author =       "B.N. Meeks",
  title =        "The Protocol Pack",
  journal =      j-BYTE,
  year =         "1989",
  month =        mar,
  pages =        "155--158",
  robnote =      "A short bit of info on the various file transfer
                 protocols and how each fits into the history of
                 things., CompHard173",
}

@InCollection{RSM:Mei86,
  author =       "B. Meier and W. Rutishauser",
  title =        "Intracoronary Electrocardiogram During Transluminal
                 Coronary Angioplasty",
  booktitle =    "Coronary Angioplasty: {A} Controlled Model for
                 Ischemia",
  editor =       "P.W. Serruys and G.T. Meester",
  publisher =    "Martinus Nijhoff Publishers",
  address =      "Dordrecht/Boston/Lancaster",
  year =         "1986",
  pages =        "33--37",
  robnote =      "thesis, PTCA046",
}

@Article{RSM:Mei87,
  author =       "B. Meier and P. Luethy and L. Finci and G.D.
                 Steffenino and W. Rutishauser",
  title =        "Coronary Wedge Pressure in Relation to Spontaneously
                 Visible and Recruitable Collaterals",
  journal =      j-C,
  year =         "1987",
  volume =       "75",
  pages =        "906--913",
  robnote =      "This is a neat paper about one way of trying to
                 quatify the amount of collateral circulation by looking
                 at the pressure difference between the proximal and
                 distal end of the inflation balloon in PTCA. thesis,
                 PTCA005",
}

@Article{RSM:Mei87b,
  author =       "J.W.H. Meijs and H.B.K. Boom and M.J. Peters and A.
                 van Oosterom",
  title =        "Application of the Richardson Extrapolation in
                 Simulation Studies in {EEG}s",
  journal =      "Med. \& Biol. Eng. \& Comput.",
  year =         "1987",
  volume =       "25",
  pages =        "222--226",
  robnote =      "One of the Meijs thesis article and one I have only
                 glanced at since it doesn't seem to apply to my stuff.
                 It does seem to talk about triangularization so I will
                 look at it more closely., TorsoModel011",
}

@PhdThesis{RSM:Mei88,
  author =       "H Meijs",
  title =        "The Influence of Head Geometries on Electro- and
                 Magnetoencephalograms",
  school =       "Dept. of Electrical Engineering, University of
                 Twente",
  address =      "the Netherlands",
  year =         "1988",
  robnote =      "This reference I have set up for all the unpublished
                 chapters of the thesis; the others will be found
                 elsewhere under their own heading. Another of the
                 important chapters in Meijs' thesis and one which talks
                 about the boundary element method., Prop022",
}

@Article{RSM:Mei89,
  author =       "J.W.H. Meijs and O.W. Weier and M.J. Peters and A. van
                 Oosterom",
  title =        "On the Numerical Accuracy of the Boundary Element
                 Method",
  journal =      j-BME,
  year =         "1989",
  volume =       "36",
  pages =        "1038--1049",
  robnote =      "An article version of the chapter on the same name in
                 Meijs' thesis, inverse problem, TorsoModel042",
}

@Article{RSM:Mel2004,
  author =       "J. Melo and P. Voigt and B. Sonmez and M. Ferreira and
                 M. Abecasis and M. Rebocho and A. Timoteo and C. Aguiar
                 and S. Tansal and H. Arbatli and R. Dion",
  title =        "Ventral cardiac denervation reduces the incidence of
                 atrial fibrillation after coronary artery bypass
                 grafting.",
  journal =      "J Thorac Cardiovasc Surg",
  year =         "2004",
  month =        feb,
  volume =       "127",
  number =       "2",
  pages =        "511--516",
  robnote =      "OBJECTIVES: Because the autonomic nervous system is an
                 important determinant in the appearance of atrial
                 fibrillation, we have assessed the role of ventral
                 cardiac denervation for its prevention. METHODS:
                 Patients undergoing low-risk coronary artery surgery
                 had ventral cardiac denervation performed in 207
                 patients, and 219 patients were used as control
                 subjects. RESULTS: The additional time for the
                 denervation was 5 +/- 2 minutes, and there were no
                 associated complications. Postoperative atrial
                 fibrillation was present in 15 (7\%) patients
                 undergoing ventral cardiac denervation (95\% confidence
                 interval, 4\%-12\%) and in 56 (27\%) control subjects
                 (95\% confidence interval, 18\%-35\%). Patients
                 submitted to ventral cardiac denervation had fewer and
                 less severe episodes of atrial fibrillation, and no
                 patient had atrial fibrillation after discharge.",
  bibdate =      "Tue Jun 12 09:33:45 2007",
}

@Article{RSM:Men70,
  author =       "C. Mendez and W.J. Mueller and X. Urguiaga",
  title =        "Propogation of Impulse Across the Purkinje
                 Fiber-Muscle Junctions in the Dog Heart",
  journal =      j-CR,
  year =         "1970",
  volume =       "26",
  pages =        "135--150",
  robnote =      "Ref 23 in Holland and Brooks rev., PTCA9, conduction
                 is delayed and then suppressed by increased potassium
                 of ischemia. don't have this one. thesis, Ischemia148",
}

@Article{RSM:Meng2013,
  author =       "Q. Meng and J. Hall and H. Rutigliano and X. Zhou and B. R.
                 Sessions and R. Stott and K. Panter and C. J. Davies and R.
                 Ranjan and D. Dosdall and R.S. MacLeod and N. Marrouche and
                 K. L. White and Z. Wang and I. A. Polejaeva",
  title =        "Generation of Cloned Transgenic Goats with Cardiac
                 Specific Overexpression Of Transforming Growth Factor
                 $\beta$1",
  journal =      "Reproduction, Fertility and Development",
  month =        jan,
  number =       "1",
  pages =        "162",
  volume =       "25",
  year =         "2013",
}

@Article{RSM:Mer77,
  author =       "W. Merx and M.S. Yoon and J Han",
  title =        "The role of local disparity in conduction and recovery
                 time on ventricular vulnerability to fibrillation",
  journal =      j-AHJ,
  year =         "1977",
  volume =       "94",
  number =       "5",
  pages =        "603--610",
  robnote =      "More background on the role of repolarization and VT
                 and VF.",
}

@Article{RSM:Mer90,
  author =       "R.R. Mercer and G.M. McCauley and S. Anjilvel",
  title =        "Approximation of surfaces in a quantitative {3-D}
                 reconstruction system",
  journal =      j-BME,
  year =         "1990",
  volume =       "37",
  pages =        "1136--1146",
  robnote =      "A lacing algorithm for trangularization, similar in
                 function to ours but a bit more refined,
                 forward/inverse model building",
}

@Article{RSM:Mer90b,
  author =       "M. Merri and D.C. Farden and J.G. Mottley and E.L.
                 Titlebaum",
  title =        "Sampling frequencies of the electrocardiogram for
                 spectral analysis of heart rate variability",
  journal =      j-BME,
  year =         "1991",
  volume =       "37",
  pages =        "99--106",
  robnote =      "A paper on sampling frequences necessary to evaluate
                 variations in heart rate. signal processing, autonomic
                 nervous system",
}

@InProceedings{RSM:Mer92,
  author =       "P.J. Mercurio and T.T. Elvins and S.J. Young",
  title =        "The microsopist's workstation",
  booktitle =    "Proceedings of the IEEE Visualization 92",
  year =         "1992",
  pages =        "419--423",
  publisher =    "IEEE CS Press",
  address =      "Los Alamitos, Calif.",
  robnote =      "A paper from Vis 92 using scientific visualization
                 with microscopy to provide users with images from a
                 remote electron microscope",
}

@Article{RSM:Mer92b,
  author =       "M. Merri and A.J. Moss and J. Benhorin and E.H. Locati
                 and M. Alberti and F. Badilini",
  title =        "Relation between ventricular repolarization duration
                 and cardiac cycle length during 24-hour Holter
                 recordings. {F}indings in normal patients and patients
                 with long {QT} syndrome",
  journal =      j-C,
  year =         "1992",
  volume =       "85",
  number =       "5",
  month  =        "May",
  pages =        "1816--21",
  robnote =      "The interval from the R wave to the maximum amplitude
                 of the T wave (RTm) contains the heart rate dependency
                 of ventricular repolarization. The relation of RTm to
                 the preceding RR interval (RTm/RR slope) was determined
                 by the best-fit linear regression equation between
                 these two parameters. 1) quantification of the dynamic
                 relation between ventricular repolarization and RR
                 cycle length can be obtained on a large number of
                 Holter-recorded heart beats; 2) beta-blockers reduce
                 the RTm/RR slope in normalpatients; and 3) LQTS
                 patients have an exaggerated delay in repolarization at
                 long RR cycle lengths. LQTS,",
}

@Article{RSM:Mes2007,
  author =       "D. R. Messroghli and K. Walters and S. Plein and P.
                 Sparrow and M. G. Friedrich and J. P. Ridgway and M. U.
                 Sivananthan",
  title =        "Myocardial {T1} mapping: application to patients with
                 acute and chronic myocardial infarction",
  journal =      "Magn Reson Med",
  volume =       "58",
  number =       "1",
  pages =        "34--40",
  year =         "2007",
}

@Article{RSM:Mes86,
  author =       "B.J. Messinger-Rapport and Y. Rudy",
  title =        "The Inverse Problem in Electrocardiography: {A} Model
                 Study of the Effects of Geometry and Conductivity
                 Parameters on the Reconstruction of Epicardial
                 Potentials",
  journal =      j-BME,
  year =         "1986",
  volume =       "33",
  month =        "Jul.",
  pages =        "667--676",
  robnote =      "A modelling paper on the inverse solution. It is
                 excellent and I use it quite a bit in the thesis.,
                 BSPM004",
}

@Article{RSM:Mes88,
  author =       "B.J. Messinger-Rapport and Y. Rudy",
  title =        "Regularization of the Inverse Problem in
                 Electrocardiography: {A} Model Study",
  journal =      j-MBS,
  year =         "1988",
  volume =       "89",
  pages =        "79--118",
  robnote =      "A major paper about the inverse solution,
                 regularization, boundary element methods and modelling.
                 thesis, TorsoModel006",
}

@Article{RSM:Mes90,
  author =       "B.J. Messinger-Rapport and Y. Rudy",
  title =        "Noninvasive recovery of epicardial potentials in a
                 realistic heart-torso geometry",
  journal =      j-CR,
  year =         "1990",
  volume =       "66",
  number =       "4",
  pages =        "1023--1039",
  robnote =      "TorsoModel, another inverse solution paper from the
                 Rudy team",
}

@Article{RSM:Met90,
  author =       "A.C. Metting van Rijn and A. Peper and C.A. Grimbergen",
  title =        "High-quality recording of bioelectric events. Part 1.
                 Interference reduction, theory and practice.",
  journal =      "Med Biol Eng Comput",
  year =         "1990",
  month =        "Sep",
  volume =       "28",
  number =       "5",
  pages =        "389--397",
  robnote =      "In the first part of the review the various mechanisms
                 that could be the cause of interference in bioelectric
                 recordings are considered. It is demonstrated that the
                 performance of a good amplifier can be seriously degraded
                 in its functioning if the whole measurement situation is
                 not taken into account. Several techniques used to reduce
                 interference, of which guarding and driven right leg
                 circuits are the most important, are analysed. In the
                 second part of the review some examples of the application
                 of the theory in practical situations are presented. The
                 instrumentation amplifier circuit normally used in
                 bioelectric recordings is improved for measurements under
                 difficult circumstances. Another application is a low-cost
                 64-channel amplifier for multichannel ECG recordings. The
                 third application is a device that can be added to
                 bioelectric measurement systems and will provide a major
                 reduction in interference.",
  bibdate =      "Sun Apr 5 13:37:43 2020",
}

@Article{RSM:Met91,
  author =       "A.C. Metting van Rijn and A. Peper and C.A. Grimbergen",
  title =        "High-quality recording of bioelectric events. Part 2.
                 Low-noise, low-power multichannel amplifier design.",
  journal =      "Med Biol Eng Comput",
  year =         "1991",
  month =        "Jul",
  volume =       "29",
  number =       "4",
  pages =        "433--440",
  robnote =      "A multichannel instrumentation amplifier, developed to be
                 used in a miniature universal eight-channel amplifier
                 module, is described. After discussing the specific
                 properties of a bioelectric recording, the difficulties of
                 meeting the demanded specifications with a design based on
                 operational amplifiers are reviewed. Because it proved
                 impossible to achieve the demanded combination of low
                 noise and low power consumption using commercially
                 available operational amplifiers, an amplifier equipped
                 with an input stage with discrete transistors was
                 developed. A new design concept was used to expand the
                 design to a multichannel version with an equivalent input
                 noise voltage of 0.35 microV RMS in a bandwidth of 0.1-100
                 Hz and a power consumption of 0.6 mW per channel. The
                 results of this study are applied to miniature, universal,
                 eight-channel amplifier modules, manufactured with
                 thick-film production techniques. The modules can be
                 coupled to satisfy the demand for a multiple of eight
                 channels. The low power consumption enables the modules to
                 be used in all kinds of portable and telemetry measurement
                 systems and simplifies the power supply in stationary
                 measurement systems.",
  bibdate =      "Sun Apr 5 13:37:43 2020",
}

@Article{RSM:Met94,
  author =       "A.C. MettingVanRijn and A. Peper and C.A. Grimbergen",
  title =        "Amplifiers for bioelectric events: a design with a
                 minimal number of parts.",
  journal =      "Med Biol Eng Comput",
  year =         "1994",
  volume =       "32",
  number =       "3",
  pages =        "305--310",
  robnote =      "A design for an amplifier for bioelectric events is
                 presented that has fewer parts than conventional designs.
                 The design allows the construction of amplifiers of a high
                 quality in terms of noise and common mode rejection, with
                 reduced dimensions and with a lower power consumption.
                 Gain, bandwidth and number of channels are easily adapted
                 to a wide range of biomedical applications. An application
                 example is given in the form of a multichannel EEG
                 amplifier (gain is 20,000), in which each channel consists
                 of three operational amplifiers (one single and one dual),
                 six resistors and two capacitors. The equivalent input
                 noise voltage and current are 0.15 microVrms and 1 pArms,
                 respectively, in a bandwidth of 0.2-40 Hz, and a common
                 mode rejection ratio of 136 dB is achieved without
                 trimming.",
  bibdate =      "Sun Apr 5 13:37:43 2020",
}

@Book{RSM:Mey93,
  author =       "Y. Meyer",
  title =        "Wavelets, Algorithms \& Applications",
  publisher =    "SIAM",
  year =         "1993",
}

@Article{RSM:Mic98,
  author =       "E.L. Michalets and L.K. Smith and TasselVan",
  title =        "Torsade de pointes resulting from the addition of
                 droperidol to an existing cytochrome {P450} drug
                 interaction.",
  journal =      "Ann Pharmacother",
  year =         "1998",
  volume =       "32",
  number =       "7-8",
  pages =        "761--5",
  robnote =      "The combination of cyclobenzaprine and fluoxetine
                 resulted in significant QT prolongation in our patient
                 that progressed to torsade de pointes after
                 preoperative droperidol administration. Resolution of
                 QT abnormalities after cyclobenzaprine discontinuation
                 provided further evidence of a drug-induced etiology.
                 Other possible medical and drug-related causes of
                 torsade de pointes are reviewed and ruled out. LQTS, No
                 hard copy",
}

@Article{RSM:Mie84,
  author =       "E.E. Mier",
  title =        "The Evolution of an Ethernet Standard",
  journal =      j-BYTE,
  year =         "1984",
  month =        dec,
  pages =        "131--142",
  robnote =      "A good into into Ethernet done up the Byte way, not to
                 complicated but inmformative., CompHard084",
}

@Article{RSM:Mii94,
  author =       "T. Miida and H. Oda and T. Toeda and N. Higuma",
  title =        "Additional {ST}-segment elevation immediately after
                 reperfursion and its effects on myocardial salvage in
                 anterior wall acute myocardial infarction",
  journal =      j-AJC,
  year =         "1994",
  volume =       "73",
  pages =        "851--855",
  robnote =      "A look at the long term outcome and clinical markers
                 in patients who show ST segment elevation even after
                 thrombolysis in anterior MIs. Suggests that when ST
                 shifts last, they are a bad sign for the outcome for
                 the patient. Mechanism not proven but proposed to be
                 reperfusion damage to cells.",
}

@InProceedings{RSM:Mil2009,
  author = 	 "M. Milanic and V. Jazbinsek and D.F. Wang and J.G. Sinstra
                   and R.S. Macleod and  D.H. Brooks and R. Hren",
  title = 	 "Evaluation of Approaches to Solving
                  Electrocardiographic Imaging Problem ",
  booktitle = 	 "Computers in Cardiology",
  year = 	 "2009",
  pages =	 "(in press)",
  publisher =	 "IEEE Press",
  robnote = 	 "inverse problems CIBC",
  bibdate = 	 "Mon Sep 07 01:58:48 2009"
}

@Article{RSM:Mil2014,
  author =       "M. Milanic and V. Jazbinsek and R.S. Macleod and D.H.
                 Brooks and R. Hren",
  title =        "Assessment of regularization techniques for
                 electrocardiographic imaging",
  journal =      j-JE,
  year =         "2014",
  month =        "Jan.-Feb.",
  volume =       "47",
  number =       "1",
  pages =        "20--28",
  robnote =      "A widely used approach to solving the inverse problem in
                 electrocardiography involves computing potentials on the
                 epicardium from measured electrocardiograms (ECGs) on the
                 torso surface. The main challenge of solving this
                 electrocardiographic imaging (ECGI) problem lies in its
                 intrinsic ill-posedness. While many regularization
                 techniques have been developed to control wild
                 oscillations of the solution, the choice of proper
                 regularization methods for obtaining clinically acceptable
                 solutions is still a subject of ongoing research. However
                 there has been little rigorous comparison across methods
                 proposed by different groups. This study systematically
                 compared various regularization techniques for solving the
                 ECGI problem under a unified simulation framework,
                 consisting of both 1) progressively more complex idealized
                 source models (from single dipole to triplet of dipoles),
                 and 2) an electrolytic human torso tank containing a live
                 canine heart, with the cardiac source being modeled by
                 potentials measured on a cylindrical cage placed around
                 the heart. We tested 13 different regularization
                 techniques to solve the inverse problem of recovering
                 epicardial potentials, and found that non-quadratic
                 methods (total variation algorithms) and first-order and
                 second-order Tikhonov regularizations outperformed other
                 methodologies and resulted in similar average
                 reconstruction errors.",
  bibdate =      "Sun Sep 21 21:53:35 2014",
  pmcid =        "PMC4154607",
}

@Article{RSM:Mil78,
  author =       "W.T. Miller and D.B. Geselowitz",
  title =        "Simulation Studies of the Electrocardiogram: {I} {T}he
                 Normal Heart and {II} {I}schemia and Infarction",
  journal =      j-CR,
  year =         "1978",
  volume =       "4",
  pages =        "301--323",
  robnote =      "The original paper about the Miller-Geselowitz model.,
                 Prop002",
}

@Article{RSM:Mil85,
  author =       "C.K. Millar and F.A. Kralios and R.L. Lux",
  title =        "Correlation Between Refractory Periods and
                 Activation-recovery Intervals from Electrograms: Effect
                 of Rate and Adrenergic Interventions",
  journal =      j-C,
  year =         "1985",
  volume =       "72",
  pages =        "1372--1379",
  robnote =      "Another Salt Lake City paper looking at a method of
                 determining refractory period and activation recovery
                 intervals from electrograms using neural control of the
                 heart., BSPM071",
}

@Article{RSM:Mil87,
  author =       "E.K. Miller",
  title =        "Electrical Engineering and the Computer",
  journal =      "IEEE Potentials",
  year =         "1987",
  pages =        "44--48",
  robnote =      "A look at pratical uses of computer modeling in
                 industry and academe., NumMath005",
}

@Article{RSM:Mil90,
  author =       "B.P. Miller and L. Fredriksen and B. So",
  title =        "Study of the reliability of {UNIX} utilities",
  journal =      j-ACM-COM,
  year =         "1990",
  volume =       "33",
  number =       "12",
  pages =        "33--44",
  robnote =      "A study which revealed a lot of inconsistency between
                 Unix implementations of a number of the basic Unix
                 tools.",
}

@Article{RSM:Min13,
  author =       "G.R. Mines",
  title =        "On Dynamic Equillibrium in the Heart",
  journal =      j-JP,
  year =         "1913",
  volume =       "4",
  pages =        "349--383",
  robnote =      "Another classic of fibrillation, arrhythmia and
                 propagation, Arrhythm007",
}

@Article{RSM:Min14,
  author =       "G.R. Mines",
  title =        "On circulating excitations in the heart muscles and
                 their possibilitie relation to tachycardia and
                 fibrillation",
  journal =      "Trans. R. Soc. Can.",
  year =         "1914",
  volume =       "14",
  pages =        "43--52",
  robnote =      "Do not have hard copy but another classic reference in
                 arrhythmias, fibrillation",
  bibdate =      "Wed Nov 11 10:47:56 1998",
}

@Article{RSM:Mir2016,
  author =       "G.R. Mirams and P.  Pathmanathan and R.A. Gray and
                  P. Challenor and R.H. Clayton",
  title =        "Uncertainty and variability in computational and
                 mathematical models of cardiac physiology.",
  journal =      j-JP,
  year =         "2016",
  month =        "Dec",
  volume =       "594",
  number =       "23",
  pages =        "6833--6847",
  robnote =      "UQ: Mathematical and computational models of
                 cardiac physiology have been an integral component of
                 cardiac electrophysiology since its inception, and are
                 collectively known as the Cardiac Physiome. We identify
                 and classify the numerous sources of variability and
                 uncertainty in model formulation, parameters and other
                 inputs that arise from both natural variation in
                 experimental data and lack of knowledge. The impact of
                 uncertainty on the outputs of Cardiac Physiome models is
                 not well understood, and this limits their utility as
                 clinical tools. We argue that incorporating variability
                 and uncertainty should be a high priority for the future
                 of the Cardiac Physiome. We suggest investigating the
                 adoption of approaches developed in other areas of science
                 and engineering while recognising unique challenges for
                 the Cardiac Physiome; it is likely that novel methods will
                 be necessary that require engagement with the mathematics
                 and statistics community. ABSTRACT: The Cardiac Physiome
                 effort is one of the most mature and successful
                 applications of mathematical and computational modelling
                 for describing and advancing the understanding of
                 physiology. After five decades of development,
                 physiological cardiac models are poised to realise the
                 promise of translational research via clinical
                 applications such as drug development and patient-specific
                 approaches as well as ablation, cardiac resynchronisation
                 and contractility modulation therapies. For models to be
                 included as a vital component of the decision process in
                 safety-critical applications, rigorous assessment of model
                 credibility will be required. This White Paper describes
                 one aspect of this process by identifying and classifying
                 sources of variability and uncertainty in models as well
                 as their implications for the application and development
                 of cardiac models. We stress the need to understand and
                 quantify the sources of variability and uncertainty in
                 model inputs, and the impact of model structure and
                 complexity and their consequences for predictive model
                 outputs. We propose that the future of the Cardiac
                 Physiome should include a probabilistic approach to
                 quantify the relationship of variability and uncertainty
                 of model inputs and outputs.",
  bibdate =      "Sat Jul 27 09:37:34 2019",
  pmcid =        "PMC5134370",
}

@Article{RSM:Mir77,
  author =       "D.M Mirvis and F.W. Keller and R.E. Ideker and J.W.
                 Cox and R.F. Dowdie and D.G. Zettergren",
  title =        "Detection and localization of a multple epicardial
                 electrical generator by a two dipole ranging
                 technique.",
  journal =      j-CR,
  year =         "1977",
  volume =       "41",
  pages =        "551",
  robnote =      "no hard copy but Mir78b builds on this one. discrete
                 source model of the heart with validation using rabbit
                 hearts in the spherical tank.",
  bibdate =      "Sun Oct 31 00:42:21 1999",
}

@Article{RSM:Mir78,
  author =       "D.M. Mirvis and F.W. Keller and R.E. Ideker and D.G.
                 Zettergren and R.F. Dowdie",
  title =        "Equivalent Generator Properties of Acute Ischemic
                 Lesions in the Isolated Rabbit Heart",
  journal =      j-CR,
  year =         "1978",
  volume =       "42",
  pages =        "676--685",
  robnote =      "A reference from either Mirvis's book or one of his
                 papers, looking at the injury current which results
                 from localized injury to the heart. thesis,
                 Ischemia152",
}

@Article{RSM:Mir78b,
  author =       "D.M. Mirvis and F.W. Keller and J.W. Cox",
  title =        "Experimenal comparison of four inverse
                 electrocardiographic constructs in the isolated rabbit
                 heart",
  journal =      j-JE,
  year =         "1978",
  volume =       "11",
  number =       "1",
  pages =        "57--65",
  robnote =      "inverse solutions based on discrete models including
                 centric multipole series, single moving dipole, two
                 moving dipoles, and moving dipole-quadripole pair. Used
                 a spherical electrolytic tank of 6.35 cm diameter with
                 32 electrodes. They alsop describe some strange signal
                 process, recording in bipoles and then creating
                 unipoles by subtracting a mean potential from each?
                 Showed that the single moving dipole was not adequate
                 but that all three other techniques performed about the
                 same.",
  bibdate =      "Sun Oct 31 00:14:21 1999",
}

@Article{RSM:Mir79,
  author =       "D.M. Mirvis",
  title =        "Electrocardiographic {QRS} Changes Induced By Acute
                 Coronary Ligation in the Isolated Rabbit Heart",
  journal =      j-JE,
  year =         "1979",
  volume =       "12",
  pages =        "141--150",
  robnote =      "This one looks good too. thesis, Ischemia153",
}

@Article{RSM:Mir79b,
  author =       "D.M. Mirvis",
  title =        "Electrocardiographic {QRS} changes induced by acute
                 coronary ligation in the isolated rabbit heart",
  journal =      j-JE,
  year =         "1979",
  volume =       "12",
  number =       "2",
  pages =        "141--150",
  robnote =      "No hard copy",
}

@Article{RSM:Mir80,
  author =       "D.M. Mirvis",
  title =        "Body Surface Distributions of Repolarization Forces
                 During Acute Myocardial Infarction: {I.} {I}sopotential
                 and Isoarea Mapping",
  journal =      j-C,
  year =         "1980",
  volume =       "62",
  pages =        "878--887",
  robnote =      "The idea of depolarizing vs repolarizing forces,
                 apparently from Taccardi, is outlined and used to
                 interpret maps of MI patients. Any event which carries
                 beyond the J-point is a repolarizing force and the
                 earliest occurance of such features is found to change
                 (happen still earlier) in MI patients. This shift of
                 repolarization has its cellular basis in the shortened
                 AP of ischemic cell. ST- maps used as well and their
                 dipolarity is discussed. Single max/min can but need
                 not be sign of dipolar source; more numerous extrema
                 cannot be produced by a dipolar source. QRST maps
                 displayed but without comment, that is left for
                 Abildskov. Precordial arrays shown to fail because they
                 do not cover enough area. Clinical criteria for
                 location of infarct based on standard leads is also
                 described. thesis, Ischemia146",
}

@Article{RSM:Mir80b,
  author =       "D.M. Mirvis",
  title =        "Body surface distribution of exercise-induced {QRS}
                 changes in normal subjects",
  journal =      j-AJC,
  year =         "1980",
  volume =       "46",
  pages =        "988--996",
  robnote =      "A look at the effect of exercise on BSPM patterns.
                 Mirvis finds difference in amplitude but not pattern
                 and concludes that changes in blood mass and atrial
                 repolarization are not enough to explain the
                 differences.",
}

@Article{RSM:Mir81,
  author =       "D.M. Mirvis",
  title =        "Body surface distributions of replarization potentials
                 after acute myocardial infarctions. {II.}
                 {R}elationship between isopotential mapping and
                 {ST}-segment potential summation methods.",
  journal =      j-C,
  year =         "1981",
  volume =       "63",
  number =       "3",
  pages =        "623--631",
  robnote =      "A nice paper showing how large the errors can be when
                 precordial leads are used instead of maps to determine
                 total ST deviation after acute MI",
}

@Article{RSM:Mir85,
  author =       "D.M. Mirvis and L. Ingram and M.K. Holly and J.L.
                 Wilson and K.B. Ramanathan",
  title =        "Electrocardiographic Effects of Experimental
                 Nontransmural Myocardial Infarction",
  journal =      j-C,
  year =         "1985",
  volume =       "71",
  pages =        "1206--1214",
  robnote =      "thesis, Ischemia157",
}

@Article{RSM:Mir85b,
  author =       "D. Mirvis",
  title =        "Spatial variation of {QT} intervals in normals persons
                 and patients with acute myocardial infarctions.",
  journal =      j-ACC,
  year =         "1985",
  volume =       "5",
  number =       "3",
  pages =        "625--631",
  robnote =      "Looks at QT durations in BSPM data for a normal group
                 and a most MI group. Nice paper, describes both the
                 methods used and the problems of determining QT
                 intervals from the signals, and the resulting map
                 distributions. Normals have a pretty stable pattern,
                 while infarct patients have a pattern that depends on
                 artery and time. There is a biphasic pattern of change
                 in the pattern.",
}

@InCollection{RSM:Mir88,
  author =       "D.M. Mirvis",
  title =        "Acute Myocardial Infarction",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "125--136",
  robnote =      ", BSPM064",
}

@InCollection{RSM:Mir88b,
  author =       "D.M. Mirvis",
  title =        "Atrial Depolarization and Repolarization",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "77--86",
  robnote =      ", BSPM060",
}

@InCollection{RSM:Mir88c,
  author =       "D.M. Mirvis",
  title =        "Chronic Myocardial Infarction",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "137--152",
  robnote =      ", BSPM065",
}

@InCollection{RSM:Mir88d,
  author =       "D.M. Mirvis",
  title =        "Methods for Body Surface Electrocardiographic
                 Mapping",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "43--62",
  robnote =      ", BSPM058",
}

@InCollection{RSM:Mir88e,
  author =       "D.M. Mirvis",
  title =        "Validation of body surface electrocardiographic
                 mapping",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "63--74",
  robnote =      ", BSPM059",
}

@InCollection{RSM:Mir88f,
  author =       "D.M. Mirvis",
  title =        "Myocardial Ischemia and Exercise Stress Testing",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "111--124",
  robnote =      ", BSPM063",
}

@InCollection{RSM:Mir88g,
  author =       "D.M. Mirvis",
  title =        "Rationale for Body Surface Electrocardiographic
                 Mapping",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "31--42",
  robnote =      ", BSPM057",
}

@InCollection{RSM:Mir88h,
  author =       "D.M. Mirvis",
  title =        "Ventricular Depolarization",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "87--96",
  robnote =      ", BSPM061",
}

@InCollection{RSM:Mir88i,
  author =       "D.M. Mirvis",
  title =        "Ventricular Repolarization",
  booktitle =    "Body Surface Electrocardiographic Mapping",
  editor =       "D.M. Mirvis",
  publisher =    "Kluwer Academic Publishers",
  address =      "Boston, Dordrecht, London",
  year =         "1988",
  pages =        "97--108",
  robnote =      ", BSPM062",
}

@Article{RSM:Mir88j,
  author =       "D. M Mirvis",
  title =        "Physiologic Bases for Anterior {ST}-Segment Depression
                 in Patients with Acute Inferior Wall Myocardial
                 Infarction",
  journal =      "Curr. in Cardiology",
  year =         "1988",
  volume =       "116",
  pages =        "1308--1322",
  robnote =      "Quite interesting but somewhat simple-minded in the
                 idea of dipole theory being applied to explain both
                 primary and secondary ST-segment changes, while
                 cancellation is never really discussed. The idea of
                 ischemia at a distance is explained here, the first
                 I've ever heard of it, but perhaps of some importance.
                 A couple of references with checking. thesis,
                 Ischemia108",
}

@Article{RSM:Mis2005,
  author =       "S. Missan and T. F. McDonald",
  title =        "{CESE}: {C}ell {E}lectrophysiology {S}imulation
                 {E}nvironment",
  journal =      "Appl. Bioinformatics",
  year =         "2005",
  volume =       "4",
  number =       "2",
  pages =        "155--156",
  robnote =      "Cell electrophysiology simulation environment (CESE)
                 is an integrated environment for performing simulations
                 with a variety of electrophysiological models that have
                 Hodgkin-Huxley and Markovian formulations of ionic
                 currents. CESE is written in Java 2 and is readily
                 portable to a number of operating systems. CESE allows
                 execution of single-cell models and modification and
                 clamping of model parameters, as well as data
                 visualisation and analysis using a consistent
                 interface. Model creation for CESE is facilitated by an
                 object-oriented approach and use of an extensive
                 modelling framework. The Web-based model repository is
                 available. AVAILABILITY: CESE and the Web-based model
                 repository are available at
                 http://cese.sourceforge.net/.",
}

@Article{RSM:Mit2000,
  author =       "J.S. Mitcheson and J. Chen and M. Lin and C. Culberson
                 and M.C. Sanguinetti",
  title =        "A structural basis for drug-induced long {QT}
                 syndrome.",
  journal =      "Proc Natl Acad Sci U S A",
  year =         "2000",
  month =        oct,
  volume =       "97",
  number =       "22",
  pages =        "12329--12333",
  robnote =      "Mutations in the HERG K(+) channel gene cause
                 inherited long QT syndrome (LQT), a disorder of cardiac
                 repolarization that predisposes affected individuals to
                 lethal arrhythmias. Acquired LQT is far more common and
                 is most often caused by block of cardiac HERG K(+)
                 channels by commonly used medications. It is unclear
                 why so many structurally diverse compounds block HERG
                 channels, but this undesirable side effect now is
                 recognized as a major hurdle in the development of new
                 and safe drugs. Here we use alanine-scanning
                 mutagenesis to determine the structural basis for
                 high-affinity drug block of HERG channels by MK-499, a
                 methanesulfonanilide antiarrhythmic drug. The
                 antihistamine terfenadine and a gastrointestinal
                 prokinetic drug, cisapride, interact with Y652 and
                 F656, but not with V625. The aromatic residues of the
                 S6 domain that interact with these drugs (Y652 and
                 F656) are unique to eag/erg K(+) channels. Other
                 voltage-gated K(+) (Kv) channels have Ile and Val (Ile)
                 in the equivalent positions. These findings suggest a
                 possible structural explanation for how so many
                 commonly used medications block HERG but not other Kv
                 channels and should facilitate the rational design of
                 drugs devoid of HERG channel binding activity.",
  bibdate =      "Tue Oct 30 15:16:38 2001",
}

@Article{RSM:Mit68,
  author =       "M. Okajima and T. Fujinaa and T. Kobayashi and K.
                 Yamada",
  title =        "Computer simulation of the propagation process in
                 excitation of the ventricles",
  journal =      j-CR,
  year =         "1968",
  volume =       "23",
  pages =        "203--211",
  robnote =      "The earliest reference I have seen for a cellular
                 automata model of propagation in the heart (other than
                 Moe)",
}

@Article{RSM:Mit86,
  author =       "H. Mittelbach",
  title =        "Easy 3-{D} Graphics",
  journal =      j-BYTE,
  year =         "1986",
  month =        jan,
  pages =        "153--155",
  robnote =      "A BASIC program for plotting 3-D surfaces. Simple but
                 effective, I guess, and explains the basics of the
                 whole 3-D graphics business., NumMath013",
}

@Article{RSM:Miu2004,
  author =       "T. Miura and Y. Ohnuma and A. Kuno and M. Tanno and Y.
                 Ichikawa and Y. Nakamura and T. Yano and T. Miki and J.
                 Sakamoto and K. Shimamoto",
  title =        "Protective role of gap junctions in preconditioning
                 against myocardial infarction",
  journal =      "AJP-Heart",
  year =         "2004",
  volume =       "286",
  pages =        "214--221",
}

@Article{RSM:Miy2006,
  author =       "Y. Miyasaka and M.E. Barnes and B.J. Gersh and S.S. Cha
                 and K.R. Bailey and W.P. Abhayaratna and J.B. Seward and
                 T.S. Tsang",
  title =        "Secular trends in incidence of atrial fibrillation in
                 Olmsted County, Minnesota, 1980 to 2000, and implications
                 on the projections for future prevalence.",
  journal =      j-C,
  year =         "2006",
  month =        "Jul",
  volume =       "114",
  number =       "2",
  pages =        "119--125",
  robnote =      "BACKGROUND:  We assessed the community-based
                 trends in AF incidence for 1980 to 2000 and provided
                 prevalence projections to 2050. METHODS AND RESULTS: The
                 adult residents of Olmsted County, Minnesota, who had
                 ECG-confirmed first AF in the period 1980 to 2000 (n=4618)
                 were identified. Trends in age-adjusted incidence were
                 determined and used to construct model-based prevalence
                 estimates. The age- and sex-adjusted incidence of AF per
                 1000 person-years was 3.04 (95\% CI, 2.78 to 3.31) in 1980
                 and 3.68 (95\% CI, 3.42 to 3.95) in 2000. According to
                 Poisson regression with adjustment for age and sex,
                 incidence of AF increased significantly (P=0.014), with a
                 relative increase of 12.6\% (95\% CI, 2.1 to 23.1) over 21
                 years. The increase in age-adjusted AF incidence did not
                 differ between men and women (P=0.84). According to the US
                 population projections by the US Census Bureau, the number
                 of persons with AF is projected to be 12.1 million by
                 2050, assuming no further increase in age-adjusted
                 incidence of AF, but 15.9 million if the increase in
                 incidence continues. CONCLUSIONS: The age-adjusted
                 incidence of AF increased significantly in Olmsted County
                 during 1980 to 2000. Whether or not this rate of increase
                 continues, the projected number of persons with AF for the
                 United States will exceed 10 million by 2050, underscoring
                 the urgent need for primary prevention strategies against
                 AF development.",
  bibdate =      "Sun May 30 21:58:39 2010",
}

@Article{RSM:Mod2002,
  author =       "R. Modre and B. Tilg and G. Fischer and P. Wach",
  title =        "Noninvasive myocardial activation time imaging: a
                 novel inverse algorithm applied to clinical {ECG}
                 mapping data.",
  journal =      j-BME,
  year =         "2002",
  month =        oct,
  volume =       "49",
  number =       "10",
  pages =        "1153--1161",
  robnote =      "We have developed an iterative algorithm solving this
                 nonlinear problem by a sequence of regularized linear
                 problems. At each step of iteration, an individual
                 L-curve is computed. Subsequent iteration steps are
                 performed with the individual optimal regularization
                 parameter. This novel approach is compared with the
                 standard Gauss-Newton approach. Both methods are
                 applied to simulated ECG mapping data as well as to
                 single beat sinus rhythm data from two patients
                 recorded in the catheter laboratory. The proposed
                 approach shows excellent numerical and computational
                 performance, even under clinical conditions at which
                 the Gauss-Newton approach begins to break down.",
  bibdate =      "Sun Dec 26 11:48:03 2004",
}

@InProceedings{RSM:Mod2002b,
  author =       "R. Modre and B. Tilg and G. Fischer and F. Hanser and
                 B. Messnarz and F.X. Roithinger and F. Hintringer",
  title =        "A clinical pilot study on the accessory pathway
                 localization accuracy applying {ECG} mapping",
  booktitle =    c-EMBS2002,
  year =         "2002",
  volume =       "2",
  pages =        "1381--1382",
  robnote =      "Our objective was to quantify the localization error
                 of noninvasive activation time (AT) imaging by
                 localizing an accessory pathway for a
                 Wolff-Parkinson-White syndrome and a pacing site for
                 left ventricle pacing. Anatomical data obtained by
                 magnetic resonance imaging and electrocardiographic
                 mapping enables the imaging of the AT map on the entire
                 surface of the heart. The nonlinear inverse problem is
                 solved by a sequence of linearized ill-posed problems.
                 The localization accuracy in this clinical pilot study
                 for localizing a single pacing site and an accessory
                 pathway was 8mm and 5mm with a inaccuracy of this
                 distance of 10mm.",
  bibdate =      "Sun Dec 26 11:53:05 2004",
}

@Article{RSM:Moe41,
  author =       "G.K. Moe and A.S. Harris and C.J. Wiggers",
  title =        "Analysis of the Initiation of Fibrillation by
                 Electrographic Studies",
  journal =      j-AJP,
  year =         "1941",
  volume =       "13",
  pages =        "473--491",
  robnote =      "Arrhythm012, used cardiac mapping techniques, looks at
                 origins of fibrillation",
}

@Article{RSM:Moe59,
  author =       "G.K. Moe and J.A. Abildskov",
  title =        "Atrial Fibrillation as a Self-Sustaining Arrhythmia
                 Independent of Focal Discharge",
  journal =      j-AHJ,
  year =         "1959",
  volume =       "5",
  pages =        "59--70",
  robnote =      ", Arrhythm009",
}

@Article{RSM:Moe64,
  author =       "G.K. Moe and W.C. Rheinboldt and J.A. Abildskov",
  title =        "A Computer Model of Fibrillation",
  journal =      j-AHJ,
  year =         "1964",
  volume =       "67",
  pages =        "200--220",
  robnote =      "Moe uses cellular automata to describe propagation and
                 fibrillation, a very nice paper., Arrhythm006",
}

@Book{RSM:Moh97,
  author =       "D.R. Mohrman and L.J. Heller",
  title =        "Cardiovascular Physiology",
  publisher =    pub-MH,
  year =         "1997",
  robnote =      "Great book on CV physiology. I use it now for the BE
                 6010 course",
  bibdate =      "Sun Jan 09 23:01:43 2000",
}

@Article{RSM:Mon74,
  author =       "D.M. Monro and R.A.L. Guardo and P.J. Bourdillon and
                 J. Tinker",
  title =        "A Fourier Technique for Simultaneous
                 Electrocardiographic Surface Mapping",
  journal =      "Cardiovasc. Res.",
  year =         "1974",
  volume =       "8",
  pages =        "688--700",
  robnote =      "A paper describing the London mapping system and the
                 way they use 2-D Fourier techniques to interpolate the
                 points between the electrodes for the purpose of
                 plotting isopotential maps., BSPM096",
}

@Article{RSM:Mon78,
  author =       "D.M. Monro and P.J. Bourdillon",
  title =        "Techniques for Surface and Epicardial Isopotential
                 Mapping",
  journal =      j-AC,
  year =         "1978",
  volume =       "21",
  pages =        "11--14",
  robnote =      "Maps produced by Fourier interpolation. This is a
                 short paper from a series of a conference. A recording
                 of both body surface and epicardial maps of the same
                 patient were done here, BSPM106",
}

@Article{RSM:Mon80,
  author =       "D.M. Monroe",
  title =        "Interpolation methods for surface mapping",
  journal =      "Comp. Prog. in Biomedicine",
  year =         "1980",
  volume =       "11",
  pages =        "145--157",
  robnote =      "Describes use of Fourier, Chebyshev, and cubic
                 inteprolation methods for regularly and irregularly
                 spaced data points.",
}

@Article{RSM:Mon81,
  author =       "T.J. Montague and E.R. Smith and D.A. Cameron and P.M.
                 Rautaharju and G.A. Klassen and C.S. Flemington and
                 B.M. Horacek",
  title =        "Isointegral Analysis of Body Surface Maps: Surface
                 Distribution and Temporal Variability in Normal
                 Subjects",
  journal =      j-C,
  year =         "1981",
  volume =       "63",
  pages =        "1167--1172",
  robnote =      "The paper which desribes what the normal integral maps
                 should look like, integral maps. thesis, BSPM011",
}

@Article{RSM:Mon84,
  author =       "T.J. Montague and J.P. Finley and K. Mukelabai and S.
                 Black and S.M. Rigby and A. Spencer and B.M.
                 Horacek",
  title =        "Cardiac Rhythm, Rate and Ventricular Repolarization
                 Properties in Infants at Risk for Suddent Infant Death
                 Syndrome: Comparison with Age- and Sex-Matched Control
                 Infants",
  journal =      j-AJC,
  year =         "1984",
  volume =       "54",
  pages =        "301--307",
  robnote =      "SIDS and BSPM study, one that Milan always said had
                 more promise but was not pursued long enough to really
                 get the information out. thesis, BSPM044",
}

@Article{RSM:Mon84b,
  author =       "T.J. Montague and E.R. Smith and D.E. Johnstone and
                 C.A. Spencer and L.D. Lalonde and R.M. Bessoudo and
                 M.J. Gardner and R.N. Anderson and B.M.
                 Horacek",
  title =        "Temporal Evaluation of Body Surface Mapping Patterns
                 Following Acute Inferior Myocardial Infarction",
  journal =      j-JE,
  year =         "1984",
  volume =       "17",
  pages =        "319--328",
  robnote =      "thesis, BSPM010",
}

@Article{RSM:Mon86,
  author =       "T.J. Montague and D.E. Johnstone and A. Spencer and
                 L.D. Lalonde and M.J. Gardner and M.G. O'Reilly and
                 B.M. Horacek",
  title =        "Non-{Q}-Wave Acute Myocardial Infarction: {B}ody
                 Surface Potential Map and Ventriculographic Patterns",
  journal =      j-AJC,
  year =         "1986",
  volume =       "58",
  pages =        "1173--1180",
  robnote =      "Another one of TJ's clinical papers on MI patients and
                 bspm. thesis, BSPM051",
}

@Article{RSM:Mon86b,
  author =       "T.J. Montague and D.D. McPherson and D.E. Johnstone
                 and A. Spencer and L.D. Lalonde and M.J. Gardner and
                 B.M. Horacek",
  title =        "Electrocardiographic and Ventriculographic Recovery
                 Patterns in {Q}-Wave Myocardial Infarction",
  journal =      j-ACC,
  year =         "1986",
  volume =       "8",
  pages =        "521--528",
  robnote =      "Another one of TJ's clinical papers on MI patients and
                 bspm. thesis thesis, BSPM052",
}

@Unpublished{RSM:Mon86c,
  author =       "T.J. Montague",
  title =        "Echocardiology Laboratory Teaching Manual, Sept.
                 1986",
  year =         "1986",
  robnote =         "A manual TJ gave me on echocardiography and how it is
                 set up the VG. Residents receive the same thing for
                 their stints in cardiology., Cardiol008",
}

@Unpublished{RSM:Mon86d,
  author =       "T.J. Montague",
  title =        "Cirriculum Vitae",
  year =         "1986",
  robnote =         "TJ's CV, vita, don't quote me on the date",
}

@Article{RSM:Mon86e,
  author =       "D.M. Monro and P.J. Bones and R. de Stanbridge and
                 R.W. Jones",
  title =        "Comparison of Epicardial and Body Surface {ECG}
                 Potentials in Man",
  journal =      "Cardiovasc. Res.",
  year =         "1986",
  volume =       "20",
  pages =        "201--207",
  robnote =      "These guys used epicardial leads which are left in
                 place after bypass surgery to monitor things, and
                 compared them with the surface potentials they recorded
                 at 37 sites. They describe some nice technical details
                 as well, eg. epicardial potentials are as large as 21.2
                 mV and surface potentials are recorded at 12 bits with
                 LSB = 2.5 microvolt. Biological noise was mostly below
                 30 microvolt, while system noise < 4 micorvolts.
                 Important sort of paper for the inverse solution. Lots
                 of good references. thesis, TorsoModel004",
}

@Unpublished{RSM:Mon87,
  author =       "T.J. Montague and D.E. Johnstone and A. Apencer and
                 R.M. Miller and B.R. MacKenzie and M.J. Gardner and
                 B.M. Horacek",
  title =        "Quantitation of Myocardial Ischemia by Body Surface
                 Potential Mapping: Exercise Maps in Patients with
                 Isolated left Anterior Descending Coronary Artery
                 Disease",
  year =         "1987",
  robnote =         "A draft of a paper - don't know what the fate of this
                 was BSPM034",
}

@InProceedings{RSM:Mon87b,
  author =       "T.J. Montague and D.E. Johnstone and R.M. Miller and
                 B.R. MacKenzie and M.J. Gardner and B.M.
                 Horacek",
  title =        "Quantitative Body Surface Mapping: Exercise Maps in
                 Patients with Single and Multiple Coronary Artery
                 Obstructions",
  booktitle =    "Canadian Cardiovascular Society Annual Meeting",
  organization = "Canadian Cardiovascular Society",
  address =      "",
  year =         "1987",
  robnote =      "Another abstract from the Canadian Cardiovascular
                 Society meeting, Oct, 1987., BSPM033",
}

@InProceedings{RSM:Mon87c,
  author =       "T.J. Montague and R.G. Macdonald and M.A. Henderson
                 and R.M. Miller and B.M. Horacek",
  title =        "Quantitative Body Surface Mapping: Resting Maps Before
                 and After Successful Angioplsty",
  booktitle =    "Canadian Cardiovascular Society Annual Meeting",
  organization = "Canadian Cardiovascular Society",
  address =      "",
  year =         "1987",
  robnote =      "An abstract Terry submitted, and subsequently
                 presented at the Canadian Cardiovascular Society,
                 BSPM031",
}

@Unpublished{RSM:Mon87d,
  author =       "T.J. Montague and R.G. Macdonald and M.A. Henderson
                 and B.M. Chandler",
  title =        "Quantitative Electrocardiographic Essessment of
                 Myocardial Ischemia Pre- and Post-Coronary
                 Angioplasty",
  year =         "1987",
  robnote =         "A protocol for PTCA mapping, pre and post procedure.
                 Terry used this one at some stage to get approval to do
                 maps on ptca patients., BSPM032",
}

@Unpublished{RSM:Mon87e,
  author =       "T.J. Montague",
  title =        "Body Surface Mapping in Acute Ischemia: Quantification
                 of Ischemic Myocardium During Coronary Angioplasty",
  year =         "1987",
  robnote =         "A grant application to the Heritage Medical
                 Scholarship fund for his work in Edmonton., BSPM043",
}

@Article{RSM:Mon88,
  author =       "T.J. Montague and D.E. Johnstone and A. Spencer and
                 R.M. Miller and B.R. MacKenzie and M.J. Gardner and
                 B.M. Horacek",
  title =        "Body Surface Potential Maps with Low-Level Exercise in
                 Isolated Left Anterior Descending Coronary Artery
                 Disease",
  journal =      j-AJC,
  year =         "1988",
  volume =       "61",
  pages =        "273--282",
  robnote =      "BSPM054",
}

@Unpublished{RSM:Mon88b,
  author =       "T.J. Montague and D.E. Johnstone and R.M. Miller and
                 B.R. MacKenzie and M.J. Gardner and B.M.
                 Horacek",
  title =        "Quantitative Body Surface Potential Mapping: Exercise
                 Maps in Single and Multiple Coronary Artery
                 Obstruction",
  journal =      "",
  year =         "",
  robnote =         "A paper Terry submitted and was published I think.
                 exercise and ECG changes in maps., BSPM042",
}

@Article{RSM:Mon89,
  author =       "T.J. Montague and F. X Witkowski",
  title =        "The Clinical Utility of Body Surface Potential Mapping
                 in Coronary Artery Disease",
  journal =      j-AJC,
  year =         "1989",
  volume =       "64",
  pages =        "378--383",
  robnote =      "The latest in Terry's mapping papers, this one in the
                 form of an editorial review of the current mapping
                 scene as he sees it. No new data, but some use of our
                 early PTCA results. thesis, BSPM111",
}

@Unpublished{RSM:Mon89b,
  author =       "Montague T.J.",
  title =        "Terry Montague -- Cirriculum Vitae",
  year =         "1989",
  robnote =         "TJ's latest CV as sent to Lynne for the department
                 report., BSPM0102",
}

@Article{RSM:Mon89c,
  author =       "T.J. Montague and R.M. Miller and M.A. Henderson and
                 R.G. Macdonald and R.S. MacLeod and F.X. Witkowski and
                 B.M. Horacek",
  title =        "Persistent Changes in the Body Surface
                 Electrocardiogram Following Successful Coronary
                 Angioplasty",
  journal =      j-JE,
  year =         "1989",
  volume =       "22",
  number =       "Suppl {III}",
  pages =        "91--98",
  robnote =      "A paper, the first from our group which mentions PTCA.
                 Actually doesn't look at PTCA maps during the procedure
                 but just before and after. thesis, PTCA034",
}

@Article{RSM:Mon89d,
  author =       "L.B. Montefusco and G. Casciola",
  title =        "Algorithm 67: $\rm {C}^{1}$ Surface Interpolation",
  journal =      j-ACM-TMS,
  year =         "1989",
  volume =       "15",
  number =       "4",
  pages =        "365--374",
  robnote =      "Nice methiod for displaying bivariate data using
                 triangulation and interpolation. Doesn't help to
                 interpolate in 3D since it requires projection to 2D
                 then interpolation.",
}

@Article{RSM:Mon90,
  author =       "T.J. Montague and F.X. Witkowski and R.M. Miller and
                 D.E. Johnstone and R.B. Mackenzie and C.A. Spencer and
                 B.M. Horacek",
  title =        "Exercise Body Surface Potential mapping in single amd
                 multiple coronary artery disease",
  journal =      "Chest",
  year =         "1990",
  volume =       "97",
  number =       "6",
  pages =        "1333--1342",
  robnote =      "Another TJ paper on BSPM in exercise in patients with
                 CAD. One interestig methodological issue was the use of
                 the sum over all leads of the change in ST-integral
                 values as an indicator of overal ischemic change. This
                 was used as the basis of statistical comparisons
                 between patients and normals and among CAD patient
                 groups with different reponses to exercise. The main
                 results are that the sum of the ST integral maps can
                 serve as a discriminator between single and multiple
                 vessle disease, although the overlap between groups is
                 too large for this tp be much use. Also, in patients,
                 the ST changes persisted at least 5 minutes after
                 exercise, while the changes in normals were gone by
                 this stage of things.",
}

@InCollection{RSM:Moo00,
  author =       "J.W. Moore",
  title =        "Voltage Clamp Methods",
  booktitle =    "Biophysics and Physiology of Excitable Membranes",
  editor =       "",
  publisher =    "unknown",
  address =      "",
  year =         "unknown",
  pages =        "143--167",
  robnote =      "Chapter 5 in a pretty good looking into many aspects
                 of excitable cells. cell electrophysiology, giant axon,
                 membranes, voltage clmap, sucrose gap. Basic chapter on
                 voltage clamp in nerve cells. See the Palti refs too,
                 since I have bound the articles for the book together.,
                 CellPhysiol095",
}

@Article{RSM:Moo75,
  author =       "J.W. Moore and F. Ramon and R.W. Joyner",
  title =        "Axon voltage-clamp simulations",
  journal =      j-BJ,
  year =         "1975",
  volume =       "15",
  pages =        "11--24",
  robnote =      "CellPhysiol, modelling of voltage clamp",
}

@Article{RSM:Moo84,
  author =       "J.W. Moore and M. Hines and E.M. Harris",
  title =        "Compensation for Resistance in Series with Excitable
                 Membranes",
  journal =      j-BJ,
  year =         "1984",
  volume =       "46",
  pages =        "507--514",
  robnote =      "A description of the sources and solutions to series
                 resistance in cellular preparations, especially with
                 regard to voltage clamp., CellPhysiol082",
}

@Article{RSM:Moo86,
  author =       "R. Moog",
  title =        "Digital Music Synthesis",
  journal =      j-BYTE,
  year =         "1986",
  month =        jun,
  pages =        "155--168",
  robnote =      "The many different shapes of the waveform of the
                 present. This article gives an overview of the use of
                 computer and digital hardware in the production of
                 music., CompHard006",
}

@Article{RSM:Moo96,
  author =       "T.K. Moon",
  title =        "The Expectation-Maximization Algorithm",
  journal =      "IEEE Sig. Proc. Mag.",
  year =         "1996",
  pages =        "47--60",
  month =        nov,
  robnote =      "Brooks",
  bibdate =      "Fri Jan 30 13:44:17 1998",
}

@Article{RSM:Mor72,
  author =       "P.R. Moroko and P. Libby and J.W. Covell and B.E.
                 Sobel and J. Ross and E. Braunwald",
  title =        "Precordial {ST}-Segment Elevation mapping: {A}n
                 Atraumatic method for Assessing Alterations in the
                 Extent of Myocardial Ischemic Injury",
  journal =      j-AJC,
  year =         "1972",
  volume =       "2",
  pages =        "223--230",
  robnote =      "the effects of pharmacologic and hemodynamic
                 interventions. This is basically early work, looking at
                 the ST-shifts in dogs and a few patients to even prove
                 the effect was present. thesis, Ischemia029",
}

@Article{RSM:Mor80,
  author =       "H. Mor\'{e}na and M.J. Janse and J.W.T. Fiolet and
                 W.J.G. Krieger and H. Crijns and D. Durrer",
  title =        "Comparison of the Effects of Regional Ischemia,
                 Hypoxia, Hyperkalemia, and Acidosis on Intracellular
                 and Extracellular Potentials and Metabolism in the
                 Isolated Porcine Heart",
  journal =      j-CR,
  year =         "1980",
  volume =       "46",
  pages =        "634--646",
  robnote =      "This ons is referred to quite a bit by the Janse group
                 thesis, Ischemia156",
}

@Article{RSM:Mor81,
  author =       "P. Morizet-Mahoudeaux and J.J. Quarante",
  title =        "Simple Microprocessor Based System for On-line {ECG}
                 Arrhythmia Analysis",
  journal =      j-MBEC,
  year =         "1981",
  volume =       "1",
  pages =        "497--500",
  robnote =      "ECG processing, monitoring, arrhythmia, Arrhythm015",
}

@Article{RSM:Mor89,
  author =       "B.C. Morton and A.J. Higginson and R.J. Chambers and
                 D.S. Beanlands",
  title =        "Late Reassessment of Percutaneous Transluminal
                 Coronary Angioplasty Results: {C}linical, Exercise and
                 Angiographic Follow-up",
  journal =      j-CJC,
  year =         "1989",
  volume =       "5",
  pages =        "291",
  robnote =      "A fairly big study on longterm outcome of PTCA. Here
                 the author staes that the first PTCA was perfomed in
                 1977 by Gruentzig and appeared in the 1978 N.E. J Med.
                 article a year later. The results indicate that PTCA
                 and bypass are equally effective in longterm
                 restoration of vessel patency and health of the
                 patient. thesis., PTCA042",
}

@Article{RSM:Mor95,
  author =       "C. A. Morillo and G. J. Klein and D. L. Jones and C.
                 M. Guiraudon",
  title =        "Chronic rapid atrial pacing. Structural, functional,
                 and electrophysiological characteristics of a new model
                 of sustained atrial fibrillation",
  journal =      j-C,
  volume =       "91",
  number =       "5",
  pages =        "1588--1595",
  abstract =     "BACKGROUND: Despite the clinical importance of atrial
                 fibrillation (AF), the development of chronic
                 nonvalvular AF models has been difficult. Animal models
                 of sustained AF have been developed primarily in the
                 short-term setting. Recently, models of chronic
                 ventricular myopathy and fibrillation have been
                 developed after several weeks of continuous rapid
                 ventricular pacing. We hypothesized that chronic rapid
                 atrial pacing would lead to atrial myopathy, yielding a
                 reproducible model of sustained AF. METHODS AND
                 RESULTS: Twenty-two halothane-anesthetized

license: MIT

license_type: permissive